#glucose tolerance test
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Know Everything About Oral Glucose Tolerance (Ogtt) Test at Livlong
Discover the complete details of the oral glucose tolerance test (OGTT) to diagnose diabetes. Visit Livlong for more information on OGTT test at Livlong
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Exploring the Intricacies of Type 4 Diabetes: Gestational Diabetes Mellitus
Greetings, esteemed readers, and welcome to this all-encompassing, highly detailed guide centered on the elucidation of Type 4 Diabetes, an intriguing and somewhat less ubiquitous entity compared to its better-known counterparts, Type 1 and Type 2 Diabetes. Our paramount objective within the confines of this article is to embark on a comprehensive exploration, traversing the labyrinthine…

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#blood sugar levels during pregnancy#diabetes and pregnancy outcomes#Diabetes during pregnancy#diabetes management during pregnancy#exercise during pregnancy#GDM#gestational diabetes#gestational diabetes mellitus#gestational diabetes symptoms#gestational hypertension#glucose challenge test#glucose tolerance test#healthy pregnancy#high birth weight#insulin injections during pregnancy#Insulin Resistance#macrosomia#managing blood sugar during pregnancy#managing gestational diabetes#preeclampsia#Pregnancy and Diabetes#pregnancy and insulin#pregnancy and nutrition#pregnancy complications#pregnancy diabetes#pregnancy diet#pregnancy health#pregnancy health tips#prenatal care#prenatal screening
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I think if you have to fast for 14 hours, spend over 2 hours sitting in a hospital waiting room, force down some sugary gloop, give 2 blood samples, feel your blood sugar spike and then crash into the floor, and have the nurse say she reckons you might fall over on your way downstairs; you should get more out of your results than waiting a good half day longer than you hoped to and the single word "normal"
#fez talks#guess that means it's not a diabetic pregnancy at least#I would not recommend the glucose tolerance test as a day out
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Early Pregnancy HbA1c: A Promising Tool for Gestational Diabetes Screening
Early Pregnancy HbA1c: A Promising Tool for Gestational Diabetes Screening @neosciencehub #healthcare #gestationaldiabetes #sciencenews #Hba1c #neoscience #OralGlucoseToleranceTest(OGTT)
Gestational Diabetes Mellitus (GDM) is a condition that develops during pregnancy, characterized by impaired glucose tolerance. This condition can lead to significant health risks for both the mother and the foetus, including increased risk of preeclampsia, caesarean delivery, and adverse birth outcomes. Early detection and management of GDM are crucial to mitigate these risks. Traditional…
#featured#Gestational Diabetes Mellitus (GDM)#Haemoglobin A1c (HbA1c)#Oral Glucose Tolerance Test (OGTT)#sciencenews
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A Glucose Tolerance Test (GTT) is a medical procedure used to assess how well your body processes glucose. It is commonly used to diagnose diabetes, gestational diabetes, and other disorders of glucose metabolism. The procedure involves fasting overnight, followed by a blood draw to measure your fasting blood glucose level. You will then consume a sugary solution, and additional blood samples will be taken at regular intervals to monitor how your blood glucose levels change over time. This test helps healthcare providers determine how efficiently your body metabolizes sugar.
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Muktotsav: A Celebration of Freedom
Muktotsav comes from the conjoining of Mukti, meaning freedom, and Utsav, meaning festival or celebration. And this is exactly what it is. Muktotsav is FFD’s annual event when our champions step into the well-deserved limelight. This is when they are felicitated and awarded for their achievements.
Read the full blog here: https://www.freedomfromdiabetes.org/blog/post/muktotsav-a-celebration-of-freedom-Freedom-from-diabetes/2637
#Muktosav#muktotsav pramod#muktotsav diabetes#GTT#Glucose Tolerance Test (GTT)#Muktotsav is FFD’s annual event
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Glucose Tolerance Test Market Analysis, Size, Share, and Forecast 2031
#Glucose Tolerance Test Market#Glucose Tolerance Test Market Scope#Glucose Tolerance Test Market Report
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I've been trying to figure out what the deal is with prediabetes so I can write a meaningful response to an ask I got about it, and I just keep going wait--okay--here's one paper--but here's another one--here's a Cochrane review--but here's a different meta-analysis--and here's newer data from an RCT...
