#Early Signs of Kidney Failure

[vaidyaslaboratory]

How Diabetes Can Lead to Heart and Kidney Failure: Symptoms, Risks, and Prevention

Diabetes silently increases the risk of both heart and kidney failure, leading to life-threatening complications. In this blog, we explore how high blood sugar levels damage vital organs, the early warning signs of heart and kidney failure, and the importance of regular tests for prevention. Learn about essential tests like HbA1c, NT-proBNP, and Urine Microalbumin, and discover how Dr. Vaidya’s Diabetic Heart & Kidney Disease Prevention Package, available at ₹2999, can help protect your health.

[tricountyanimal]

Recognizing the Signs of Kidney Failure in Pets

Kidney failure is a serious condition that can affect pets, often catching owners by surprise. The kidneys play a crucial role in filtering waste from the blood and balancing fluids in the body. When they fail, it can lead to severe health issues. Understanding and recognizing the signs of kidney failure in pets is vital for early intervention and effective treatment. At Tri-County Animal Hospital, we are dedicated to providing the best care and information to ensure your pet's well-being.

Understanding Kidney Failure in Pets

What is Kidney Failure?

Kidney failure, or renal failure, occurs when the kidneys lose their ability to function properly. This condition can be acute (sudden onset) or chronic (developing over time). In both cases, it’s crucial to identify the problem early to manage it effectively.

Types of Kidney Failure: Acute vs. Chronic

Acute Kidney Failure: Often caused by toxins, infections, or severe dehydration, this type of kidney failure comes on suddenly and can be reversible with prompt treatment.

Chronic Kidney Failure: This is a progressive condition often associated with aging, underlying diseases, or genetic factors. It’s managed over time rather than cured.

Causes of Kidney Failure in Pets

Common Causes in Dogs and Cats

Several factors can lead to kidney failure in pets, including:

Toxins (e.g., antifreeze, certain medications)

Infections (e.g., leptospirosis)

Trauma to the kidneys

Chronic diseases (e.g., diabetes, high blood pressure)

Genetic Predispositions

Some breeds are more prone to kidney issues due to genetic factors. For instance, Persian cats and certain dog breeds like the Shih Tzu and Bull Terrier are more susceptible to kidney diseases.

Environmental Factors

Environmental toxins and poor diet can also contribute to kidney failure. Ensuring a clean, safe environment and a balanced diet is crucial for kidney health.

Early Signs of Kidney Failure

Recognizing the early signs of kidney failure can make a significant difference in the outcome for your pet.

Behavioral Changes

Increased thirst and urination

Decreased appetite

Lethargy or depression

Physical Symptoms to Watch For

Weight loss

Bad breath with a chemical odor

Vomiting and diarrhea

Poor coat condition

Advanced Symptoms of Kidney Failure

As the condition progresses, symptoms become more severe and require immediate veterinary attention.

Severe Symptoms Indicating Progression

Severe lethargy

Swelling in limbs (edema)

Seizures or disorientation

When to Seek Emergency Vet Care

If you notice any advanced symptoms, contact Tri-County Animal Hospital immediately. Early intervention can prevent further complications and improve the quality of life for your pet.

Diagnosing Kidney Failure

Veterinary Tests and Procedures

Diagnosing kidney failure typically involves:

Blood tests to check for elevated waste products (BUN, creatinine)

Urinalysis to assess kidney function and detect abnormalities

Imaging (ultrasound or X-rays) to view the kidneys

Importance of Early Detection

Early detection is key to managing kidney failure. Regular check-ups at Tri-County Animal Hospital can help catch the disease in its early stages, allowing for more effective treatment options.

Treatment Options 

Medical Treatments

Treatment varies depending on the severity and type of kidney failure but may include:

Medications to manage symptoms and slow disease progression

Antibiotics for infections

Medications to control blood pressure

Dietary Changes

Special diets low in phosphorus and protein can help reduce the workload on the kidneys and manage symptoms.

Fluid Therapy

Hydration is crucial. Subcutaneous or intravenous fluids may be administered to help maintain electrolyte balance and hydration.

Home Care for Pets with Kidney Failure

Daily Care Routines

Ensuring your pet has access to fresh water at all times

Administering medications as prescribed

Providing a comfortable resting area

Monitoring and Adjusting Treatment

Regularly monitor your pet’s symptoms and keep in touch with Tri-County Animal Hospital for follow-up visits and adjustments to the treatment plan as needed.

Preventive Measures

Tips for Preventing Kidney Failure

Maintain a balanced diet for your pet

Ensure regular veterinary check-ups

Avoid exposure to toxins

Importance of Regular Veterinary Check-Ups

Regular visits to Tri-County Animal Hospital allow for early detection and prevention strategies, helping to keep your pet’s kidneys healthy.

Conclusion

Recognizing the signs of kidney failure in pets and seeking timely veterinary care can make a significant difference in your pet’s health and quality of life. At Tri-County Animal Hospital, we are committed to providing comprehensive care and support for pets with kidney failure. By staying informed and proactive, you can help your furry friend live a happy, healthy life.

[healthnews101]

"13 Signs and Symptoms of Failing Kidneys: Don't Ignore These Symptoms for Early Detection and Treatment"

Are you aware of the signs and symptoms of failing kidneys? Your kidneys play a vital role in maintaining your overall health and well-being, but when they start to fail, it’s crucial to recognize the warning signs early on. In this comprehensive article, we will delve into the various indicators that may suggest kidney dysfunction. Whether you want to be proactive about your health or are simply…

[literaryvein-reblogs]

Writing Notes: Hypothermia

Hypothermia - A potentially fatal condition; occurs when body temperature falls below 95°F (35°C).

The danger signs include:

intense shivering;

stiffness and numbness in the arms and legs;

stumbling and clumsiness;

sleepiness, confusion, disorientation, amnesia, and irrational behavior; and

difficulty speaking.

The signs and symptoms of hypothermia follow a typical course, though the body temperatures at which they occur vary from person to person depending on age, health, and other factors.

The impact of hypothermia on the nervous system often becomes apparent quite early. Coordination, for instance, may begin to suffer as soon as body temperature reaches 95°F (35°C).

The early signs of hypothermia also include cold and pale skin and intense shivering; the latter stops between 90°F (32.2°C) and 86°F (30°C).

As body temperature continues to fall:

speech becomes slurred,

the muscles go rigid, and

the victim becomes disoriented and experiences eyesight problems.

Other harmful consequences include:

Dehydration

Liver and kidney failure

Heart rate, respiratory rate, and blood pressure rise during the first stages of hypothermia, but fall once the 90°F (32.2°C) mark is passed.

