#darr a cell

[miss-sheepy]

Darr headshot 🐲

[tsreviews]

Ignis Review: Transform Your Phone into a $563/Day Machine!

Ignis is an AI-driven solution designed to turn any Facebook account into a passive income source. Here’s how it works:

Facebook Integration: Ignis use the force of the Facebook application. Indeed, the equivalent application you use to interface with companions and offer feline recordings can now turn into a lucrative machine.

Content Enhancement: Ignis improves your Facebook profile by helping content, pictures, and recordings. It makes your profile really captivating and powerful, drawing in designated rush hour gridlock.

Monetization:  Ignis doesn't stop at commitment. It likewise gives adaptation strategies, changing over guests into paying clients. Envision acquiring up to $563 each day straightforwardly from your cell phone!

Ignis App Overview: 

The Ignis App claims to revolutionize income generation by transforming your everyday smartphone into a powerful profit-making machine. Here are the key points:

Facebook Monetization: Ignis is intended to transform any Facebook account into a recurring, automated revenue source, possibly procuring up to $563 each day.

Boosted Engagement: It improves your Facebook profile by upgrading content, pictures, and recordings, making them seriously captivating and compelling.

No Complex Setup: You needn't bother with a site or unpredictable arrangement. Ignis uses the force of the Facebook application itself.

Step-by-Step Training: Even amateurs can begin with Ignis utilizing its exhaustive preparation program.

Creator: Billy Darr

Product: IGNIS

Date Of Launch: 2024-Aug-11

Time Of Launch: 11:00 EDT

Launch Price: $12.95

Refund: YES, 30 Days Money-Back Guarantee

Product Type: Software (online)

Bonus: Hug, Bonuses

Official Website: Click Here

Support: Effective Response

Discount: >> GET THE BEST DISCOUNT HERE <<

Recommended: Highly Recommended

Skill Level Needed: All Levels

Coupon Codes: No coupon

Ignis Review: Benefits

Faceless Channels In 60 Seconds

Built-In A.I Powered Video Creator…

100+ Templates To Choose From…

Intuitive Drag-and-Drop Interface…

Works With Voice Prompts Or Keywords…

Hundreds Of Stock Assets Included For Free…

Newbie Friendly Interface…

App Works On All Popular Devices…

All Major 3rd Party Integrations Supported.

Automatic Facebook™ Channel Creation…

Automated AiTraffic Feature Built-In …

Built-In Monetisation Included…

OpenAI & ChatGPT4 Integration…

1-Click Facebook™ Keyword Finder…

Auto Like/Comment Campaign…

No Monthly Fees…

Share YouTube Videos To 100+ Sources…

Biz-In-A-Box Commercial Licence Included…

>>Grab a copy now to discover the remaining awesome features

[princeofthecactus]

October Writing Prompts—Day Six—October 6th 2021:

Prompt: Alone

This ended up being much shorter than my others so far, but It's one of my favourites, mostly because I just love writing creepy nursery rhyme themed things. So, if you like FIRE!!!!, casual jailbreaks, and ominous nursery rhymes, you might enjoy reading this!

Thanks once again to the marvellous @mythyk-art for this prompt list!

One phoenix, two phoenix, someone’s gonna burn.

Mummy had a little cat and now it’s in an urn.

Darr had a cell to himself—it was the third they’d put him in in the last few days, and it was the worst of them all, damp and dingy, mould growing in the corners of the dark stone walls, broken shackles on the floor marred by rust and stained with blood. His wrists were bound in new chains, made out of some sort of sleek black metal that was like ice against his skin, cold metal chafing his wrists and where normal cuffs would be rubbing them red-raw, these just left his blood frozen in his veins.

Both his ankles were shackled together with the same metal, and a long chain linked them to another shackle around his neck—it was too short to let him stand to his full height without falling face-first onto the cold stone floor, and it brushed against the bare skin of his back every time he moved, sending shivers running up his spine.

They’d certainly learnt their lesson.

He’d shared his first cell with two other men, so it had been large enough for him to pace around, walk in the same circles again and again until the soles of his feet begged for relief and sweat poured down him, stuck his tousled hair to his forehead like blood.

His fellow prisoners had watched him, bemused, drag himself up at the earliest hours of the morning just to traipse the same beaten track around the cell. Their eyes had gleamed with curiosity and mirth—like that had any place in a jail cell—as the new guy worked himself to the bone, running back and forth, diving into push-up after push-up, rubbing his hands together until he’d worked up enough heat to start a fire in his fingertips.

That was his Witching. Fire, heat, and everything that came with it: the phoenix was reborn in the flames, the guilty burned in it. Fire was the ultimate bringer of justice, retribution, peace. But the others feared it.

They didn’t understand the beauty in the way it danced, or the magic in the way it grew into something bigger, better, stronger—they only feared what it could let him do, and they punished him for every campfire he created as a child, every marshmallow he placed on his palm and roasted until it was too crisp to eat, caned him for every childish indulgence until the very thought of his power brought tears to his eyes.

That was what the Institution for Youth with Dangerous Witchings was for. Bringing pain, and suffering, and injustice. And what was unjust had to be burnt.

Daddy had a real mean dog, he thought it big and strong,

But it burnt down, all to the ground, and now it’s dead and gone.

Nothing made fire happier than swallowing those who would stand in its way, proving the true strength of its heat.

After his first escape attempt, they had him in a smaller cell, all on his own, so there was no room to pace around—there hadn’t even been enough room to lie down and do push-ups. He just had to sit and sit and sit, make do with rocking back and forth, back and forth, again and again, and again, a swinging pendulum on a clock counting down until the fire had its justice.

This cell had been closer to the courtyard—the sun beat against its walls, and he rested his back against them, let it fill him up like a mug overflowing. Overflowing until there was nowhere for the fire to go but out, out, out.

Now, he was so deep in the dungeons he’d never see the light of day again.

Like that would stop him.

One phoenix, two phoenix, someone’s gonna burn,

Mummy’s down, and Daddy’s gone, and now it’s your turn.

The grate on the cell door screeched open, letting a small sliver of candlelight fall into the room.

The problem with having him so deep in the castle was that the Guards needed to see to deliver food. So, when the grate slid open, and the plate of food—cold, of course, frozen for hours before it was brought to him—slotted in, the Guard needed a lantern to see.

Darr breathed in, feeling the fire fill his lungs.

Now it’s your turn.

[oh-atlas]

7, 17, 27, and 37 for darr and someone else of your choice! (second time's the charm lol

thank you!! gunna throw maxarra in / development asks!

How do they physically engage with other people, inanimate objects, and their environment? What causes the differences between these?

DARR tends to be fairly gentle with other people. She’s very big in comparison to humans and takes care to remember that. In fact, she’s very gentle with the way she interacts in general. Firm but careful to preserve the beauty and integrity of something. When with Goliath, because in this case, she’s smaller, she exerts her physicality more to prove herself and to avoid coming off as weak.

MAX is harsh. she doesn’t have much respect for things and tends to be quite aggressive in her treatment of existence. She doesn’t have much regard for the environment and is the type to push past people without any notion to apology. She tries to take up space.

Are they more shaped by nature or nurture — who they are, or what has happened to them? How have these shaped who they’ve become as a person?

DARR is primarily shaped by nature. She’s always had a fairly solid sense of identity and purpose in life and wants to live up to what is supposed to be good and right in her society. Since she woke up and found out she’s missing like, a month of her life and now has a magic tattoo she’s set out to figure out what happened in that blank spot and that’s probably going to shift how she feels about herself.

MAX — nurture all the way. she could’ve been such a different person if her parents hadn’t died and she wasn’t taken in, abused and raised to be the next chief. She’s got such bad coping mechanisms and such unhealthy emotional releases but she doesn’t know any better! She’s constantly in pain! She’s had to become something cruel and volatile to survive.

Me @ My Own Oc: Get Better Soon. Or Else.

What do they strongly like and dislike, in any category? Why?

DARR — Strongly dislikes the mage hunters (who i need to give an official organization name to whoops, was going to do inquisitors but like also feel like that’s overdone but that’s a brain cell and topic for another post) because they won’t leave her the alone! she didn’t choose to have a magic tattoo appear on her back! let her rest! She really likes the mountains and nature. She likes the feeling of being alone in the woods but it’s not lonely you know? She also likes Kasia. Because she’s pretty and nice and very fun to talk to.

MAX — Strongly dislikes most things and it’s really just to lash out. Or there are things that remind her of her childhood and better times that she just, can’t handle. She likes mystera, it calms her nerves but she also likes it too

Have they had any special education or training that colors their means of learning about or understanding the world? Conversely, do they lack some kind of education considered essential in their world? What kind of impact has this addition or lack had on them?