It's nuts! It's bananas. And anybody who says we have good, crisp, clear guidelines around what prediabetes even IS, much less what to do about it, is FULL OF SHIT.
What I really need to know in order to feel more confident about my handle on whether to medicate pre-diabetes is the population incidence. Not prevalence. Because if I take the most optimistic studies about medication as an intervention, specifically, I could be looking at about a 30-40% reduction in risk of progression to diabetes. But! How many people is that, actually? Because medication is not without its harms! We need to compare number needed to treat with number needed to harm, we need to have high-quality evidence that says yes, if we give this medication to everyone who meets X level of criteria for pre-diabetes (it's different in different sources AND it's changed repeatedly over our lifetime!), we will see a level of benefit sufficient to justify making these other people who would not have progressed to diabetes without it endure the hassle and side effects of taking a medication for the rest of their lives.
AND HERE'S THE REAL FUN PART: we don't really know where tissue damage begins! We thought we did! 6.5-7ish A1c. But it turns out there is a marked risk of retinopathy beginning at 5.5! Which is considered normal. AND ALSO we should probably be thinking of it as at least three separate disease based on our current ability to measure--A1c is a broad marker that collapses multiple forms of dysregulated blood sugar, and when we use more fine-grained tests, we see meaningful distinctions that probably affect preferred treatments between people who have impaired fasting glucose, people who have abnormal values on an oral glucose tolerance test, and people who have both. We should treat these groups differently because they reflect different underlying pathways: elevated fasting glucose means your liver is breaking down too much glycogen while you sleep, which is one issue, while elevated post-prandial glucose means your skeletal muscles (OR SOMETHING ELSE they're not totally sure) are behaving abnormally in response to insulin. IT'S NOT THE SAME THING and people with both impaired fasting glucose and abnormal post-prandial glucose are at higher risk of progression to diabetes/tissue damage than people with just one of those. AND WHILE WE'RE AT IT, what is diabetes? What's the best cutoff? What's the best measure? How many underlying pathophysiologies are getting collapsed into the same group????
THE MORE I LEARN ABOUT THIS THE MORE QUESTIONS I HAVE and experts are all being serenely confident while contradicting each other so I have to actually dig in the data a lot harder than I usually do. I've been meaning to do this for months, but one of the presenters this morning made a comment about the benefits of putting prediabetics on metformin that made me go "hm, do I need to start doing that?" and I've gone from my kneejerk answer being "no, we studied this and it doesn't help" to "I don't fucking know and neither does anyone else."
...as always, Cochrane is probably right.
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blood taken during d&e’s autopsies

JSCO#8773 — taken from dylan, one grey top tube of blood.
JSCO#8774 — taken from eric, one red, one purple, and one orange tubes of blood.
(i’ve redacted the details of which vials were drawn from the victims out of respect for their memory.)
⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡ ⟡
from what i’ve researched about phlebotomy, the colors refer to the following tests:
grey: fasting blood sugar (FBS), glucose tolerance test (GTT), lactic acid, blood alcohol concentration (BAC)
red: serum tests, therapeutic drug monitoring
purple: complete blood count (CBC), A1c, erythrocyte sedimentation rate (ESR), sickle cell screen
orange: STAT serum chemistries
as a disclaimer, i’m not a phlebotomist or medical professional, so i’m going off of reading alone rather than firsthand experience 😅
#source material#evidence documents#tc documents#post-nbk#tcc tumblr#tccblr#eric columbine#dylan columbine#eric and dylan#tcc columbine#true cringe community#teeceecee
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(I’m sure you’re getting a lot of asks right now, so if this isn’t a fun avenue for you, feel free to pass on over.)
So, the Vax of it all.
As a person who really loved Vax’s portrayal and his arc, the end of C1 was powerful and poignant. Dalen’s Closet was the perfect cherry on top of a truly bittersweet ending - a really touching way to give the characters some final interactions and show that he didn’t feel trapped or tortured by his duties as a champion. It gave a lovely sense of closure - with the button put on it that Scanlan couldn’t even cast the spell again, so it really reinforced the idea that magic had natural rules and consequences to big asks.