Below 86°F (30°C) most victims are comatose, and

below 82°F (27.8°C) the heart’s rhythm becomes dangerously disordered.

However, even at very low body temperatures, people can survive for several hours and be successfully revived, though they may appear to be dead.

TREATMENT

Until emergency help arrives, a victim of outdoor hypothermia should be brought to shelter and warmed by:

removing wet clothing and footwear,

drying the skin, and

wrapping him or her in warm blankets or a sleeping bag.

Gentle handling is necessary when moving the victim to avoid disturbing the heart.

Rubbing the skin or giving the victim alcohol can be harmful.

Warm drinks such as clear soup and tea are recommended for those who can swallow.

Anyone who aids a victim of hypothermia should also look for signs of frostbite and be aware that attempting to rewarm a frostbitten area of the body before emergency help arrives can be extremely dangerous. For this reason, frostbitten areas must be kept away from heat sources such as campfires and car heaters.

Rewarming is the essence of hospital treatment for hypothermia.

How rewarming proceeds depends on the body temperature.

Other considerations, such as the patient’s age or the condition of the heart, can influence treatment choices.

Different approaches are used for patients who are:

mildly hypothermic (the patient’s body temperature is 90–95°F [32.2–35°C]),

moderately hypothermic (86–90°F [30–32.2°C]), or

severely hypothermic (less than 86°F [30°C]).

Mild Hypothermia

Reversed with passive rewarming.

This technique relies on the patient’s own metabolism to rewarm the body.

Once wet clothing is removed and the skin is dried, the patient is covered with blankets and placed in a warmroom.

The goal is to raise the patient’s temperature by 0.9–3.6°F (0.5–2°C) an hour.

Moderate Hypothermia

Often treated first with active external rewarming and then with passive rewarming.

Active external rewarming - applying heat to the skin (e.g., by placing the patient in a warm bath or wrapping the patient in electric heating blankets).

Severe Hypothermia

Requires active internal rewarming, which is recommended for some cases of moderate hypothermia as well.

There are several types of active internal rewarming:

Cardiopulmonary bypass - the patient’s blood is circulated through a rewarming device and then returned to the body, is considered the best, and can raise body temperature by 1.8–3.6°F (1–2°C) every 3–5 minutes. However, many hospitals are not equipped to offer this treatment. The alternative is to:

Introduce warm oxygen or fluids into the body.

Hypothermia treatment can also include, among other things:

insulin,

antibiotics, and

fluid replacement therapy.

When the heart has stopped, both cardiopulmonary resuscitation (CPR) and rewarming are necessary. Once a patient’s condition has stabilized, he or she may need treatment for an underlying problem such as alcoholism or thyroid disease.

Source ⚜ More: Notes & References ⚜ Writing Realistic Injuries

[fattlestacks]

Sweet Spots is back home, unfortunately no good news. He has been in the early stages of kidney failure for the past few years which has thankfully stayed stable but on the ultrasound they could see he also has polycystic kidney disease as well as thickened intestinal lining which would indicate either Inflammatory Bowel Disease or cancer. The only way to know for sure would be biopsy but because of the kidney disease he is not a good candidate for surgery.

Our only option now is to treat the IBD with steroids and hope for the best. We don't have an estimate or anything for how long we may have left with him but I plan to make the most of it. As much as I don't want to lose him, I can at least say that he's had a good long life. Hopefully the meds will help him feel good and maybe even give him some extra time. FWIW he was playing last night so I hope he doesn't feel too crappy at least.

Thank you to everyone who's signed up for my commission wait list, it's really helped a lot. His diagnostics ended up being a little over $1600 so I really, really appreciate the support! And Sweets does too. I will keep y'all updated on how he and Fat Nerms are doing (Nerms recently had an ultrasound with similar IBD/Cancer indicative results but he seems stable for now, he at least has good kidneys).

<3

[tethered-heartstrings]

genuinely not trying to dunk on molly but when ppl say will and molly were so compatible they loved dogs i just think of molly feeding them canned food from china and not even knowing it could harm the dogs, i feel like this is significant because this was what led to dolarhyde being undetected and her attack which led to her estrangement from will, even her common values w will she did not actually share at all there's a meta here

ngl this is pretty clearly dunking on molly. But let’s refute. I personally haven’t seen many metas/posts about the compatibility between will and molly, so I can’t really contest what they say. However, will and her were as compatible as will led her to believe, as much as he would let. He lied to her about his past, which made it so she couldn’t know all of him and couldn’t connect to parts she couldn’t see. Not a fault of molly’s at all

Having looked back on the script for that episode, no credible veterinarian would ever say “was it canned food from china”. It’s unprofessional and xenophobic. Also, a vet in an emergency situation isn’t going to use scare tactics. They just need to know what went in so they can take care of it. Yes, molly was feeding the dogs canned food because will was out of town so he couldn’t make them food. and I can guarantee will is rational enough to say “hey, these brands are okay” and molly isn’t stupid. And they are very well off, so they would need to buy “cheap dog food from china”. If we want to talk about the dialogue in that scene, we have to admit to ourselves fuller is a misogynist who can’t write women, so ofc he made her say “is it bad to be made in china”. It also neglects how SOME but not ALL food has shown problems, but dog food made in the US has also had toxicity issues! And (I can’t remember if this was in show or just in script) the vet says “pet food safety isn't regulated the same way as human food” which is just false. There is high regulation in pet food, and pet food is made to be human grade and safe for human consumption (and has been for decades). Dogs are more likely to be poisoned by xylitol or chocolate from your candy or get pancreatitis from your table scraps than die from “dog food from china”.

But let’s humor for a second it was the cheap dog food molly bought. The concern and toxic component is melamine, which is added to food to fraudulently increase the protein content. it’s nontoxic (ish) by itself and only becomes a problem when combined with cyanuric acid that forms a crystal and leads to renal toxicosis and failure. Some gi biomes have bacteria that can convert melamine to cyanuric acid, but not all, making it more toxic for these patients. It also wasn’t solely a dog food thing, it happened in human children, too. All that aside, most melamine toxicosis cases are chronic, aka molly would have had to feed the dogs for a while, building up these crystals, and slowly killing the kidneys. This wouldn’t happen suddenly to all the dogs at once. Early signs of toxicosis are very detectable symptoms (vomiting, polyuria, polydipsia, lethargy) and these are signs molly would have noticed and done something about. ALSO if it was melamine the veterinarian was worried about, she wouldn’t use activated charcoal as her treatment (as she states in script, again, don’t remember if this was said in the show). Activated charcoal only works for something currently in the stomach, and that isn’t the pathophysiology for melamine. To treat melamine toxicity, you need iv fluids and supportive care. So what we can take from the veterinarian interaction is 1) fuller doesn’t know how medical professional talk to clients and 2) he doesn’t even understand the toxin he mentioned so it feels xenophobic as fuck to even bring that up.