DARR — so Darr is a persistence hunter by trade. Shee’s very good at tracking, fighting with various weaponry and the process of hunting including cooking and tanning and butchering. She’s very patient because hunts can take days and even weeks and is just, very thorough with her work. It’s hard work and you need to take care in using your resources. Not to be a Goliath Cliche but I haven’t decided if Goliath have a written language so, can Darr read? Much less read Common? Unclear. She also doesn’t get how money works very well because Goliath primarily operate off the barter system. 

MAX — Max has a background in herbal medicine that’s pretty unique but has also been sort of buried by getting the shit beaten out of her until she could beat it up better. 

[niladri123]

Magic Mobile Traffic App Review (Billy Darr) - AutoPilot Buyer Traffic In Seconds.

Magic is the first mobile traffic app on the online marketplace. All you have to do is click a button to obtain unlimited free buyer traffic. Set up your affiliate networks, affiliate offers, and free traffic to generate sales, that simple it is.

By using the Magic App you can drive traffic to any links by only using your smartphone.

>>Click Here to visit Magic Traffic App Official Page and also check my Huge Bonus packages<<

There are only simple 2 steps to start with Magic App:

#1: Purchase the Magic app.

#2: In less than 60 seconds, push the magic button to send free buyer traffic through up to 5.2 Billion mobile phones to any website.

Vendor:

Company name – Digital page designs.

Name – Billy Darr, Finn Goswami, Justin Opay.

Location – London, U.K.

Few previous good products created by this vendor are Aura, Orion – Dares You To Send 1 More Email, Krown – Deal Of The Week, Xtreme – Send 1 More Email, Hive, etc.

It works on most of the popular platforms like:

Warrior Plus

JVzoo

ClickBank

PayDotcom

E-bay

Amazon

Maxbounty

Shopify

What exactly will you find inside Magic App?

The Magic Software:

Newbie-Friendly ‘Mobile' Traffic Software Gets You Free Traffic From A 5.2 Billion Buyer Source In 60 Seconds Or Less.

Complete video tutorial step by step:

They will teach you exactly how to convert the free visitors to generate into a steady flow of sales.

$100 a day case study:

There is also included a link to a fresh new case study that shows you how they made $100+ utilizing this software and the power of cell phones.

24x7 Excellent support:

If you have any questions or need any assistance, the world-class support team will be glad to assist you.

Quick Start Guide:

Don't worry if you don't like watching videos, they are covered with a quick start guide that simply teaches you how to turn on Magic and start getting visitors and purchases in seconds.

Customer Only Q & A Call:

A special live Q&A call for customers exclusively to guarantee that everyone can utilize Magic to obtain results quickly. The call will be recorded.

>>Click Here to visit Magic Traffic App Official Page and also check my Huge Bonus packages<<

Pros:

Get instant access to 5.2 Billion buyer traffic.

Complete autopilot system.

No need for any technical skills.

Get faster results.

No need to create videos.

Complete tutorial inside.

Get high-quality visitors.

Newbie-friendly method.

No recurring cost, one-time payment only.

No email list, no website is required.

Works for any niche.

Can operate on any device.

Cons:

No learning curves, there is no opportunity to learn new things.

Various upsells are there.

Who needs the Magic App?

Affiliate and CPA Marketers.

Product Creators.

List Builders.

​Shopify Store Owners.

Local Business Owners.

​Influencers & Coaches.

Price details:

Front End: Magic - $17

Magic The World's First Mobile Traffic App provides you with an unlimited supply of free buyer traffic.

Upsell 1: Magic - Unlimited Edition - $39

Multiple Templates, limitless usage, unlimited traffic, and infinite sales are all included with the Unlimited Edition of Magic. There are also more tutorials available.

Upsell 2: Magic - Done-For-You Edition - $197

Our objective with this upgrade is to provide your clients with Done-For-You traffic and sales.

Upsell 3:Magic - Automated Edition - $39

Your clients will be able to use magic to activate all of the automation systems.

Upsell 4: Magic - DFY Templates Club - $39

You will receive 200 video templates that they can customize and utilize with magic.

Upsell 5:Magic - 50x Click & Go Campaigns - $39

They will provide your customer with 50 proven TikTok campaigns to copy and paste in order to generate affiliate sales on TikTok.

5 Bonuses Worth $3,985:

#1: never-seen-before $0 To $1K A Day In 30 Days!

#2: 1st Sale In 60 Seconds

#3: Passive Income System

#4: $1,000,000 With Affiliate Offers

#5: Sales 'Same Day' System

Is this really worth money? Final conclusion:

This is a new innovative membership software that combines remarkable user-friendliness with unrestricted power and flexibility

Billy Darr's Magic App is a game-changer that automates affiliate marketing from start to finish. It removes the time-consuming, costly, and difficult procedures required in traditional methods. As a result, anyone can get up and running, making money faster than ever before.

Also, purchasing this is completely risk-free as there is a 365 days money-back guarantee. If for any reason you are not happy with the software just send a mail to them, they will refund you the full amount without any question asked.

>>Click Here to visit Magic Traffic App Official Page and also check my Huge Bonus packages<<

[peaceandprosperty]

Faqeeri Say Darr

#KingdomofPakistan #PeaceandProsperity #Lodhi 

 Faqeeri Say Darr By Faisal Zafar Khan Lodhi (Faisal khan  Dave Kahn) 

Faisal Zafar Khan Lodhi Cell: 518-986-6027 Fax: 1-800-794-9560 E-mail: naazfoods@gmail.com 2535 Broadway Schenectady, NY 12306, USA  

 Also follow Us on Twitter /Facebook https://twitter.com/peaceprosperit3 https://www.facebook.com/Peace-Prospe...

[theliberaltony]

Donald Trump (2017)

President Trump addresses a joint session of Congress on Feb. 28, 2017. (Photo: Pablo Martinez Monsivais, AP)

Approval rating: 43%*

Nominees formally submitted/confirmed by Senate: 24/22*

Major successes: Neil Gorsuch confirmed for Supreme Court; some Obama-era regulations repealed

Major setbacks: Proposal to repeal and replace the Affordable Care Act withdrawn from House; immigration orders blocked by federal courts; national security adviser Michael Flynn forced to resign

Of note: FBI confirmed investigation into whether Trump associates colluded with Russian meddling in election

Barack Obama (2009)

President Barack Obama is sworn in on Jan. 20, 2009. (Photo: Tim Sloan, AFP/Getty Images)

Approval rating: 65%

Nominees formally submitted/confirmed by Senate: 190/69

Major successes: Stimulus bill passed; children's health care expanded; equal-pay protections bolstered; federal ban on embryonic stem-cell research lifted

Major setbacks: Nominee for key role of Health and Human Services secretary, Tom Daschle, forced to withdraw

Of note: Stock market bottomed out in March, a sign that the end of the worst economic downturn since the Great Depression was in sight

George W. Bush (2001)

President Bush speaks to newly sworn-in White House staff members in an East Room ceremony on Jan. 22, 2001. (Photo: H. Darr Beiser, USA TODAY)

Approval rating: 62%

Nominees formally submitted/confirmed by Senate: 85/35

Major successes: House passed tax proposal, eventually signed in June, to slash income tax rates

Major setbacks: Failed to act on a blue-ribbon commission report urging changes in homeland security or on warning signs before the terror attacks on New York and Washington that would follow in September

Of note: U.S. spy plane flying over the South China Sea clipped by Chinese fighter jet and forced to land on Chinese soil

Bill Clinton (1993)

President Bill Clinton is sworn in on Jan. 20, 1993. (Photo: Ed Reinke, AP)

Approval rating: 55%

Nominees formally submitted/confirmed by Senate: 176/49

Major successes: Family and Medical Leave Act signed

Major setbacks: Furors over gays in the military, firing of White House travel office staffers

Of note: Hillary Rodham Clinton put in charge of signature health care overhaul, which eventually would fail.

George H.W. Bush (1989)

President George H.W. Bush is sworn into office on Jan. 20, 1989. (Photo: Bob Daugherty, AP)

Approval rating: 56%

Nominees formally submitted/confirmed by Senate: 95/50

Major successes: Submitted plan to bail out troubled savings and loans, eventually signed in August

Major setbacks: Nominee for Defense secretary, Texas Sen. John Tower, rejected by Senate

Of note:  Worst oil spill on U.S. territory in history when Exxon Valdez supertanker ran aground in Alaska

Ronald Reagan (1981)

President Ronald Reagan in the Oval Office on Jan. 20, 1981. (Photo: Barry Thumma, AP)

Approval rating: 68%

Nominees formally submitted/confirmed by Senate: 128/80

Major successes: Proposed major cuts in taxes and domestic spending and an increase in military spending; Iran released U.S. hostages as he was inaugurated

Major setbacks: Fragility in the economy, which would head into recession in July

Of note: Survived assassination attempt by John Hinckley Jr.