And then C3.
Obviously Vax was always going to factor into this campaign (one of the cast described this as their Avengers Assemble plot, after all), but with the way Matt had him appear and knowing how the rest of the cast was going to react to it, it really seemed like this ending was inevitable.
Considering that she was the bait in the first place, Keyleth was always going to realize where Vax was, always going to draw in the de Rolos to save him, and being familiar with Matt’s DM style (as well as any of us can be) I have a hard time believing he was going to do all that and then steer them towards an ending that would just have left Vax back as a champion - or even dead. Possible, but seemed pretty unlikely. (forgive my ignorance, I’m sure this is exactly what people were saying about Molly’s resurrection too, I wasn’t in the fandom then, but that at least was a DICE roll that concluded on camera, no way around that)
But now I’m just… so confused by so many choices. When did Matt decide this? Did Liam agree? If this was going to be the ending, why did he have the Raven Queen explicitly say ’you have one more night on Exand-- JK, hang out as long as you like, go look up that girlfriend of yours!” Was it JUST so the Vaxleth reunion would be the last scene of the campaign? W h y a n y o f t h i s ? But-- none of those are things we can really know the answers to, of course.
So my REAL question is, how would you have liked to have seen Vax brought into this story? A defender of the Raven Queen, going as far as to oppose Bell’s Hells (gods, can you imagine what the fandom would have done)? Would you have liked him to appear at all?
Btw, I’ve loved following your blog through this campaign - these last handful of episodes, I’ve been checking in daily like it’s my morning paper. Even on the rare occasion I do find my opinion differs, I find your analysis so thorough, so thoughtful and always entertaining. Excited (and maybe a little wary…) to see what we’ll get in C4! I, uh... sorry for the ask-wall-of-text.
So I will admit, I thought, until early in the finale when it became clear this was just the equivalent of the flavorless pure sugar drink they give pregnant people to test glucose tolerance, that Vax would be freed from his duties and laid to rest. The part with champions serving as protectors of the gods' realms honestly hadn't occurred to me but you could have done it with Morrighan (still physically alive) taking on the mantle and Vax passing on to the afterlife. Because the thing was, Vax was dead, the Raven Queen said "you can be alive temporarily as a revenant," and then once his mission was over, he died. He was literally already dead. I also maintain it was not an inevitability from the Orb situation; obviously I have no fucking idea what Matt had in mind, clearly, but in a case where Predathos remains sealed, then the Vax situation remains as it was; and in a case where Predathos is freed and devours the gods I think he dies more horribly vs. a gentle and kind passing (or perhaps some hail Mary scenario where after Predathos has glutted itself and left, he can perform the rites of ascension himself).
I guess the short answer is I really don't think this was inevitable because I think the vast majority of the finale and no small part of the campaign was again just. things happening because they needed to happen to get to the ending where Bells Hells were ostensibly happy (it's not very fulfilling to have everything given to you without it meaning anything), but I can think of a number of ways to run any final scenario re: Predathos and the Raven Queen where Vax doesn't come back. That was a very specific choice, and it was, as many of us have pointed, an immensely stupid one that was utterly unnecessary.
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Feedism health - Diabetes Mellitus
TW: feederism, feedism reality, medical issues, explicitly explained medical conditions
Hi! This post gonna be long, it is a bit more medical again. We are feedists, right. Many of us are overweight or obese, some also have high blood pressure and many other comorbidities. We overeat a lot, stuffing so much sugar and fat into our bellis or bellies of our feedees so that we gain as much as we want. Therefore we are at HIGH risk of developing diabetes.
I am a student of physical therapy, NOT A DOCTOR. But I kinda feel the need to educate our community a bit 📚. So there are some facts (from medical literature which i study for my exames) about diabetes that I think should be commonly known. It may scare you, it may make you horny (we are weird, especially death feedists, hi guys 🖤), I just want you to know this, if you feel strong enough:
What it is and important vocabulary:
It is a disease caused by malfunction of insulin secretion from pancreas, or by insulin resistence of target tissue (such as muscles) or combination -> in every case you have a problem with insulin and glucose in your body.