And no, her canon (fuck you fuller) ignorance to the danger of canned dog food isn’t the cause of dolarhyde being undetected and attacking her. Even if she was feeding them dog food with melamine, Will literally figures out and says that dolarhyde poisons the dogs in the family to get rid of the “alarm system”, so he can attack and not have the family alerted. It didn’t matter what dog food molly fed them, dolarhyde intentionally poisoned them. Even if she fed them food will handmade, dolarhyde would have still poisoned them. And she was a good owner who brought all 7 (probably) puking dogs to the vet as soon as she could. molly had nothing to do with dolarhyde attacking her. dolarhyde had something to do with dolarhyde attacking her (and hannibal ofc). At least give her some credit for, you know, surviving

And no, her getting attacked was not the cause of will estranging himself from her. He never really loved her and used her as a fill in for a man he outright rejected. He was having an emotional affair with Hannibal and not giving a shit about molly. That was nothing molly could control.

And we really can’t thoroughly discuss her values because we barely see her onscreen. Her screentime is a plot motivator, we aren’t privy to who she is as a person. She loves her son, lost her first husband, and loves will. That’s kind of it. Oh, yeah, she also takes her son fishing without Fisherman Husband because he chose to estrange himself.

[mizufae]

So, because of my kidney stone and the election happening at the same time, I started watching Friends from the beginning all the way through, as a pleasant departure from reality. Today I finished watching all ten seasons. I watched it when it was first airing on nbc and again multiple times throughout my life but I don’t think I ever watched it start to finish like this before. Notably it was as background tv to playing animal crossing and sundry tasks, so it’s not like I was truly binging it in the hyper focus sense. But anyway I have some thoughts.

One, Chandler’s dad is a trans woman who is also a professional drag queen and she is unfairly maligned by the entire cast repeatedly, which sucks, but if we extend the fiction twenty years she would be a repeated guest judge on drag race and Chandler would get cool points galore.

Two, Chandler is nonbinary. Obviously the language and common awareness of this way of being wasn’t really out there when the show aired but presumably Phoebe or his dad would bring it up eventually, and my god is it written into the show. Every time Chandler is powerfully uncomfortable and disingenuous it’s because of a failure of fitting into explicit gender roles. Every time he expresses disgust at girly things, he is shown indulging in them in secret or with his most trusted confidants later. Every time he enjoys girly things comfortably he has to balance it out with machismo afterwards, but he is hopelessly uncomfortable with machismo as well. And it’s not just that kind of stuff, it’s his role in the friend group as occasional genderless clown, his self presentation, his repeatedly acceptance of mediocre compromise in his professional and romantic life because to really ask for what he wants seems impossible… I could go on. But anyway hopefully mid 50s Chandler is signing his emails with “Chandler Bing, he/they” and crossing his fingers that nobody asks him about it and then his work friends throw him a surprise coming out party he didn’t want.

Three, Chandler, Monica, and Joey are clearly in a poly situationship - we have regressed societally in a lot of ways but the ease with which male friendship and deep affection was expressed on Friends sticks out particularly in light of our current culture, but the three of them have something definitely beyond that. Like, Joey has a room in their house. Joey asks if he can have an aquarium and a sex swing in that room. Who are you sharing the sex swing with out in the suburbs, Joey???

Four, a lot has been said about the insidious silent racism of Friends’ extremely white fictional NYC, but wholly shit it pales in comparison to the rampant, explicit, unquestioned fatphobia. The fatphobia shows up in every episode. There is not a single fat character presented in a positive light. The only reason ugly naked guy is ugly is because he gained weight. Monica’s entire character is built on fatphobia. Even the kindest characters most accepting of individuality use fatness as an insult. We never even get a fat and happy stereotype, unless you count oblivious past fat Monica dancing while eating pizza, but we know she becomes thin shortly after this and also immediately before that shot she is described as “a pile of coats” on a bed. So like, ew. Anyway, the time period the show was filmed during is one of the worst for fat positivity. The perilously thin ideal body type of the mid 90s to early oughts is reflected in the show, but the show was making the culture for nearly half its seasons so it’s not like the writers were powerless to stop it or anything.

Five, Rachel should have ended up with Ross but Ross should have given up tenure to go to France with her! What absolute bullshit. He could have taken sabbatical and spent a gloriously romantic French summer with her and then they could have done the international young parents thing for a bit and Rachel could have come back after a few years to an even better job in the city. The way the whole show was about her slowly growing up and becoming assertive and taking risks and then the complete doormat Ross keeps her from a Parisian high fashion adventure??? Complete dud. I remember thinking this when it first aired, but goddamn, as a 40 year old I am so deeply disappointed.

Six, Phoebe is obviously bisexual and hopefully she came out to everyone some time soon after the show end, and also Mike made no sense. Like, he was fine, and fit in with the friend group okay, and obviously he is Paul Rudd so he is super cute and hot enough for Phoebe, but he was obviously shoehorned in and then barely developed. Half of the boys’ single episode dates had more personality than Mike shows for his entire run. David on the other hand was romantically planted for Phoebe for the entire show and was shockingly memorable considering how few episodes he was actually in. Phoebe’s ending should have been her getting a passport and flying to France with Rachel to be her translator and David meeting them at the airport because it turns out he and Phoebe had been penpals the whole time. But I digress.

Seven, did Ben and Carol die? Ben is phased out slowly, like, he shows up a few times as an older kid and then is mentioned in passing a few times, but Carol is basically disappeared into the void. She is seen like, once in the back half of the seasons and then never shows up or is mentioned again. She should have been there to tell Ross to go to France. She was supposed to be his friend. And his coparent. The rest of the friends all really liked her, and she fit into the expanded world perfectly well, alongside characters like Janice and Gunther, both of whom had important scenes in the last few episodes. So why not have a Carol cameo? A phone call??? Anything? A mention that Carol and Susan had moved to the burbs earlier and now Monica and Chandler were doing the same? Something?????? Poor work.