Sources: Gallup Poll; Partnership for Public Service; USA TODAY research by Susan Page

*As of April 21

[nukteler]

Ed-Dârr Esmasının Zikri, Faziletleri ve Faydaları

Ed-Dârr Esmasının Zikri, Faziletleri ve Faydaları

Ed-Dârr

Elem ve zarar verecek şeyleri yaratan, hüsrana uğratan, dilediğini zarara uğratan, felaket, keder ve şiddet veren anlamlarına gelmektedir.

Ed-Dârr esmasının Ebced değeri ve zikir adedi 1001 adettir. Zikir saati Merih, günü Salı’dır.

Ed-Dârr ism-i şerifi Cenab-ı Hakk’ın darlığa uğratan, darlık veren, hikmeti gereği sıkıntılar, musibetler, elem ve zarar verici şeyleri yaratan esmasıdır.

Ed-D…

[movie-magic]

Netflix is removing a huge number of movies and TV shows this month

This month, Netflix is taking down a large number of titles.

The streaming service doesn’t publicise the films and TV show it’s removing each month.

But should you search for a title that happens to be leaving over the coming weeks, Netflix will let you know that you only have a short space of time to finish watching it before it vanishes.

Below, we have collated the full list of everything leaving Netflix over the next 30 days:

Angus, Thongs and Perfect Snogging

Annabelle

BA Pass

Bad Teacher

Barnyard

Beach Rats

Black Man White Skin

Bloodsport

Christmas Made to Order

Christmas Wonderland

Darr Sabko Lagta Hai

Death Wish 3

Dharmakshetra

Dreamgirls

Everything You Always Wanted to Know About Sex* (*But Were Afraid to Ask)

Friday

Galaxy of Terror

Galaxy Quest

Get Hard

Hearts in Atlantis

The Hole in the Ground

Hotel for Dogs

I Am Ali

Inception

Invaders from Mars

The Island of Dr. Moreau: Director’s Cut

Jack Reacher: Never Go Back

Jose Jose, el principe de la cancion

Lemony Snicket’s A Series of Unfortunate Events

Little Giants

Los heroes del Mal

Love Is a Story

Mean Girls 2

Menace II Society

Mid90s

A Midsummer Night’s Sex Comedy

Mission: Impossible – Ghost Protocol

Mountain

My Christmas Inn

Natsamrat – Asa Nat Hone Nahi

No Country for Old Men

Outnumbered seres one to four

Pan

A Private War

Raja Rsaoi Aur Anya Kahaniyan

Ronin

Satan & Adam

#Selfie

#Selfie 69

Species

Speed Kills

Spy Time

Stories by Rabindranath Tagore

Support Your Local Sheriff

10 Cloverfield Lane

The Titan Games season one

True Grit (2010)

Two Weeks

Unfinished Business

Wishmaster

Zombeavers

7 June:

Alpha and Omega

Dirt

Searching for Bobby Fischer

8 June

A New Capitalism

10 June:

Black Butterfly

Black Snow

Destination Wedding

LEGO Friends: Masters of Spinjitsu: Happy Birthday to You!

11 June:

LEGO Friends: The Power of Friendship

Q Ball

12 June:

Addicted to Life

Don’t Crack Under Pressure

Don’t Crack Under Pressure 2

Don’t Crack Under Pressure 3

Magnetic

13 June:

The Ant’s Scream

The Cell

Convict

Filmflam

The Right One

Savage Raghda

Welcome to Marwen

15 June:

Five Feet Apart

Humsafar

The Karate Kid (1984)

LEGO House – Home of the Brick

Memory Games

Prince Avalanche

Red Corner

Red Dwarf series one to eight

Zindagi Gulzar Hai season one

16 June:

The Grandmaster

18 June:

Antariksha Ke Rakhwale

Bob Lazar: Area 51 & Flying Saucers

Club Friday The Series 7

Dragonkala Ka Rahasya

Love Rhythms – Accidental Daddy

Samudri Lootere

Super Bheem Bana Vajraveer

The Underwear

19 June:

Jurassic World

20 June:

American Dreamer

An Upper Egyptian (Wahid Saeiday)

Angel of Mine

Bewildered Bolbol (Bolbol Hayran)

Bridge of Spies

The Chord

Mary, Queen of Scots

Ocean’s 8

21 June:

Mock the Week multiple series

23 June:

Cooking on High season one

- Independent

[hudsonespie]

MSC Continues To Invest In Low-Carbon Future: Operating Modern, Green Fleet

Improving the environmental performance of the container shipping supply chain is a common goal shared by shipping lines, shippers, freight forwarders and non-vessel operating common carriers, as well as governments, regulators, and the public around the world.

The UN International Maritime Organization (IMO) requires that from 1 January 2020, Sulphur content in the fuel used for international shipping must be limited globally to 0.5%, compared with the current standard of 3.5%, in order to minimize emissions of Sulphur oxides from ships.

MSC is committed to reducing the environmental footprint of global supply chains and keeps implementing concrete plans to modernise its green and efficient fleet via the largest container shipping investment program in the industry.

Image Credits: msc.com

Its fleet improvement program has resulted in a 13% reduction in CO2 emissions per transport work* in 2015-18. The company also completed a massive retrofit program in its existing fleet with the installation of Exhaust Gas Cleaning Systems (ECGS) to reduce emissions of sulphur oxides and other pollutants. In its compliance strategy, MSC will combine the use of ECGS, conventional low-sulphur fuel and biofuels.

Furthermore, the next generation of 23,000+ TEU newbuilds – led by MSC Gülsün, the largest container ship in the world – has introduced a new class of sustainable container shipping, with the lowest carbon footprint by design, at 7.49 grams of CO2 emissions to move 1 ton of cargo 1 nautical mile.

“MSC is on a well-defined pathway to meet the 2030 IMO level of ambition for CO2 emissions intensity reduction. The great challenge which remains for container shipping this century is how to decarbonise and meet the UN IMO’s future emissions goals beyond 2030. While we are fully supporting these more distant targets, this will not be achievable without some major breakthroughs in fuel and propulsion technologies,” said Bud Darr, Executive Vice President, Maritime Policy & Government Affairs, MSC Group.

While recent improvements have depended largely on better-performing engines, more efficient propeller and rudder designs and technologies to reduce hull friction, MSC is actively studying the potential of new alternative fuel sources. The company is engaging with potential vendors to investigate solutions related to biofuel blends, hydrogen fuel cells, complementary battery power and, possibly, wind and solar, as part of a long road of discovery in relation to future policy goals.

MSC Samar (built 2019), 23 756 TEU | Image Credits: msc.com

Improving the environmental performance of the container shipping supply chain is a common goal shared by shipping lines, shippers, freight forwarders and non-vessel operating common carriers, as well as governments, regulators, and the public around the world.

The UN International Maritime Organization (IMO) requires that from 1 January 2020, Sulphur content in the fuel used for international shipping must be limited globally to 0.5%, compared with the current standard of 3.5%, in order to minimize emissions of Sulphur oxides from ships.

MSC is committed to reducing the environmental footprint of global supply chains and keeps implementing concrete plans to modernise its green and efficient fleet via the largest container shipping investment program in the industry.

Its fleet improvement program has resulted in a 13% reduction in CO2 emissions per transport work* in 2015-18. The company also completed a massive retrofit program in its existing fleet with the installation of Exhaust Gas Cleaning Systems (ECGS) to reduce emissions of sulphur oxides and other pollutants. In its compliance strategy, MSC will combine the use of ECGS, conventional low-sulphur fuel and biofuels.

Furthermore, the next generation of 23,000+ TEU newbuilds – led by MSC Gülsün, the largest container ship in the world – has introduced a new class of sustainable container shipping, with the lowest carbon footprint by design, at 7.49 grams of CO2 emissions to move 1 ton of cargo 1 nautical mile.

“MSC is on a well-defined pathway to meet the 2030 IMO level of ambition for CO2 emissions intensity reduction. The great challenge which remains for container shipping this century is how to decarbonise and meet the UN IMO’s future emissions goals beyond 2030. While we are fully supporting these more distant targets, this will not be achievable without some major breakthroughs in fuel and propulsion technologies,” said Bud Darr, Executive Vice President, Maritime Policy & Government Affairs, MSC Group.