There are two types, type I (DMI) that is caused by autoimunne reactions and you can not prevent it. And type II (DMII) which is hella important for our community because you can literally eat yourself into it. The more you over eat, the more you weight, the less you move, the higher the probability of developing that disease. This post is mainly about DMII.
Glycaemia = how much glucose (form of sugar) is in your blood
Norm is 3,9-5,5 mmol/l. After eating usually max 7,8 mmol/l
Hypoglycemia = less than 3,3 mmol/l
Hyperglycemia = over 11 mmol/l
Insulin causes that glucose goes from blood to your cells so it can become part of your metabolism. On the other hand there are hormones that causes the opposite - more sugar in your blood (by various mechanisms) and those are adrenaline, kortisol, growth hormone and glukagon.
How to get diagnosis of diabetes mellitus type II:
Doctor takes a sample of your blood plasma and tests its glycaemia:
If it is done in two different days and in both cases your glycaemia is over 11 mmol/l
OR if it is over 7 mmol/l after not eating for at least 8 hours*
OR if you undergo oral glucose tolerancy test and it is positive (you drink 75 g of glucose in 200 ml of water, wait for 2 hours and your glycaemia is over 11 mmol/l)
...in any of these cases they probably give you a diagnosis of Diabetes Mellitus. This apllies for my country in the middle of Europe, idk about your countries but it could be very similiar.
OR! I know that in USA they are also supposed to measure glucated hemoglobin (HbA1c) and diagnose you with DM if it is over 48 mmol/l.
*if your results are between 5,6 to 6,9 mmol/l, you are prediabetic which means that your body already suffers but you can stop it and go back to full health by changing your lifestyle (read more bellow).
Smyptoms of DMII:
I gonna explain them in "normal" language. You may have just some or all of them:
you are thirsty a lot, you drink a lot, you pee a lot, you are still thirsty though
there is glucose in your urine which definitely should not (you will not notice it, lab will)
you lose weight, you feel tired
your vission is blurred
you have some of acute or chronical complications (more bellow)
Complications of diabetes AKA what may happen to you:
They are usually devided into two groups - acute that actually can kill you pretty quickly and chronic that deteriorate your quality of life. (In the worst hypothetical case you can become blind, with neurological pain, amputated leg and close to a stroke that may kill your ability to move and speak. Nice, isnt it? 🤢) So lets get a closer look into that. These things happen when you do not treat your diabetes well or ignore it at all (for example continue in overeating and gaining even after being diagnosed):
Acute complications:
Hypoglycemia - may occur in patients that are treated with insulin (or glinids or derivates of sulfonylurey), also after drinking alcohol (even when you eat with it or dink juice etc). You do not have enough glucose in your blood so your brain cells become to die and in the worst case you will fall "asleep" (into coma) in the evening at party and will not wake up in the morning because you simply die. Your body fights hypoglycemie by making more glucose from storages in your liver, muscle and fat mass. Symptoms are anxiety, blurred vision, inability to concentrate but also seizure and coma.
Diabetic ketoacidosis - occurs in patients with DMI, very dangerous, also can lead to death. If you dont aplicate insulin when you should, you become hyperglycemic, dehydrated and your body catabolise fat into ketone bodies.
If you overdo it with your stuffing session while you are diabetic you may hypothetically cause yourself a hyperglycemic hyperosmolar coma. You are dehydrated, pee a lot, your blood pressure is very low, so low that it can reach hypovolemic shock and you faint. Also you kinda damage your kidneys.
Cronic complications:
Instability between insulin and glucose causes damage to your blood vessels and nerves which may result in
Retinopathy - you slowly lose your vision or even become completely blind
Nephropathy - if you ignore that you have diabetes, you damage your kidneys, it is asymptomatic for a long time but may result in need of dialysis or even transplantation if not treated.