Eight, despite some of my issues above, Friends is an incredible piece of television. The ensemble never stops having chemistry. The combinations of characters always reveal a new element or dynamic. The costumes and sets are absolutely iconic, in ways I only now appreciate but inherently understood the first time. There is some of the tightest sitcom writing of all time. The guest stars were outrageous but always integrated, and the ensemble actors stood toe to toe with them no problem. There are genuine moments of love and stupidly funny gags all the time. Like, I’m annoyed by plenty of little things. But taken as a whole I now get why it was so big at the time and why it continues to endure.

[fooltemps]

Leukemia and Lymphoma Awareness Flags!!

This flag was designed by us, as we currently have a family member with Leukemia and wish to bring awareness to this kind of cancer.

color meaning:

#FF2D34: Myeloma

#00DC0E: Non-Hodgkin Lymphoma

#FF8C2E: Leukemia

#D12DFF: Hodgkin Lymphoma

Below is information all about Leukemia and Lymphoma Cancers.

Leukemia and Lymphoma are both cancers that are not associated with a tumor. Lymphomas are cancers that affect the lymph system and start in cells called lymphocytes. Leukemia is a cancer of the early blood-forming tissues, including your bone marrow and lymph system.

There are many types of lymphoma. Some grow and spread slowly and some are more aggressive. There are two main types of Lymphoma:

1. Hodgkin Lymphoma is cancer that starts in the B lymphocytes (B cells) of the lymph system. Your lymph system helps you fight infection and control the fluids in your body.

2. Non-Hodgkin Lymphoma (NHL) is cancer that starts in the lymphocytes anywhere lymph tissue is found:

Lymph nodes

Spleen

Bone marrow

Thymus

Adenoids and tonsils, or

The digestive track.

Leukemia typically involves white blood cells, the cells that are your infection fighters. Leukemia can be divided into categories: fast growing (acute) and slow growing (chronic); and by which white blood cells are affected:

Acute lymphocytic leukemia (ALL)

Acute myelogenous leukemia (AML)

Chronic lymphocytic leukemia (CLL

Chronic myelogenous leukemia (CML)

A screening test is used to detect cancers in people who may be at higher risk for developing the disease. With leukemia and lymphoma, there are no early detection tests. The best way to find them is to be aware of the symptoms:

Swollen lymph nodes which can appear as a lump in the neck, armpit or groin;

Fever

Night sweats

Weight loss without trying, and

Fatigue.

Leukemia can have similar symptoms but also can include:

Easy bleeding or bruising;

Recurring nosebleeds; and

Bone pain or tenderness

Myeloma is cancer of the plasma cells. Plasma cells are white blood cells that produce disease- and infection-fighting antibodies in your body. Myeloma cells prevent the normal production of antibodies, leaving your body's immune system weakened and susceptible to infection. The multiplication of myeloma cells also interferes with the normal production and function of red and white blood cells. An abnormally high amount of these dysfunctional antibodies in the bloodstream can cause kidney damage. Additionally, the myeloma cells commonly produce substances that cause bone destruction, leading to bone pain and/or fractures.

Myeloma cells are produced in the bone marrow, the soft tissue inside your bones. Sometimes myeloma cells will travel through your blood stream and collect in other bones in your body. Because myeloma frequently occurs at many sites in the bone marrow, it is often referred to as multiple myeloma.

Signs and symptoms of myeloma include the following:

Hypercalcemia (excessive calcium in the blood)

Anemia (shortage or reduced function of red blood cells)

Renal damage (kidney failure)

Susceptibility to infection

Osteoporosis, bone pain, bone swelling, or fracture

High protein levels in the blood and/or urine

Weight loss

In 2022, more than 62,650 people are expected to be diagnosed with leukemia. In addition:

Leukemia accounts for 3.6% of all new cancer cases.

The overall 5-year survival rate for leukemia has more than quadrupled since 1960.

62.7% of leukemia patients survive 5 years or more.

The diagnosis of leukemia requires specific blood tests, including an examination of cells in the blood and marrow.

Treatment and prognosis depend on the type of blood cell affected and whether the leukemia is acute or chronic. Chemotherapy and blood and marrow transplant are often used to treat leukemia.

If you wish to read more about Leukemia and Lymphoma cancer, please visit this website!

[oneshotnewbie]

Maybe a oneshot where B!D gets diagnosed with type 1 diabetes.

---

You sighed and ran your hand through your hair for seemingly the three hundredth time in half an hour while you were nervously waiting for your GP, who had summoned your sisters and you to her office.

For weeks you had no idea what was happening to you, denying every symptom as if it were kind of like a flu that caused them but didn´t break out properly; the excessive drinking and double the amount of urination, the increased hunger and urge to eat while losing weight and throwing up at the same time, getting up tired from a nine hour sleep and the weakness your body carried with it.

Not to mention the most violent mood swings your sisters have ever experienced with you.

The silence and your ongoing thoughts were interrupted by a heavy swing of the opening door before your doctor walked in. She was young, in her early thirties yet she was far better than anyone that has ever treated you in your life. "Hello, Y/n. Nice that you could come so quickly."

"You said it was urgent so I wasted no time."

Convincingly, she nodded with a soft smile and shook hands with each of your siblings in greeting. Wasting no time, your doctor settled into her much more comfortable looking chair than you were sitting in, being careful over her coat so it didn´t crumple under her.

"So Y/n, I have good and bad news." she started to speak, looking down at her tablet to review your blood results, letting the panic in you rise further with each second that passed. "The good news is; your blood results are almost perfect and you have no signs of kidney failure or anything close to the symptoms."

At that moment, an incredibly large stone fell from your heart and the adrenaline disappeared from your body. However, the panic was not immediately banned; only afterwards did you think about her choice of words and got stuck on one- almost.

"Almost perfect? Doctor, what does that mean?" Kara had taken the question out of your mouth; she too was probably confused by what the doctor said.

"Well, all but one." your doctor now shifted her gaze between the blonde and redhead, probably to add depth to her words. "An increased glucose value can be seen in your sisters venous plasma, and auto antibodies can also be detected in her blood, which should not normally be there."

Alex shifted uncomfortably in her seat and leaned further forward to rest her elbow on the young woman´s wooden desk. She had covered her mouth with her hand and widened worryingly her eyes at you as if she knew what was about to come.

After all, she was the only one of the three of your who had completed medical school.

"Y/n, unfortunately I have to tell you that you have type 1 diabetes." Your heart suddenly dropped and your stomach felt like a roller coaster, the lump in your throat expanded to an abnormal size. You slumped back in your seat, eyebrows furrowed in uncertainty.

Diabetes. You´d had it for roughly four weeks, hence why you felt like crap.