While recent improvements have depended largely on better-performing engines, more efficient propeller and rudder designs and technologies to reduce hull friction, MSC is actively studying the potential of new alternative fuel sources. The company is engaging with potential vendors to investigate solutions related to biofuel blends, hydrogen fuel cells, complementary battery power and, possibly, wind and solar, as part of a long road of discovery in relation to future policy goals.

Sustainability Report

The latest edition of MSC’s Sustainability Report details some of the company’s activities around preserving the environment, as well as social inclusion, health and safety and business ethics. To download a copy please go to msc.com/sustainability.

Reference: msc.com

Report an Error

from Storage Containers https://www.marineinsight.com/shipping-news/msc-continues-to-invest-in-low-carbon-future-operating-modern-green-fleet/ via http://www.rssmix.com/

[maritimemanual]

MSC Alongside CMA CGM, Hapag-Lloyd Rules Out Arctic Exploration On Environmental Concerns

The leader in transportation and logistics in the world, MSC Mediterranean Shipping Company has made up its mind to not use the Arctic which is shorter route between the Northern Europe and Asia and has decided to focus on steps that can improve the environment performance on the present routes of global trade.

Many shipping lines have been testing the Northern Sea Route that lies completely in the Arctic waters thereby, taking advantage of the ice that is melting there due to global warming. The MSC has decided that the 21 million vessels that transport annually can be move around the world without making use of the Arctic Corridor.

“As a responsible company with a longstanding nautical heritage and passion for the sea, MSC finds the disappearance of Arctic ice to be profoundly disturbing. Every drop in the oceans is precious and our industry should focus its efforts on limiting environmental emissions and protecting the marine environment across existing trade routes,” said Diego Aponte, President & CEO, MSC Group.

The quality of the air and the natural marine habitat of the Arctic can be damaged due to the shipping traffic and thus, MSC does not want to take that risk.

The lowest carbon footprint by design

The decision of the company to not use the Northern Sea Route complements the strategic sustainability approach of MSC.

MSC retrofitted more than 250 of its vessels with the latest available green technology thereby, reducing the emission of 2 million tons of CO2 annually. Moreover, the largest container ship in the world is the most recent vessel added to the company’s fleet that was led by MSC Gülsün. It is a new class sustainable ship that has the lowest carbon footprint by design of 7.49 grams emission of CO2 for moving 1 ton of cargo 1 nautical mile.

The fleet improvement Program by MSC has helped to the CO2 emission by 13% for every transport work in 2015-18. This will help the maritime sector move towards the 2030 CO2 target of the United Nations International Maritime Organization. The company is committed to work continuously towards the adoption of plans to update it’s fleet to being green and efficient using the industry’s largest container ship investment program.

“MSC is on a well-defined pathway to meet the 2030 IMO level of ambition for CO2 emissions intensity reduction. The great challenge which remains for container shipping this century is how to decarbonise and meet the UN IMO’s future emissions goals beyond 2030. While we are fully supporting these more distant targets, this will not be achievable without some major breakthroughs in fuel and propulsion technologies,” said Bud Darr, Executive Vice President, Maritime Policy & Government Affairs, MSC Group.

MSC is also working to study the potential of the new alternative fuels. It is joining hands will probable vendors to find out solutions that relate to biofuel blends, hydrogen fuel cells, complementary battery power and, potentially, wind and solar.

  Reference: msc

from WordPress https://www.maritimemanual.com/msc-alongside-cma-cgm-hapag-lloyd-rules-arctic-exploration-environmental-concerns/

[adultstories4u]

Aaakhir woh waqt aahi gaya jisska intezaar tha. Vicky ki plan mazedar tha. To hua yeh!!

MLA ki watchman ne raat ko Rathod ko phone karke bataya ke MLA parivaar sameth kisi party mein jaa rahe hein aur sirf secutity wale aur wo maujood hein ghar ke compound mein. Watchman ne kaha ke security walon se ussne baat kar liya hai aur weh sab saath hai usske kyun ke weh log bhi tang aagaye hein uss MLA se. Phir Rathod ne Vicky ko bataya ke wo aur Khan jaa rahe hein MLA ke ghar ko.

Vicky turant apne 5 nakaabposh aadmiyon ke saath police station ke paas pohuncha unn donon ke jaane ke baad. Ek time bomb lagaya baahar se uss cell mein mein jiss mein Judish quaid tha.

Assal mein Khan se baat ho chuki thi ke Jugdish ko uss waqt uss cell se nikaal kar dusri cell mein daal diya jae nahin to woh ghayal ho jaega, aur dusri cell ki chaabhi Khan ne Vicky ko pehle se de diya tha.

To police station ke baahar se uss cell par time bomb lagaya Vicky ne jiss mein har roz Jugdish rehta tha, deewaar phata, ek darwaze jeisi entry hui, ek sub inspector aur ekaat hawaldar the jo hakbakaaate baahar dawde aaye aur baaki ke 5 nakaabposh aadmiyon ne sab ko hands up par rok liya bandook taane hue unn sab par. Pehle se Khan ne Vicky ko bata diya tha ke kitne guards mawjood rahenge police station mein uss waqt. Sirf ek hawaldaar andar reh gaya tha darr ke mare woh baahar nahin nikla, aur Vicky bhi nakaab pehne uss bomb se ki gayi raaste se andar ghussa aur Jugdish ki cell kholkar ussko baahar chalne ko kaha yeh kehte hue, “mujhe aap ke MLA chacha ne bheja hai aap ko yahan se nikaalne ke liye, chaliye jaldi waqt kum hai……” Jo hawaldaar andar tha uss ne sab suna ke MLA ke aadmi aaye the jugdish ko nikaalne police ki hiraasat se! Aur jugdish bahot khush dawdte hue usske saath nikal gaya, aur uss taraf jinn aadmiyon ne baaki ke sub inspector aur halwaldaron ko hands up par roke hue the, unn se sab ne kaha, “hum sab MLA ke aadmi hein unn ke bhatije ko nikaalne aaye hein, aap mein se kissi ne kuch bola to naukri se haath dhona padega sun liya sab ne!!!??” Aur sab jugdish ko gadi mein lekar rawana ho gaye wahan se!!

Udhar Khan aur Rathod bhi kaamyaab hue MLA ke ghar ke andar hathyaar, drugs aur bomb rakhne mein. Rathod aur Khan ke ilawa kissi ne nahin dekha ke unhon ne kahan par weh samaan rakhe! Baad mein to inn ko hi wapas aakar nikaalna tha samaan ko!

Vicky ek 4×4 drive karte hue Jugdish ko apne saath leja raha tha, chehre par nakaab pehne hue. Beech raaste mein uss ne unn 5 aadmiyon ko utaar diya aur cell se phone karke khan se baat kiya ke kaam ho gaya ab woh wapas jaa sakta hai.

Khan jab police station gaya to wahan ke sub inspector aur hawaldaron ne ussko bataya ke MLA ke aadmi ne bomb se raasta banakar apne bhatije ko baahar nikaal le gaye! Khan Rathod ko dekhkar hansste hue kaha, “Chalo bhai ab MLA ko giraftaar karna hai bomb lagaya unhon ne idhar chalo usske ghar ki talaashi lein kia pata usske ghar mein hi bomb mil jaaye, chalo tum log bhi hawaldaron sab chalo ek saath!!” Rathod sar jhukae hue peth daba kar hansste jaa raha tha!! Phir sar uthaakar Khan se kaha, “Sala MLA sochega ke kiss se pala par gaya hai aaj usska hahahahahaha!!!” Aur Khan ne “shhh” kaha Rathod ko aur sab nikle aur chale dobara MLA ke ghar ke taraf! Jate jate Khan ne chief minister ko phone karke bataya ke kia hua aur woh uss MLA ko arrest karne jaa raha hai, chief minister ne kaha, “chhodna mat kutte ko!!”