Neuropathies - very common and very annoying. Harms your nerves - all kinds of nerves which means motor (problems with movement), sensoric (problems with feeling anything - touch, pressure, pain, cold, warmth, vibrations etc. and "problems" means you feel it less, more or differently so for example contant pain tha cannot be stopped) and autonomus (causes erectile dysfunction and decrease of libido, slows down motility in your stomach and gut, makes you feel sick, causes vomiting, constipation and diarrhoea and many more)
Diabetic foot - tissues in your leg are so damaged that it may literally start to rot and in the worst cases leads to amputations. This complication is related to many things from little injuries to ulcerations to gangrenes with bacterias that kinda eats your fat, muscles and bones.
Aterosclerosis - higher risk for ischemic heart disease (angina pectoris, heart attack), lower limb ischemia (may cause pulmonary embolism) and stroke.
Other problems such as: inflamation of thyroid gland, celiac disease (you can not eat anything with wheat, barley and others), diseases of skin, mycotic infections, urological infections etc.
Treatment:
I hope you are at least a bit frightened now... So what can we do when we are prediabetic or even diabetic? Three things!
Diet - if you are overweight or obese then it is weight-loss diet plus diet counting how many carbohydrates and fat you eat. Losing weight really works honestly.
Physical activity - helps so much!!! In general you should walk at least 10k steps per day and do some aerobic exercise for at least 30 minutes 3-4 times per week. And it should be on 75 % of your maximal heart rate (how to count that at home: "220 - your age = ideal load") plus ofc any sport you like. If you do have diabetes, be very careful about any injuries because it can lead to the diabetic foot.
Meds - DMI insulin for sure. DMII usually gets first oral antidiabetics and only in some cases insulin. But over all meds are only part of the treatment, it reallly does not work well without taking care of your food or exercising. You need to change your lifestyle if you wanna get better (I know that some of you don't).
______________________________________
I hope this post gave you something, tought you something new and you know the risks of our kink better now. I do not want to tell you not to do it - I have that kink too and love it, gonna continue gaining. Just be aware about the consequences 💕
Uffff... that was long and complicated, I actually did my research for that and spent few hours making that post 😂. But it is still possible that i did some mistakes, did not understand something well etc - I am NOT a doctor, please believe more your health proffesionals than me, thank you. Im sorry if anything does not make sense or if I use some words in a strange way - english is not my native language and I do not know many medical terms and phrases, know them only in czech and latin so I translate it somehow based on that XD
Enjoy the candy that our kink brings to our life and stay as healthy as you wish 💕💕💕
~ Your Tessie
#feedism.#feederism.#feedism health#diabetes#feedism consequences#kink education#feedism education#feedee girl
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I’m so exhausted with this, whatever it is. Is this normal? Is it just me? Is it because I had Covid before? Is it just because I’m weak and pathetic?
It’s really getting to me; I can’t pretend otherwise. I can barely do anything. My head aches constantly and I feel woozy. I’m weak, wobbly, have no energy. There hasn’t been a good day in weeks.
Last few days I just want to cry all the time; there’s no relief. Worst of all, I feel guilty for feeling this way, like it’s all my fault and I’m doing something wrong. All I can do is sleep, or sit up in bed like a woman in a Victorian novel, pleading for liquids. I don’t know how to feel or what to do. I felt shaky all last night, like there were bees and wind inside me.
Doctor on Thursday morning. Glucose tolerance test, anatomy scan, checkup. Partner will be there to advocate for me; I’m too tired to even know what to say anymore.
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Reader Eliah sent me the following email:
I’ve seen you write about how the idea of “ob*sity”* as a disease is problematic which is I think why I started noticing that in all the articles about these new diet drugs it seems like it says at least once that “ob*sity is a disease like asthma or type 2 diabetes” I thought you might have some insight into the concept in general and why this specific phrasing is being used.
This is a great question. This specific phrase “ob*sity is a chronic disease like asthma or type 2 diabetes” is indeed making the rounds. I’ll start by talking about the veracity of the claim, and then answer the question as to why people are saying it.
Before I dig in, I want to be clear that there is absolutely no shame in having a disease or diagnosis of any kind, this is about the intentional misapplication of the concept and the damage it does.
First let’s answer the basic question: Is “ob*sity” a chronic disease like asthma or type 2 diabetes? In order to get to the bottom of this, let’s examine each diagnosis in turn.