"For understanding; Type 1 Diabetes is a chronic autoimmune disease caused by your pancreas not producing enough insulin. Insulin is the principle hormone in regulating the glucose in your blood and without it, the glucose will not be absorbed properly, resulting in higher blood glucose levels. Higher glucose levels can result in polyuria and polydipsia which means an increase in urination and in thirst which you´re already going through- they are the first steps into this illness."

You only understood half of what she said to you, some words that she had in her vocabulary so complicated that you would have needed a dictionary and a ten-page guide if she wouldn´t explain to you how the further process would work.

"You need to start poke your finger with this little needle poker device here." From a drawer in her desk, she pulled out a burgundy, funny-looking thing and laid it in your hands that you had already outstretched, longing for this device that´s gonna save your ass probably more times that you´re going to count.

Your sisters also leaned into you to see for themselves what it was. They listened intently to offer you the best support they could give you, swallowing every information they could get. "And then you have this little thing here, which will read the level of your blood sugar. Also you have to inject yourself insulin manually into your leg or stomach- wherever you´re comfortable."

You rolled your eyes and put the thing back on the table; annoyed.

To put it into other words- type 1 diabetes was a fucking annoying disease where your body randomly decided to give you a big "FUCK YOU!" and stop giving your body what it craved. And unless you got it under control, your organs could fail.

But you didn´t seem aware of this.

[materialsscienceandengineering]

This ultrasound sticker senses changing stiffness of deep internal organs

The sticky, wearable sensor could help identify early signs of acute liver failure.

MIT engineers have developed a small ultrasound sticker that can monitor the stiffness of organs deep inside the body. The sticker, about the size of a postage stamp, can be worn on the skin and is designed to pick up on signs of disease, such as liver and kidney failure and the progression of solid tumors. [...] In an open-access study appearing today in Science Advances, the team reports that the sensor can send sound waves through the skin and into the body, where the waves reflect off internal organs and back out to the sticker. The pattern of the reflected waves can be read as a signature of organ rigidity, which the sticker can measure and track.

Read more.

[dr-smita-goel-homeopathy-clinic]

*Dr. Smita Goel Homeopathy Clinic*

www.thehomeopathyclinic.co.in

A person with short stature, or restricted growth, does not grow as tall as other people of the same gender, age, and ethnicity. The person's height is below the 3rd percentile.

Short stature can be a variant of normal growth, or it may indicate a disorder or condition.

Growth rate is an important indicator of overall health. Children who do not reach the 5th percentile by the age of 5 years are said to be small for gestational age (SGA). A pediatrician will look out for signs of "failure to thrive."

Early intervention can prevent future problems in many cases.

Normally, at 8 years of age, a child's arm span is around the same as their height. If these measurements are out of proportion, this may be a sign of disproportionate short stature (DSS), sometimes known as "dwarfism."

Fast facts on short stature

Here are some key points about short stature. More detail is in the main article.

• Short stature can happen for a wide range of reasons, including having small parents, malnutrition, and genetic conditions such as achondroplasia.

• Proportionate short stature (PSS) is when the person is small, but all the parts are in the usual proportions. In disproportionate short stature (DSS), the limbs may be small compared with the trunk.

• If short stature results from a growth hormone (GH) deficiency, GH treatment can often boost growth.

• Some people may experience long-term medical complications, but intelligence is not usually affected.

Causes

Growth depends on a complex range of factors, including genetic makeup, nutrition, and hormonal influences.

The most common cause of short stature is having parents whose height is below average, but around 5 percent of children with short stature have a medical condition.

Conditions that can underlie short stature include:

• Undernutrition, due to a disease or lack of nutrients

• Hypothyroidism, leading to a lack of growth hormone

• A tumor in the pituitary gland

• Diseases of the lungs, heart, kidneys, liver, or gastrointestinal tract

• Conditions that affect the production of collagen and other proteins

• Some chronic diseases, such as celiac disease and other inflammatory disorders

• Mitochondrial disease, which can affect the body in different ways, including growth

Sometimes, an injury to the head during childhood can lead to reduced growth.

A lack of growth hormone can also lead to delayed or absent sexual development.

Rheumatologic diseases, such as arthritis, are linked to short stature. This may happen because of the disease, or as a result of the glucocorticoid treatment, which can affect the release of growth hormone.

Disproportionate short stature (DSS) usually stems from a genetic mutation that affects the development of bone and cartilage and undermines physical growth.

The parents may not have short stature, but they may pass on a condition that is linked to DSS, such as achondroplasia, mucopolysaccharide disease, and spondyloepiphyseal dysplasia (SED).

Types

There are different types and causes of short stature, or restricted growth, and they will present differently. Because the range of conditions is so broad, restricted growth can be classified in various ways.

One categorization is:

• Variant restricted growth

• Proportionate short stature (PSS)

• Disproportionate short stature (DSS)

Each of these categories includes a number of types and causes of short stature.

Variant restricted growth

Sometimes a person is small but otherwise healthy. This can be referred to as variant restricted growth. It may happen for genetic or hormonal reasons.

If the parents are also small, this can be called familial short stature (FSS). If it stems from a hormonal issue, it is a constitutional delay in growth and adolescence (CDGA).

The limbs and the head develop in proportion with the spine, and the individual is otherwise healthy.

Growth happens throughout the body, so the legs, for example, are in proportion with the spine.

In most cases, the individual's parents are also small, but sometimes small stature happens because the body does not produce enough growth hormone (GH), or the body does not process growth hormone properly. This is known as GH insensitivity. Hypothyrodism can lead to low hormone production.

Growth hormone treatment during childhood may help.

Proportionate short stature (PSS)

Sometimes, overall growth is restricted, but the person's body is in proportion, and the individual has a related health problem. This is known as proportionate short stature (PSS).

If the individual is heavy for their height, this can suggest a hormone problem. The problem could be hypothyroidism, excess glucorticoid production, or too little GH.

A person who is small and their weight is low for their height may be experiencing malnutrition, or they may have a disorder that leads to malabsorption.

Whatever the underlying reason, if it affects overall growth, it may impact development in at least one body system, so treatment is needed.

During adulthood, a person with this type of restricted growth is more likely to experience:

• osteoporosis

• cardiovascular problems

• reduced muscle strength

Rarely, there may be cognitive problems, or problems with thinking. This depends on the cause of the short stature.

Disproportionate short stature (DSS)

Disproportionate short stature (DSS) is linked to a genetic mutation. The parents are usually of average height. As with other types of short stature, a range underlying causes is possible.