Uss taraf Vicky chala hi jaa raha tha sheher se door nikal gaya to jugdish ne sawaal kiya, “Hum jaa kahan rahe hein ji?” Vicky ne kaha, sheher se door lejane ka order mila hai tumko jahan police nahin ponhuche……” Aur Jugdish itminaan se baith gaya. Vicky ussi jangal ke taraf jaa raha tha jahan weh dehati log rehte hein. Gadi mein ek bag main 1 crore rupay bhi liya tha ussne wahan ke sardaarni ko dene ke liye. Aur gadi ke dikki mein usske hathyaar bhi the jinn se uss ne baaki ke chaar rapist ko maara tha, ek alag sa revolver bhi tha, lohe wala ghunsssa bhi tha aur woh special lakdi jiss se sabke haddiyan tode the uss ne! Aur drive karte waqt Vicky unn drishton ko phir se yaad karne laga, jiss raat ko Sanjana ki rape hui thi…. Vicky khaas kar unn hisson ko yaad kar raha tha jinn mein jugdish involve tha, Vicky ne yaad kiya keise jab Sanjana suicide karna chahti thi aur unn ke ek dost ne sanjana ko rokne ki koshish kiya tha to jugdish ne keise uss se kaha tha ke sanjana ko khudkhushi karne do, yehi achah hai….. Vicky sab yaad karte karte gadi ko jangal ke ek kone mein roka aur gadi ki dikki ko kholkar haath mein lohe wala ghunssa pehna aur hath mei uss lakdi ko lekar aya aur jugdish ke sar par zor se mara…… Jugdish chohunk gaya aur apne sar par hath dabaaye Vicky ke taraf dekhte hue poocha, “paagal ho gaye ho kia, kyun mara mujhe? Mera chacha tera gaand maar dega!” Vicky ne phir usske collar pakar kar zor se khincha gadi se baahar, itne zor se khincha ke jugdish seedhe zameen par jaa girra! Aur Vicky ne usske jabde par bahot zor se uss ladki se mara ke jabda phat gaya aur woh lahu lohaan hogaya, bhaagne ki nakaam koshish kiya ussne kyun ke bus do kadam age badhtehi phir zameen par girr gaya aur khauff bhare nazron se Vicky ke chehre mein dekhte hue bola,

“Kyun maar rahe ho mujhe kaun ho tum?”

Tab Vicky ne nakaab utara aur bola,

“Teri maut hoon, tere chaar saathiyon ko maar dala to tu keise bach sakta hai tu to unn sabka boss tha? Order diya tha meri masoom behen ko atmahatiya karne ke liye nah yeh le ab tu marr kutte, kamine teri gaand mein iss lakdi ko ghusedne ko mann kar raha hai mera!!”

Aur Vicky ussi pehle wale josh mein aakar andhadhun maarta gaya Jugdish ke shareer ke jo bhi hissa maarne ko mil raha tha, haddiyon ke tutne ki awaaz sunaayi de rahi thi, usske peyr par, ghutnon par, baazu ke haddiyon par chehre par, khopdi par, peeth par, kaandhe par kissi jagah ko bina mare nahin chorra Vicky ne…. Jugdish behosh ho gaya phir bhi Vicky maarta chala jaa raha tha…lagta tha Jugdish mar gaya hai phir bhi Vicky ruk nahin raha tha maarte chala jaa raha tha hanffte hue, enkhon se jharr jharr ensoo beh rahe the aur woh bolta jaa raha tha maarte waqt,

“Meri naazuk, masoom behen marr gayi teri vaja se, uss ko till till marte dekha main ne, usski izat luti tum ne mere enkhon ke saamne, aur tu chahta hai ke tujhe chorr doon, dekh SANJANA, DEKH MAAR DALA MAIN NE SABHI UNN KUTTON KO, AAJ MERA BADLA PURA HUA MERI GUDYA, KUTTE KI MAWT MARA MAIN NE SAB KO EK EK KARKE!! MAIN NE APNA VADA PURA KIYA SANJANA!! MAIN KAMZOR NAHIN HOON BABY, MAIN DARPOK BHI NAHIN HOON, TU KHUSH HAI NAH AB BABY? MAAR DALA MAIN NE SAB KO MAAR DALA MAIN NE!!!”

Aur Vicky bhi zameen par baith gaya Jugdish ke laash ke paas phir bhi revolver nikala aur usske laash mein sabhi goliyan utaar dala….. tab bhi ussko chain nahin mila, usske baad ussne uss lohe wale ghusse se usskA thopda bigaar dala ek ke baad ek ghuNssa maarte hue usske marre hue chehre par…Jugdish ke enkhen baahar nikal aaye, honth latak gaye, baal sameth khopdi ka ek hissa bagal mein latak gaya aur Vicky ghunssa maarta jaaa raha tha……

Kuch der Vicky wahin baitha raha zor se hanffte hue…. Phir ahiste ahiste khada hua aur chal kar apne 4×4 ke paas gaya, ek botal paani nikala gadi se aur piya, phir chehre par paani ko dala, haath munh dhoya aur jangal ke andar 4×4 drive karta gaya sardaarni se milne ke liye…….

Uss taraf Khan ne MLA ko hirasat mein le liya sabhi uss ke ghar se baramat ki gayi samaan ke saath. MLA kuch nahin kar saka. Bilkool hakbaka gaya. Khan TV walon ko saath legaya tha aur MLA ke ghar se mile saamaan sab TV walon ne public ko dikhaaya, hathyaar, bakse bhare golian, drugs sab kuch…MLA ke hathon mein hatkadi pehnaaye Khan ussko khinch kar police station legaye aur apne bhatije ko bhagaane ki aur drugs and weapons ki possession ki jurm mein salaakhon ke piche dala!!

To be continued………………….

The post Faraar (Completed) Update No 34 appeared first on Desi Stories.

https://ift.tt/eA8V8J

[dentalinfotoday]

There are many kinds of doctors—Doctors of English, Doctors of Philosophy, Doctors of Medicine, Dr. Pepper… (wink). Of course, when we use the term doctor, we most often mean a physician or Doctor of Medicine. But did you know that dentists are doctors, too? Every doctor has a specialty, and a dentist’s specialty is oral health.

William S. Darr, DDS explains how dentistry is a specific branch of medicine, and what it all means for the link between oral health and overall wellness.

Dentists are Doctors

Dentists are every bit as trained and educated as physicians. They have the same general education in science as physicians before they get clinical training in dentistry. This background education helps dentists look at you—the whole package—when taking care of your teeth.

Dentists are specialists in the science and mechanics of oral health, which extends to your head, face, and neck (the craniofacial region)—important parts of your body! Your oral health is an MVP when it comes to overall health and wellness, and dentists are just as important as other doctors when it comes to total body health.

The Oral-Systemic Link

Have you ever had your whole printer jam because one tiny piece of paper is out of line? It’s endlessly frustrating and time-consuming, and a reminder that machines require all parts, big and small, to be in good shape to get the job done. You are not a printer, but you are made of countless cells, organs, muscles, and bones that all work together to make you human and allow you to survive and be healthy.

Dentists look at your mouth, gums, and teeth, but they also look at your jaw (TMJ), face, head, and neck, keeping an eye out for signs of a problem such as swelling, discoloration, and more. Often times, dentists also perform tissue biopsies, diagnose illnesses, and screen for high blood pressure and oral cancer. Dentists can tell just by the look of your mouth if you might have other health issues like stress, chronic inflammation or poor quality of sleep.

Time and time again, doctors, dentists, and researchers come together to find important overlaps in medicine and dentistry. We call these overlaps “the oral-systemic link.” There are already many studies about oral health links to diabetes and heart conditions.

Seeing the dentist regularly is important in assessing your risks for health conditions all over your body. For example, respected family physician Bradley Bale says: “Periodontal disease is a medical disease with a dental solution.” Periodontal disease is advanced gum disease that’s linked to other chronic and acute illnesses like cardiovascular disease, stroke, diabetes, and more. The best dentists and physicians will insist on working together to help you reach your optimal health.

Good oral hygiene and regular visits with Dr. Darr accomplish much more than just polish your pearly whites. Everything from eating and sleeping to working and playing depends on a healthy smile. The healthier your mouth is, the healthier and happier your whole life will be.

Choose a dentist who asks questions about your personal goals for life and health. Ask them about their views on whole body dentistry that works toward your wellness as one, big, interconnected machine.

If you’re ready to take care of your health from top to bottom, make an appointment with your Broussard dental team today!

The content of this blog is not intended to be a substitute for professional medical advice, diagnosis, or treatment. Always seek the advice of qualified health providers with questions you may have regarding medical conditions.

The post Dentists Are Doctors, Too! appeared first on William S. Darr, DDS.

from William S. Darr, DDS https://www.wdarrdds.com/blog/dentists-are-doctors-too/ WILLIAM S. DARR, DDS 107 South Morgan Ave. Broussard, LA 70518 Serving Broussard, Youngsville, Lafayette, New Iberia & beyond. (337) 451-0458

[tumimmtxpapers]

A nanoparticle-incorporated STING activator enhances antitumor immunity in PD-L1-insensitive models of triple-negative breast cancer.