In order to be diagnosed with asthma, there has to be documentation of signs or symptoms of airflow obstruction, reversibility of obstruction (that the signs or symptoms decrease with asthma therapy,) and no clinical suspicion of an alternative diagnosis.
In order to be diagnosed with type 2 diabetes one of the following thresholds must be met (typically with at least one repetition): an A1C of greater than or equal to 6.5%, fasting blood glucose of greater than or equal to 126 mg/dl, a two-hour blood glucose of greater than or equal to 200 mg/dl on an Oral Glucose Tolerance Test, or blood glucose of greater than or equal to 200 mg/dl on a Random Plasma Glucose Test.
In order to be “diagnosed” as “ob*se” one’s weight in pounds times 703 divided by their height in inches squared has to be 30.0 or higher. There is no shared symptomology among this group of people, it includes people with various health diagnoses, people without any health diagnoses, and with widely varying cardiometabolic health, body composition, etc. Literally, the only thing this group has in common is a similar ratio of weight and height (and being “diagnosed” using a deeply problematic math equation.)
When you take into account the “class” system (ie: class 1, class 2, and class 3 ob*sity) the folly of this becomes even more apparent. Consider that, for class 1 ob*sity, 1 pound, or 1 inch in height canbe the only difference between someone who is “diagnosed” with “ob*sity” and someone who is not, again with no shared symptomology or cardiometabolic profile. Even more ridiculous, while class 1 and 2 each encompass a 4.9 point BMI spread, class 3 is defined as a BMI of 40 to…infinity. That does not have the ring of sound science.
Then there is the issue of treatment. For both asthma and type 2 diabetes, treatments focus on managing the common symptomology. With “ob*sity” the “treatment” is focused on making the patient look different by changing their size. Setting aside that the “treatment” almost never works and has considerable risks, no matter what size someone ends up at, their actual health status may be exactly the same (or worse) and there will be people of that new size who have the same (and different) actual health issues.
So the answer to the question of whether ob*sity is a disease like asthma and type 2 diabetes is: No, it absolutely is not. This is drawn into sharp relief by the fact that there isn’t even a clear, consistent, scientific definition among major health organizations.
This is pretty clear cut, so why are people (including doctors) still spouting this nonsense? In a word (or four): the weight loss industry. The classification of simply existing in a higher-weight body as a “disease” is the holy grail for them. It increases their market to every fat person for the entire time that they are alive. They’ve been pushing this (both blatantly and surreptitiously,) pouring money into the effort for many years. I’ve been writing and speaking about this since 2009, and there are many people who have been doing it far longer (including since before I was born.) It’s actually one of the first things I wrote about when I launched this newsletter. So we’ve heard them try to claim that being fat is a “chronic lifelong health condition.” The addition of this comparison to type 2 diabetes and asthma seems to be specifically in the service of selling diet drugs like Wegovy.
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12/03/25
Had a small break, but now I'm back on track.
Topics covered today :-
Cerebellum
Examination of nervous system
Ascending tracts
Functional areas of cerebrum
Oral glucose tolerance test
#medical school#medicine#med studyblr#med student#med school#med#studying#student#university student#studyblr#study motivation#study blog#study#my thoughts#spilled ink#writing#light academia#spilled words#spilled thoughts#spilled writing#study journal
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https://twikkers.nl/blogs/221429/Glucose-Tolerance-Test-Market-Analysis-Size-Share-and-Forecast-2031

Glucose Tolerance Test Market Analysis, Size, Share, and Forecast 2031
#Glucose Tolerance Test Market#Glucose Tolerance Test Market Scope#Glucose Tolerance Test Market Report
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I’m about to go for my glucose tolerance test at the hospital where I will find out if I have diabetes in my pregnancy and I’ve been fasting for 12 hours now and let me tell you I’m struggling and my baby is kicking me like where is the food?
And I’ll have to drink a disgusting drink that I hear can make you feel really sick and I’m already feeling kinda sick. Yesterday I had to go home sick from work because I was so nauseous and felt so faint and that was WITH breakfast.
I’m hungry and nervous.
I fear the only thing that will get me through is dirty Starker thoughts 🫣
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