An individual with DSS will be small in height, and they will have other unusual physical features. These may be visible at birth, or they may develop in time as the infant develops.

Most individuals will have an average-sized trunk and short limbs, but some people may have a very short trunk and shortened, but disproportionately large limbs. Head size may be disproportionately large.

Intelligence or cognitive abilities are unlikely to be affected unless the person has hydrocephalus, or too much fluid around the brain.

Achondroplasia underlies around 70 percent of cases of DSS. It affects around 1 in 15,000 to 1 in 40,000 people.

Features include:

• an average-sized trunk

• short limbs, especially the upper arms and legs

• short fingers, possibly with a wide space between the middle and ring fingers

• limited mobility in the elbows

• a large head with a prominent forehead and flattened bridge of the nose

• bowed legs

• lordosis, a progressive development of a swayed lower back

• average adult height of 4 feet, or 122 cm

Hypochondroplasia is a mild form of achondroplasia. It may be difficult to differentiate between familial short stature and achondroplasia.

Achondroplasia and hypochondroplasia result from a genetic mutation.

Genetic conditions, such as Turner syndrome, Down syndrome, or Prader Willi syndrome, are also linked to DSS.

Diagnosis

Some types of short stature can be diagnosed at birth. In other cases, routine visits to a pediatrician should reveal any abnormal growth pattern.

The doctor will record the child's head circumference, height, and weight.

If the doctor suspects restricted growth, they will carry out a physical examination, look at the child's medical and family history, and possibly carry out some tests.

These may include:

An x-ray, to assess for problems with bone development

An insulin tolerance test, to check for a deficiency in the growth hormone insulin-like growth factor-1 (IGF-1).

In this test, insulin is injected into a vein, causing blood glucose levels to drop. Normally, this would trigger the pituitary gland to release growth hormone (GH). If GH levels are lower than normal, there may be a GH deficiency.

Other tests include:

• a thyroid-stimulating hormone test, to check for hypothyroidism

• a complete blood count, to test for anemia

• metabolic tests, to assess liver and kidney function

• erythrocyte sedimentation and C-reactive protein tests, to assess for inflammatory bowel disease

• urine tests can check for enzyme deficiency disorders

• tissue transglutinase and immunoglobulin A tests, for celiac disease

• imaging scans, such as an x-ray of the skeleton and the skull or an MRI, can detect problems with the pituitary gland or hypothalamus

• bone marrow or skin biopsies may help confirm conditions associated with short stature

Treatment

Treatment will depend on the cause of the short stature.

If there are signs of malnutrition, the child may need nutritional supplements or treatment for a bowel disorder or other condition that is preventing them from absorbing nutrients.

If growth is restricted or delayed because of a hormonal problem, GH treatment may be necessary.

Pediatric hormone treatment: In children who produce too little GH, a daily injection of hormone treatment may stimulate physical growth later in life. Medications, such as somatropin, may eventually add 4 inches, or 10 centimeters, to adult height.

Adult hormone treatment

: Treatment for adults can help protect against complications, for example, cardiovascular disease and low bone mineral density.

Somatropin, also known as recombinant GH, might be recommended for people who:

• have a severe growth hormone deficiency

• experience impaired quality of life

• are already receiving treatment for another pituitary hormone deficiency

Adult patients generally self-administer daily with an injection.

Adverse effects of somatropin include headache, muscle pain, edema, or fluid retention, problems with eyesight, joint pain, vomiting, and nausea.

The patient may receive treatment to control chronic conditions, such as heart disease, lung disease, and arthritis.

Treatment for DSS

As DSS often stems from a genetic disorder, treatment focuses mainly on the complications.

Some patients with very short legs may undergo leg lengthening. The leg bone is broken and then fixed into a special frame. The frame is adjusted daily to lengthen the bone.

This does not always work, it takes a long time, and there is a risk of complications, including:

• pain

• the bone forming badly or at an inappropriate rate

• infection

• deep vein thrombosis (DVT), a blood clot in a vein

Other possible surgical treatments include:

• use of growth plates, where metal staples are inserted into the ends of long bones where growth takes place, to help bones grow in the right direction

• inserting staples or rods to help the spine form the right shape

• increasing the size of the opening in the bones of the spine to reduce pressure on the spinal cord

Regular monitoring can reduce the risk of complications.

Complications

A person with DSS may experience a number of complications.

These include:

• arthritis later in life

• delayed mobility development

• dental problems

• bowed legs

• hearing problems and otitis media

• hydrocephalus, or too much fluid in the brain cavities

• hunching of the back

• limb problems

• swaying of the back

• narrowing of the channel in the lower spine during adulthood and other spine problem

• sleep apnea

• weight gain

• speech and language problems

Individuals with proportionate short stature (PSS) may have poorly developed organs and pregnancy complications, such as respiratory problems. Delivery will normally be by cesarean section.

Outlook

Most people with short stature will have a normal life expectancy, and 90 percent of children who are small for their age at 2 years will "catch up" by adulthood.

The 10 percent who do not catch up are likely to have a condition such as fetal alcohol, Prader-Willi, or Down syndrome.

A person with achondroplasia can also expect a normal lifespan.

However, some serious conditions that are linked to some cases of short stature can be fatal.

[falloutnewvegastransedmygender]

Mae was born on a small Brahmin Ranch back in 2257. That's where she learned to shoot. Always preferred a revolver but she was also a fine shot with Da's hunting rifle. Ma died when she was 12 (2269), some raiders had attacked the farm. They shot back at them from the upstairs windows, but she took a bullet to the heart. Life was, never the same after that. Da sank into the bottle and never came out, died of kidney failure when she was 16 (2273). Mae held onto that ranch with bloody fingers for one long ass year, she was unable to make ends meet though. Sold it to a Brahmin Baron for a small sum, not nearly what it was worth. Managed to survive on that for a few months, trying to find some job. Never could find one though... She turned 18 (2275), without any job offers yet, and caps running out, she joined up with the NCR. The Treaty of New Vegas had only been signed the year prior (2274), and soldiers were needed to guard this territory. Scouts reported movements further East, but Legion and NCR had yet to skirmish. Mae disliked her service issue rifle but it was better than nothing so she held her tongue. After her 19th birthday (2276) however it was noticed she was rapidly losing weight and experiencing extreme fatigue. A medical checkup revealed she had developed Diabetes. A few decades ago this would have been a death sentence, however, the Followers of the Apocalypse had been able to resume synthetic insulin production in the previous years. New Vegas Clinic helped her secure an initial supply, and sold her a monthly supply at cost. She began to work as a prospector and a Courier. Once more living hand to mouth, just now in the Mojave. Mae became friends with some of the Followers in the Mojave. When the First Battle for Hoover Dam took place in 2277, she hunkered down at Old Mormon Fort, hoping to evacuate should Legion cross the dam. Mae fell flat on her ass, gasping as rubble flew a mile into the sky from the destruction of Boulder City, many other joined her in shock. Glad to hear the NCR had won the day, if only barely, she returned to work, just working to make ends meet. Until a fateful day many years later, early October, 2281, a twenty-four year old Mae took a jaw droppingly well paying job to deliver a small Platinum Poker Chip to New Vegas.