Related Articles A nanoparticle-incorporated STING activator enhances antitumor immunity in PD-L1-insensitive models of triple-negative breast cancer. JCI Insight. 2018 Nov 15;3(22): Authors: Cheng N, Watkins-Schulz R, Junkins RD, David CN, Johnson BM, Montgomery SA, Peine KJ, Darr DB, Yuan H, McKinnon KP, Liu Q, Miao L, Huang L, Bachelder EM, Ainslie KM, Ting JP Abstract Triple-negative breast cancer (TNBC) has few therapeutic options, and alternative approaches are urgently needed. Stimulator of IFN genes (STING) is becoming an exciting target for therapeutic adjuvants. However, STING resides inside the cell, and the intracellular delivery of CDNs, such as cGAMP, is required for the optimal activation of STING. We show that liposomal nanoparticle-delivered cGAMP (cGAMP-NP) activates STING more effectively than soluble cGAMP. These particles induce innate and adaptive host immune responses to preexisting tumors in both orthotopic and genetically engineered models of basal-like TNBC. cGAMP-NPs also reduce melanoma tumor load, with limited responsivity to anti-PD-L1. Within the tumor microenvironment, cGAMP-NPs direct both mouse and human macrophages (M), reprograming from protumorigenic M2-like phenotype toward M1-like phenotype; enhance MHC and costimulatory molecule expression; reduce M2 biomarkers; increase IFN-γ-producing T cells; augment tumor apoptosis; and increase CD4+ and CD8+ T cell infiltration. Activated T cells are required for tumor suppression, as their depletion reduces antitumor activity. Importantly, cGAMP-NPs prevent the formation of secondary tumors, and a single dose is sufficient to inhibit TNBC. These data suggest that a minimal system comprised of cGAMP-NP alone is sufficient to modulate the tumor microenvironment to effectively control PD-L1-insensitive TNBC. PMID: 30429378 [PubMed - as supplied by publisher] http://dlvr.it/QrNx3m

[graphicdesignonline]

Ears in Quran

Kaan (ears) insaani jism ka ek bahut hi important organ hai. Insaan jitni tezi se apni skin par kuchh chiz mehsoos karta hai, apni zubaan se kuchh chiz taste karta hai ya jitni tezi se apne naak se koi chiz smell karta hai, in teeno functions se bhi kaaee zyada tezi se insan apne kaano ke zariye sounds ko sunta hai. Jab sound wave kaano tak pahuchti hai to insan ka dimag (brain) use sirf 0.05 seconds me pechan leta hai. Iske teen hisse hote hain, outer ear, middle ear and innner ear. Outer ear ke do hisse hote hain, Pinnaaur ear canal. Middle earek chhoti katori ki tarha hota hai, iski bahar ki deewar par ek parda sa laga rahta hai. Bich me teen chhoti chhoti haddiyan zanjeer ki tarha judi hoti hain. ke 2 hisee aur unke mukhtalif functions hote hain, eardrum aur auditory  muscles ka kaam soundwave vibrations ko ander ke hisse tak pahuchana hota hai.. Kaan ke sab se andarooni hisse ki banavat bahut hi pechida (complex) hoti hai jise bhool bhulayya bhi kehte hain. Isme  half moon ki tarha teen hisse hote hain jinme bahut sare holes hote hain aur inhi ke ande sunne ki vein rahti hai jiska direct relation sar ya dimag (brain) ke sath hota hai. Inme bahut sare hair cells hote hain jo vibration ko electrical signals me tabdeel karte hain aur hearing nerve ke zariye brain tak pahuchate hain. Har ek hair cell ke ander microscopic (aam taur par jise dekha na ja sake waise bahut hi bareek) projections hote hain jinhe stereocilia kaha jata hai. Agar awaz particular decible se ya  hadd se badh jaye to yeh stereocilia bend ho jate hain to toot bhi sakte hain. Phir uske baad sound signals brain tak nahi pahuch sakte. yeh hair cells apne aap repair nahi hote aur naahi ek martaba damage hone ke baad medically regenerate kiye ja sakte hain. Aawaz ki tarang jo hawa me phailti hai, yeh kaan ke bahari hisse me jama ho kar kaano se takraati hai jisse vibration create hota hai ya thartharahat hoti hai. Phir yeh awaz andarooni hisse tak pahuchti hai jisse insan ko ek tarike ki feeling hoti hai yani kuchh mehsoos karta hai. Yahi mehsoos karne par insan ke kaee sari subsidiary feelings dependent hoti hai, jaise khushi, darr-khauf, baukhlaahat, gussa, muskurahat, hasi, wagairah. Agar upar mention kiye gaye kisi bhi organ me se koi ek bhi kharab ho jaye to insan partially ya totally sunne ki salahiyat kho sakta hai ya kho deta hai, ya ek tarike ka disorder paida ho jata hai jiske liye use hearing aid ka sahara lena padta hai. Ek aur dilchasp baat yeh hai ki sunne ke alawa jism ka balance banaye rakhne me bhi kaano ek aham kirdar nibhate hain. Sunne, dekhne ke alawa skeletal system ko (haddiyon ke dhanche ko) maintain karne ka kaam bhi kaano ko zariye hota hai jisse body ka equilibrium manage hota hai aur body balance hoti hai. Agar insan ke kaano me sunne ki salahiyat kam ho jaye, ya khatm ho jaye, ya sardi zukam (cough and cold) ki wajah se ya depression aur stress ya kisi aur bimari ki wajah se kaano me shadeed dard paida ho jaye, ya kaano me bahut mel jama ho jaye, ya koi chiz ke sariye physically kano me damage ho jaye, ya bimari ki wajah se kaan bhari lage ya sar chakrane lage to insan ki poori body ka balance mutassir hone lagta hai. iske liye doctors advise dete hain ki kano me buds, machis ki tili, nukili chiz wagairah na dali jaye aur diagonisis ke taur par ent doctors kano ke drops wagairah dete hain aur agar takleef bahut badh jaye to surgical steps bhi lene padte hain. Kano me takleef ho to swimming na karne ki bhi advise di jati hai. Quran me insan ko di gayi kano ki aur sunne ki salahiyat ki is azeem nemat ka jagah jagah tazkera hai aur kaee makamat par aaya hai ki, "woh log jo sunte hain" yani woh log hidayatyafta insano me shumar kiye jate hain jinho ne Allah ke kalaam ko suna, phir us par ghauro fikr kiya, phir use tasleem kiya aur phir amalqarda hue. Sura fussilat (41:44) me bataya gaya hai ki jo log nahi sunte, unke kano me behrapan hai, aur unki aankhe andhi hai. (mafhoom) Bazahir bhale hi kufr o shirk me mubtala log sunnte dekhte nazar aaye, magar islam ki nazar me aise log behre hain kyon ki Allah ki nishaniyan wazeh ho jane ke baad bhi unho ne ek kaan se sun kar doosre kaan se baat ko nikal diya, ya nazar andaaz kar diya. Isi tarha Surah Nooh me ata hai ki jab 950 years tak dawat iLallah ka paigham Nooh alayhis salam ne logo ko diya to kuchh logo ne apne kano me ungliya daal di, yani woh sunna nahi chahte the. Isi tarha Allah ne insano par maazi me jo azabaat nazil farmaye unme ek azab awaaz ka azaab bhi hai aur jahannamiyo ko bhi awaz ke is azab ka maza chakahya jayega, jaise ki Surah Mulk me 67:7 me aata hai ki jab jahannmi log jahannum ki aag me jhonke jayenge to woh usme se ek khatarnaak awaz ko sunenge jab woh ubal rahi hogi (mafhoom) Aur phir baad ki ayat me aata hai ki kaafir tasleem karenge (67:10) ki agar hum (hame deen ki dawat dene walo ki baat) sunte to jahannamiyon me hamara shumar na hota (mafhoom) Aur phir Surah Mulk me hi iske baad wali ayat me aata hai (67:23) ki wahi Allah hai jisne tumhe sunne ki dekhne ki salahiyat di aur dil diye, tum bahut kam shukrguzar hote ho. (mafhoom) Salih Alayhis salam ki qaum, qaum me Samood par phir jo sakht'tareen azab aya jab ek bahut zordaar awaz ne unhe aa pakda aur woh itni khatarnaak awaz thi ki us qaum ke nafarmaan logo ke kano ke parde phat gaye aur zameen me zalzala aa gaya aur woh sab halaq o barbaad ho gaye. Iska zikr quran me (11:67) Surah Baqarah me un logo ko behra, goonga aur andha bataya gaya hai jo raah e haqq se bhatak gaye aur jinho ne Allah ki tawheed ka inkar kiya. Quran ke bahut sare huqooq me se ek huqooq quran ko sunna bhi hai. aur is tarha hum Allah ki di gayi is azeem nemat ka ba-khoobi istemal kar sakte hain. Aaj ke jadeed daur me hame ek se badh kar ek khoobsoorat qirat internet ke zariye sunne mil sakti hai, agar hum isse istefada karne ke bajaye, music, layani bate, jokes, movies aur lahw chizo ko sunenge to zaroor ba zaroor hame iske bare me roze qayamat poochh hogi. ( https://en.muqri.com/  http://urdubooksaudio.com/) Allah ham sab ko hame di gayi kaano ki is azeem nemat se fayeda utha kar apni aakhirat sanwarne ki tawfeek ata farmaye. Aamin.________ "And if We had made it a non-Arabic Qur'an, they would have said, "Why are its verses not explained in detail [in our language]? Is it a foreign [recitation] and an Arab [messenger]?" Say, "It is, for those who believe, a guidance and cure." And those who do not believe - in their ears is deafness, and it is upon them blindness. Those are being called from a distant place." (41:44) Sūrat Fuṣṣilat "And for those who disbelieved in their Lord is the punishment of Hell, and wretched is the destination. When they are thrown into it, they hear from it a [dreadful] inhaling while it boils up." (67: 6-7) Sūrat Al-Mulk "And they will say, "If only we had been listening or reasoning, we would not be among the companions of the Blaze." (67: 10) Sūrat Al-Mulk "Say, "It is He who has produced you and made for you hearing and vision and hearts; little are you grateful." (67: 23) Sūrat Al-Mulk "And the shriek seized those who had wronged, and they became within their homes [corpses] fallen prone" (11:67) Sūrat Hud "Deaf, dumb, and blind, They will not return (to the path)" (2:18) Sūrat Al-Baqarah wallaho aalam https://youtu.be/gqZ5and1HCI   https://youtu.be/rQiH8Hbxgjw https://youtu.be/flIAxGsV1q0 https://youtu.be/P82hhdWsOpM https://youtu.be/lTGQmvCuFqA