[transcendentalmaggot]

When I was 26, I contracted sepsis twice within an 18 month period due to kidney infections from certain particularities of my birth defects and the many surgeries I’ve had my lifetime. I had overextended myself when I already felt sick and foolishly went on a weekend vacation with my friends. By Tuesday, I was unable to lower even a persistent fever of 102 and went to see my PCP, who immediately wanted to call me an ambulance as my heart rate was extremely high and I could barely exist. In my infinite and foolhardy wisdom, more like stubbornness about being stuck with a bill for thousands of dollars, convinced him to let him drive me immediately to the ER. When I entered, I lost my breath and passed out before being immediately whisked into the monitoring room for cardiac issues. My EKG was awful - low blood pressure, high fever, high heart rate, muscle weakness and fatigue… all signs of sepsis. Of course because I was honest with them about my weekend debauchery. They immediately assumed I was having a reaction to cocaine use.. from an event that happened two days prior. I’ve done cocaine exactly one time in my life in my early twenties and I have no interest in using it again. I was thankfully treated with antiobiotics but experienced the worst full body pain I can possibly imagine. My high potassium levels from acute kidney failure caused my muscles to contract but did not allow them to relax. I shook constantly, full body shivering as if submerged in ice water. Even once my kidney infection was diagnosed, they still discharged me with a diagnosis of anticholinergic syndrome, or an overdose of OTC allergy medicine. It’s rare and would not have caused my symptoms but they were dead set on it having to do with drugs despite testing me extensively. When I went for my follow up with a nephrologist I greatly admire she immediately had that diagnosis removed from my records somehow since there was no evidence for that conclusion. They spent almost $600 drug testing me for everything you can possibly imagine. Even though the tests proved I had spoken the truth, I still felt that giant well of shame.

Anyway, my friends I went on vacation with asked how I was doing since I said I might go to the ER and this picture was my only response until I re-emerged several weeks later.

[rayoffuckingsun-shine]

Y’all, if you can, please help out my sister, Jennie. Due to long term effects from COVID, her health has been declining. She has been told she has congestive heart failure, early signs of kidney and liver failure, and now on oxygen 24/7. She has been fired from her job due to not being able to work, and has filed for disability. Her and her husband are now living off of his single salary and struggling.

She has started pulmonary and cardiovascular rehabilitation at a Long Term COVID clinic, but it will be a long road.

[sleuthy-scientist]

La Famiglia è per Sempre Chapter 1

Summary: David Rossi finds out he might be dying. Afraid of the reality of his situation, he tries to rely on the people around him for support. His new reality makes him question the meaning in his life. Along the way he discovers the truth about family.

~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~

~~~~~~~~~~

The Doctors said he was lucky they caught it when they did, considering the common early detection signs and symptoms for his condition hadn't occured. Luck had nothing to really do with it, Emily had been the one to convince him to get tested. First goading him to try and prove her wrong. And when that tactic didn't work, guilt tripping him with full on puppy dog eyes pleading, for her sake and peace of mind.

They told him that he had Polycystic kidney disease (PKD), Autosomal dominant polycystic kidney disease (ADPKD) to be exact. That his condition is a genetic disorder that causes cysts to grow in the kidneys, where they can disrupt functioning.

His high blood pressure had been noted in his charts by his regular physician with the suspected underlying cause of the stress due to a combination of his job and age. Yet unfortunately because of this slight oversight with his hypertension, his condition had progressed to the beginning stages of kidney failure.

Dave knew this immediately spelled desk duty for him. That his time as a field was done and his agent status may soon to have effectively come to an end. He had always assumed he would have retired this time due and work related injury or died a noble death on the job. It pained him knowing this could be the beginning of the end for him and his career.

The doctors with his input immediately came up with a plan of action. They decided to start him on medications and dialysis once a week, hoping that would be sufficient. That within a few weeks they would reevaluate, having a clearer picture and better handle on the needs and limitations of his condition.

With an actual diagnosis and regular treatment plan in place, Dave knew he wouldn't be able to hide it from the team for long. Especially with his treatment starting in two days. He knew he needed to start to get the priorities of his life in order, if he hoped to have any life left to live. His other priority was Joy and his BAU family. Until he had more news he didn't want to worry his daughter with his medical condition. That left the individuals who had already seen him at his worst, the ones who shared his everyday life. The people who he was responsible for, who he wanted to rely on him and be able to always support. Not the other way around, but he knew that may no longer be his decision. He couldn't lie or decide to up and leave them with no explanation. In their line of work they lost too many people and were left with to many regrets. He knew they would never forgive him if he kept this a secret. If he chose to disappear and slowly die on his own without them, out of pride or fear.

David Rossi was a lot of things, but he wasn't stupid, he knew he needed their support if he had any hopes of beating this thing. So he invited them over, for what might be the last team dinner he hosted for awhile. He wanted to have one more meal as a family with some semblance or sense of normalcy, knowing his news could wait until after dessert to be announced.

Everyone had been shocked and rendered momentarily speechless, worried for him upon learning of the life altering news he received. No one expected the nerve wracked Reid to be the first one to find words to fill the silence that had overtaken the room.

With his diagnosis, Spencer informed them all that typically his disease was caught a decade of two earlier and managed through diet, excercise and medication. That it had a fairly decent outcome and life expectancy if he followed the treatment plan.

He spouted off some other statistics most instantly forgot, too distracted by their own thoughts and feelings on the matter. But they still understood the gist of the genius's intended spiel. He was just as worried and distraught as the rest of them, try to make sense of the information they learned. But most of all afraid of the possibility of losing a piece of their family.

Once Reid finished speaking, he couldn't hold off the tears he had been trying to keep at bay. No one in the room was completely dry eyed as they all tried to digest the news. Slowly they all began to ask clarifying questions of the man of the hour about his condition and next steps going forward.

Hours later, Emily had been the last to leave, only showing the depth of her true emotions to him. Dave held onto her a little longer than usual, needing her strength, afraid to let go knowing he couldn't stop whatever came next for him. That this wasn't some nightmare he could simply wake up from.

As their embrace broke, he asked her what made her force him to get tested. He was hoping she would say something profound and wise, that would resonate with in him, and help him make sense of things.

Yet, she simply told him, it was a gut feeling she had, especially with his abnormal headaches as of late. That to her, something about him just seemed off, that he didn't seem himself with his usual laser sharp level of focus and observation. She couldn't explain it beyond that, beside knowing she needed him to be okay.

While her words hadn't provided him the type of clarification he had secretly and niavely hoping for, nor could she fix the predicament he found himself in, what she said provided another form of relief from his pain.

He wasn't surprised she of all people had noticed, Emily was the best profiler he had ever had the pleasure of working with. With her extreme empathy and gut instincts, she had always been the one looking out and supporting others, making sure they were ok. She had always been the first person to notice something was wrong and to begin trying to make it right.

He was just glad to have someone who obviously cared that deeply for his well-being in his life. For her sake and the team's, he wanted to bravely fight until he ran out of options. He hadn't given up against an enemy before, he wasn't about to start now.