[phooll123]

Game-Changing Study Finds Dozens 

 of Genes Tied to Depression

Scientists have uncovered dozens of genes that increase the risk of depression, a major finding that underscores the complexity of the disease and reveals why antidepressant therapies work well for some people but are utterly ineffective for others. A global consortium of more than 200 scientists identified 44 gene variants, or small changes in genes, each one contributing in some small way to a person's risk of depression. Thirty of the gene variants had not been identified in any previous study. The hope is that the discovery will pave the way to new, diverse therapies for depression, an often-crippling disease that affects nearly 15 percent of adults worldwide, according to the World Health Organization. Only about half of patients respond well to existing treatments, which include both drug and psychotherapy. [9 DIY Ways to Improve Your Mental Health] Depression is one of the world's most serious yet elusive public health problems, said Dr. Steven Hyman, director of the Stanley Center for Psychiatric Research at the Broad Institute of MIT and Harvard, who was not involved with this study. "Despite decades of effort there have been, until now, only scant insights into its biological mechanisms," Hyman told Live Science. "This landmark study represents a major step toward elucidating the biological underpinnings of depression." The massive study, which was published yesterday (April 26) in the journal Nature Genetics, analyzed the genomes of more than 135,000 patients with major depressive disorder, colloquially called major depression, and compared them to the genomes of nearly 350,000 people with no depression. Many of the genes tied to depression also are associated with other psychiatric conditions, such as schizophrenia and bipolar disorder, as well as health issues linked to depression, including obesity and insomnia, the study found. Some of the gene variants control neurotransmitters in the brain, such as dopamine and adrenaline, which the current class of antidepressant drugs can target. Other gene variants have nothing to do with neurotransmitters, though, which may be why antidepressants don't work for some people if the genetic underpinnings of their depression lie elsewhere in the brain, the scientists said. [7 Ways Depression Differs in Men and Women] Each of the newly identified gene variants is essentially a target that could be addressed with a drug therapy, said co-lead study author Dr. Patrick Sullivan, director of the Center for Psychiatric Genomics at the University of North Carolina School of Medicine. (Sullivan, along with several other of the study researchers, has ties to either pharmaceutical or genetics companies.) In the new study, the researchers found no single gene variant that was a strong risk factor for depression, the way that mutations in the BCRA1 gene frequently lead to breast cancer. Instead, each gene variant contributes to depression in an incremental way. "If there were something major, we would have found it," Sullivan told Live Science. All people carry some of these gene variants for depression, but some people have more than others, placing them at a greater risk for depression, said lead study author Naomi Wray, a professorial research fellow at the University of Queensland in Australia. Earlier studies on identical twins have revealed that genes may be responsible for half of all cases of depression. Other times, the cause may be stress or trauma. Some people, for reasons not understood, can remain resilient despite living through experiences that would make others depressed. "We know that many life experiences also contribute to risk of depression, but identifying the genetic factors opens new doors for research into the biological drivers," Wray said in a statement. Sullivan compared the study on depression and genetics to research that was done on cardiovascular disease decades ago, which has led to drug therapies that significantly reduce the risk of heart attacks and stroke. Most of the antidepressant drugs used today were discovered by chance, but now the search for new drugs can be "rationally driven" by biological discovery, he said. For example, gene variants are associated with RNA and the movement of molecules out of the nucleus of nerve cells, Sullivan noted. This discovery could lead to "biologically driven therapeutics" targeting RNA, he said. Scientists involved in this study are working on an online tool to allow volunteers with depression to take part in further genetic studies.  Members of the public who want to be notified about the launch of the study can e-mail darr@kcl.ac.uk. Follow Christopher Wanjek @wanjekfor daily tweets on health and science with a humorous edge. Wanjek is the author of "Food at Work" and "Bad Medicine." His column, Bad Medicine, appears regularly on Live Science.

Game-Changing Study Finds Dozens of Genes Tied to Depression

Scientists have uncovered dozens of genes that increase the risk of depression, a major finding that underscores the complexity of the disease and reveals why antidepressant therapies work well for some people but are utterly ineffective for others. A global consortium of more than 200 scientists identified 44 gene variants, or small changes in genes, each one contributing in some small way to a person's risk of depression. Thirty of the gene variants had not been identified in any previous study. The hope is that the discovery will pave the way to new, diverse therapies for depression, an often-crippling disease that affects nearly 15 percent of adults worldwide, according to the World Health Organization. Only about half of patients respond well to existing treatments, which include both drug and psychotherapy. [9 DIY Ways to Improve Your Mental Health] Depression is one of the world's most serious yet elusive public health problems, said Dr. Steven Hyman, director of the Stanley Center for Psychiatric Research at the Broad Institute of MIT and Harvard, who was not involved with this study. "Despite decades of effort there have been, until now, only scant insights into its biological mechanisms," Hyman told Live Science. "This landmark study represents a major step toward elucidating the biological underpinnings of depression." The massive study, which was published yesterday (April 26) in the journal Nature Genetics, analyzed the genomes of more than 135,000 patients with major depressive disorder, colloquially called major depression, and compared them to the genomes of nearly 350,000 people with no depression. Many of the genes tied to depression also are associated with other psychiatric conditions, such as schizophrenia and bipolar disorder, as well as health issues linked to depression, including obesity and insomnia, the study found. Some of the gene variants control neurotransmitters in the brain, such as dopamine and adrenaline, which the current class of antidepressant drugs can target. Other gene variants have nothing to do with neurotransmitters, though, which may be why antidepressants don't work for some people if the genetic underpinnings of their depression lie elsewhere in the brain, the scientists said. [7 Ways Depression Differs in Men and Women] Each of the newly identified gene variants is essentially a target that could be addressed with a drug therapy, said co-lead study author Dr. Patrick Sullivan, director of the Center for Psychiatric Genomics at the University of North Carolina School of Medicine. (Sullivan, along with several other of the study researchers, has ties to either pharmaceutical or genetics companies.) In the new study, the researchers found no single gene variant that was a strong risk factor for depression, the way that mutations in the BCRA1 gene frequently lead to breast cancer. Instead, each gene variant contributes to depression in an incremental way. "If there were something major, we would have found it," Sullivan told Live Science. All people carry some of these gene variants for depression, but some people have more than others, placing them at a greater risk for depression, said lead study author Naomi Wray, a professorial research fellow at the University of Queensland in Australia. Earlier studies on identical twins have revealed that genes may be responsible for half of all cases of depression. Other times, the cause may be stress or trauma. Some people, for reasons not understood, can remain resilient despite living through experiences that would make others depressed. "We know that many life experiences also contribute to risk of depression, but identifying the genetic factors opens new doors for research into the biological drivers," Wray said in a statement. Sullivan compared the study on depression and genetics to research that was done on cardiovascular disease decades ago, which has led to drug therapies that significantly reduce the risk of heart attacks and stroke. Most of the antidepressant drugs used today were discovered by chance, but now the search for new drugs can be "rationally driven" by biological discovery, he said. For example, gene variants are associated with RNA and the movement of molecules out of the nucleus of nerve cells, Sullivan noted. This discovery could lead to "biologically driven therapeutics" targeting RNA, he said. Scientists involved in this study are working on an online tool to allow volunteers with depression to take part in further genetic studies.  Members of the public who want to be notified about the launch of the study can e-mail darr@kcl.ac.uk. Follow Christopher Wanjek @wanjekfor daily tweets on health and science with a humorous edge. Wanjek is the author of "Food at Work" and "Bad Medicine." His column, Bad Medicine, appears regularly on Live Science.