~~~~~~~~~~

[bcisurat]

Multiple Myeloma: Explained By the Blood Cancer Specialist In Surat

Multiple myeloma is a type of blood cancer that affects plasma cells, a crucial part of the immune system. Plasma cells, found in bone marrow, produce antibodies to help fight infections. In multiple myeloma, these cells become cancerous, multiplying uncontrollably and crowding out healthy blood cells. This disease can weaken the immune system, damage bones, and lead to other serious complications. Our Cancer specialists in Surat, at BCI-Blood and Cancer Institute help us in understanding multiple myeloma — its symptoms, causes, diagnosis, and treatment — for early detection and better outcomes.

What Is Multiple Myeloma?

Multiple myeloma is classified as a cancer of the plasma cells. Normally, plasma cells create antibodies to help the body fight infections. In multiple myeloma, these cells produce abnormal proteins, known as monoclonal proteins or M proteins, instead of functioning properly.

These defective proteins can accumulate in the body, causing damage to the kidneys and other organs. Meanwhile, the overproduction of cancerous plasma cells can disrupt the production of healthy blood cells, leading to anemia, infections, and bone problems.

Symptoms of Multiple Myeloma

Blood cancer specialists in Surat warn that the symptoms of multiple myeloma may vary depending on the severity of the disease and the organs affected. Some common symptoms include:

Bone Pain: Persistent pain in the back, ribs, or hips is often a hallmark symptom, caused by the breakdown of bone tissue.

Frequent Infections: The immune system weakens, making it harder for the body to fight infections.

Fatigue and Weakness: A decrease in red blood cells (anemia) can lead to extreme tiredness.

Fractures: Bones may become fragile and break easily due to bone loss.

Kidney Problems: High levels of abnormal proteins can damage the kidneys, leading to issues such as kidney failure.

Numbness or Tingling: Nerve damage (neuropathy) can occur, particularly in advanced stages.

High Blood Calcium Levels (Hypercalcemia): Symptoms may include nausea, confusion, constipation, or excessive thirst.

Not everyone experiences all these symptoms, and some people may have no noticeable signs in the early stages.

Who Is at Risk for Multiple Myeloma?

While the exact cause of multiple myeloma is unknown, hemato-oncologists in Surat list several risk factors that increase the likelihood of developing the disease:

Age: The majority of cases occur in people over 60.

Gender: Men are slightly more likely to develop multiple myeloma than women.

Race: African Americans have a higher risk of multiple myeloma compared to other racial groups.

Family History: A family history of the disease can increase the risk.

Exposure to Toxins: Prolonged exposure to harmful chemicals or radiation may raise the likelihood of developing multiple myeloma.

Pre-existing Conditions: People with a condition called monoclonal gammopathy of undetermined significance (MGUS) have a higher risk of progressing to multiple myeloma.

How Is Multiple Myeloma Diagnosed?

Early detection is critical for managing multiple myeloma effectively. The diagnostic process typically involves:

Blood Tests: Doctors look for high levels of abnormal proteins (M proteins) and other markers, such as elevated calcium or low red blood cell counts.

Urine Tests: These detect abnormal proteins, known as Bence Jones proteins, in the urine.

Bone Marrow Biopsy: A sample of bone marrow is examined to confirm the presence of cancerous plasma cells.

Imaging Tests: X-rays, MRIs, or CT scans are used to identify bone damage or tumors.

The results of these tests help doctors determine the stage of the disease and develop a treatment plan tailored to the patient’s needs.

Treatment Options for Leukemia-Multiple Myeloma

While there is no cure for multiple myeloma, treatments aim to control the disease, relieve symptoms, and improve quality of life. Common leukemia cancer treatment in Surat include:

1. Targeted Therapy

Targeted drugs attack specific molecules involved in the growth of cancerous plasma cells. These therapies, such as proteasome inhibitors (e.g., bortezomib), interfere with the cancer cell’s ability to survive.

2. Chemotherapy

Chemotherapy drugs kill rapidly dividing cancer cells. They are often used in combination with other treatments.

3. Immunotherapy

Immunotherapy boosts the immune system to fight cancer. Monoclonal antibodies or CAR T-cell therapy may be used to target and destroy cancer cells.

4. Stem Cell Transplant

In eligible patients, a stem cell transplant can replace diseased bone marrow with healthy cells. This procedure is often combined with high-dose chemotherapy.

5. Radiation Therapy

Radiation is used to target and shrink tumors or relieve bone pain caused by cancer.

6. Supportive Care

Treatments for anemia, bone pain, or infections can help manage symptoms and improve quality of life.

Coping with Multiple Myeloma

Living with multiple myeloma can be physically and emotionally challenging. Patients undergoing blood cancer treatment in Surat should work closely with their healthcare team to manage symptoms and side effects. Additionally:

● Stay Informed: Understanding the disease and your treatment options can empower you to make informed decisions.

● Seek Support: Join support groups or connect with others who understand the journey of living with multiple myeloma.

● Focus on Wellness: Eating a healthy diet, staying active, and managing stress can improve overall well-being.

Conclusion

Multiple myeloma is a complex disease that requires ongoing management, but advances in treatment have significantly improved outcomes for many patients. Early diagnosis, personalized treatment, and supportive care from the best blood cancer hospital in Surat can help individuals maintain a good quality of life.