ShareThis Copy and Paste

Game-Changing Study Finds Dozens of Genes Tied to Depression Scientists have uncovered dozens of genes that increase the risk of depression, a major finding that underscores the complexity of the disease and reveals why antidepressant therapies work well for some people but are utterly ineffective for others. A global consortium of more than 200 scientists identified 44 gene variants, or small changes in genes, each one contributing in some small way to a person's risk of depression. Thirty of the gene variants had not been identified in any previous study. The hope is that the discovery will pave the way to new, diverse therapies for depression, an often-crippling disease that affects nearly 15 percent of adults worldwide, according to the World Health Organization. Only about half of patients respond well to existing treatments, which include both drug and psychotherapy. [9 DIY Ways to Improve Your Mental Health] Depression is one of the world's most serious yet elusive public health problems, said Dr. Steven Hyman, director of the Stanley Center for Psychiatric Research at the Broad Institute of MIT and Harvard, who was not involved with this study. "Despite decades of effort there have been, until now, only scant insights into its biological mechanisms," Hyman told Live Science. "This landmark study represents a major step toward elucidating the biological underpinnings of depression." The massive study, which was published yesterday (April 26) in the journal Nature Genetics, analyzed the genomes of more than 135,000 patients with major depressive disorder, colloquially called major depression, and compared them to the genomes of nearly 350,000 people with no depression. Many of the genes tied to depression also are associated with other psychiatric conditions, such as schizophrenia and bipolar disorder, as well as health issues linked to depression, including obesity and insomnia, the study found. Some of the gene variants control neurotransmitters in the brain, such as dopamine and adrenaline, which the current class of antidepressant drugs can target. Other gene variants have nothing to do with neurotransmitters, though, which may be why antidepressants don't work for some people if the genetic underpinnings of their depression lie elsewhere in the brain, the scientists said. [7 Ways Depression Differs in Men and Women] Each of the newly identified gene variants is essentially a target that could be addressed with a drug therapy, said co-lead study author Dr. Patrick Sullivan, director of the Center for Psychiatric Genomics at the University of North Carolina School of Medicine. (Sullivan, along with several other of the study researchers, has ties to either pharmaceutical or genetics companies.) In the new study, the researchers found no single gene variant that was a strong risk factor for depression, the way that mutations in the BCRA1 gene frequently lead to breast cancer. Instead, each gene variant contributes to depression in an incremental way. "If there were something major, we would have found it," Sullivan told Live Science. All people carry some of these gene variants for depression, but some people have more than others, placing them at a greater risk for depression, said lead study author Naomi Wray, a professorial research fellow at the University of Queensland in Australia. Earlier studies on identical twins have revealed that genes may be responsible for half of all cases of depression. Other times, the cause may be stress or trauma. Some people, for reasons not understood, can remain resilient despite living through experiences that would make others depressed. "We know that many life experiences also contribute to risk of depression, but identifying the genetic factors opens new doors for research into the biological drivers," Wray said in a statement. Sullivan compared the study on depression and genetics to research that was done on cardiovascular disease decades ago, which has led to drug therapies that significantly reduce the risk of heart attacks and stroke. Most of the antidepressant drugs used today were discovered by chance, but now the search for new drugs can be "rationally driven" by biological discovery, he said. For example, gene variants are associated with RNA and the movement of molecules out of the nucleus of nerve cells, Sullivan noted. This discovery could lead to "biologically driven therapeutics" targeting RNA, he said. Scientists involved in this study are working on an online tool to allow volunteers with depression to take part in further genetic studies.  Members of the public who want to be notified about the launch of the study can e-mail darr@kcl.ac.uk. Follow Christopher Wanjek @wanjekfor daily tweets on health and science with a humorous edge. Wanjek is the author of "Food at Work" and "Bad Medicine." His column, Bad Medicine, appears regularly on Live Science. Game-Changing Study Finds Dozens of Genes Tied to Depression Scientists have uncovered dozens of genes that increase the risk of depression, a major finding that underscores the complexity of the disease and reveals why antidepressant therapies work well for some people but are utterly ineffective for others. A global consortium of more than 200 scientists identified 44 gene variants, or small changes in genes, each one contributing in some small way to a person's risk of depression. Thirty of the gene variants had not been identified in any previous study. The hope is that the discovery will pave the way to new, diverse therapies for depression, an often-crippling disease that affects nearly 15 percent of adults worldwide, according to the World Health Organization. Only about half of patients respond well to existing treatments, which include both drug and psychotherapy. [9 DIY Ways to Improve Your Mental Health] Depression is one of the world's most serious yet elusive public health problems, said Dr. Steven Hyman, director of the Stanley Center for Psychiatric Research at the Broad Institute of MIT and Harvard, who was not involved with this study. "Despite decades of effort there have been, until now, only scant insights into its biological mechanisms," Hyman told Live Science. "This landmark study represents a major step toward elucidating the biological underpinnings of depression." The massive study, which was published yesterday (April 26) in the journal Nature Genetics, analyzed the genomes of more than 135,000 patients with major depressive disorder, colloquially called major depression, and compared them to the genomes of nearly 350,000 people with no depression. Many of the genes tied to depression also are associated with other psychiatric conditions, such as schizophrenia and bipolar disorder, as well as health issues linked to depression, including obesity and insomnia, the study found. Some of the gene variants control neurotransmitters in the brain, such as dopamine and adrenaline, which the current class of antidepressant drugs can target. Other gene variants have nothing to do with neurotransmitters, though, which may be why antidepressants don't work for some people if the genetic underpinnings of their depression lie elsewhere in the brain, the scientists said. [7 Ways Depression Differs in Men and Women] Each of the newly identified gene variants is essentially a target that could be addressed with a drug therapy, said co-lead study author Dr. Patrick Sullivan, director of the Center for Psychiatric Genomics at the University of North Carolina School of Medicine. (Sullivan, along with several other of the study researchers, has ties to either pharmaceutical or genetics companies.) In the new study, the researchers found no single gene variant that was a strong risk factor for depression, the way that mutations in the BCRA1 gene frequently lead to breast cancer. Instead, each gene variant contributes to depression in an incremental way. "If there were something major, we would have found it," Sullivan told Live Science. All people carry some of these gene variants for depression, but some people have more than others, placing them at a greater risk for depression, said lead study author Naomi Wray, a professorial research fellow at the University of Queensland in Australia. Earlier studies on identical twins have revealed that genes may be responsible for half of all cases of depression. Other times, the cause may be stress or trauma. Some people, for reasons not understood, can remain resilient despite living through experiences that would make others depressed. "We know that many life experiences also contribute to risk of depression, but identifying the genetic factors opens new doors for research into the biological drivers," Wray said in a statement. Sullivan compared the study on depression and genetics to research that was done on cardiovascular disease decades ago, which has led to drug therapies that significantly reduce the risk of heart attacks and stroke. Most of the antidepressant drugs used today were discovered by chance, but now the search for new drugs can be "rationally driven" by biological discovery, he said. For example, gene variants are associated with RNA and the movement of molecules out of the nucleus of nerve cells, Sullivan noted. This discovery could lead to "biologically driven therapeutics" targeting RNA, he said. Scientists involved in this study are working on an online tool to allow volunteers with depression to take part in further genetic studies.  Members of the public who want to be notified about the launch of the study can e-mail darr@kcl.ac.uk. Follow Christopher Wanjek @wanjekfor daily tweets on health and science with a humorous edge. Wanjek is the author of "Food at Work" and "Bad Medicine." His column, Bad Medicine, appears regularly on Live Science. ShareThis Copy and Paste

via Blogger https://ift.tt/2KnhfrB