#and jaundice ig

[feelingpure]

FELLOW TRAVELERS 1.01 ‘You're Wonderful’

[ladysophiebeckett]

her mom watched 'working girl' and it reminded her of lety but with a tropicana lime twist to make her pop.

[boag]

Also ummmm

Colleen Ballinger ukulele improv “apology” video is killing me

[0verseer-o9]

Opinion on the beta Crisp sprite remnants?

CRISP WAS CANONICALLY BLACKWASHED?? /j

Hi Pyre!! I’m glad you brought this up I need to ramble abt this.

Thoughts:

I think it’s cool that we got to see part of his beta design but I wish we got to see more about the design processes of the party members, along w/ scrapped ideas like Fardy becoming a joy mutant. I love the final design of Crisp more tho.

GAME THEORIES!!:

Something I theorized was that Crisp wasn’t meant to be in the game but when the pink robe guy was changed to be an NPC Dingaling was like “hmm maybe removing the only black party member from this game would be problematic” so he made Crisp.

A related theory to this is that Crisp was not originally black but was changed to be after the pink robe guy thing.

Another ANOTHER theory is that Dingaling thought “hm, let’s make him gay” and took Crisp’s beta (male /j) sprite and made Toby. I don’t believe this theory but it’s fun to think abt.

Another x3 theory is that beta Crisp had jaundice /j

Some stuff I noticed!!

• Crisp is 75% headless (had to save his robux ig /j)

• He has no facial hair (his goatee and a place where his mustache would show doesn’t show it)

• Crisp’s shawl was shaded and tbh I’m glad Dingaling changed it

• Instead of cyan glove things he has white ones

• He has four swords instead of six

• The middle of his chest is closer to the left by one pixel

• The swords are less shaded

• There’s more shading on the right arm

• There’s less shading on the torso

I think the white glove things on his hand are and it would be cool if Crisp still had bandages

TLDR: I think it’s cool we got to see this and I have many thoughts abt it

[neptuniadoesstuff]

The Stoopid Idiot is done!

This was more if me idk... experimenting with the basic brushes in IbisPaint.

Yeah anyways this is Addax (Full name: Addax Envii Montegue) or commonly referred to as "King". He's a cannibalistic entitled brat who was the favorite child of his father (a scientist) who he affectionately calls "Daddy". (I swear to God if some of you take this the wrong way I WILL STRANGLE YOU!)

He is naturally blonde but the way he dyed his hair is uh.... bl00d. Yep, bl00d. He used frikin bl00d of his prey to dye his hair.

He is MEANT to be human looking buypt considering he was experimented on by his father, Dr. Oslo Grey Montegue, he kinda doesn't too much NORMAL human anatomy. (Like the fact he is actually Inteseggs)

Hews is also a member of Delarium, but he's kinda a silent a-hole to them & a complete menace to the group's messenger, Paris, who he only sees as a roasted chikin. (Also he really doesn't listens to the clothing rules of the team which explains his outfit-)

Yeah uh... this was smtn... ig? Idk I just drew a more human-lookin Malice w/o the jaundice part but he still h8s mailmen. (Btw that wine loving fella with jaundice named Malice belongs to my frend, Gali/Leo)

This character was created by me (for the Egolandia Universe) but is originally owned by my frend.

Art is mine.

Program: IbisPaint.

Bubs' TOS: Plz don't repost/steal, trace, or recolor my art WITHOUT MY PERMISSION! If you do, I'll take yur femur and pelvis.. SO, DON'T THINK ABOUT IT! (The PNS on my blog's pinned post clearly means "Please No Steal" plz follow that rule.) If you do post my art on anything like yur blog or somewhere else (With my permission) PLEASE CREDIT ME!

[adonischildsupportcase]

Apparently Chime does throw you your tax returns back quick as shit since @theblacktroymcclure finally did something with that lonely ass jaundice eyed Jordan hued incisor he runnin around with

( @theblacktroymcclure via IG Stories)

[porcelain-animatronic]

It probably doesn’t help that I have the complexion of a Victorian woman who died of being too pale and every single blush makes me look like some random Yankees mocking of the macaroni fashion trend but natural blush just doesn’t work for me

Unpopular makeup opinion: I really don’t like blush. Like unless it is the cartooniest application it just doesn’t work for me

[tea-with-evan-and-me]

“i genuinely can't tell if this is serious or not and thus how to proceed with my answer”

She does have a skin condition I believe she said so in a live once but I don’t know how you were supposed to know it’s not your fault at all.

Honestly she looks really sweet and fun, she also said multiple time that she loves Evan (and dislike Emma). I kind like her !

😭 thank you! i didn’t even know this lady’s name, much less about any health/skin conditions she has…..

i had to google this, for anyone curious like me: she talked about having a bilirubin disorder that left her skin jaundiced. if your liver doesn’t properly process the yellowish pigment in your bile, then your skin and even the whites of your eyes can become jaundiced. it’s not dangerous if that is the cause, but i have seen a couple of people jaundiced due to end stage liver failure. that’s completely separate from the orange fake tan and that particular filter that bronzes and contours you ridiculously, which is what makes her look so strange. if you go on her IG page you can see she doesn’t always look like that, either. in fact, she looks normal without it 💀 look at how the filter missed her arm.

[princess-pill-enjoyer]

I think I fucked my liver up bad

Uh. Soo the jaundice shit never went away. It's never as bad don't get me wrong. But while before I only jaundiced when I was going overboard, I now keep a light shade of yellow pretty much 24/7. My eyes are never fully white

I'm not gonna bother going over all the symptoms I've gained since I oded earlier this month. Shit hurts but no where near unbearable. I've been holding out on talking bout it cause of that. Partially because I didn't want to explain and partially cause I didn't think anyone would take it serious because of how mild the symptoms were at first. Ah well mild aint the word. Expected..? I dunno in my mind knowing that most of that was the leftover dph beating my ass made it feel a lot better. Least I knew exactly what I'd be feeling and around when. Now? So many random pains ;_;

I'm thinking I have hemolytic anemia that was more than likely because of the excess bile shit I never could fully fix. Ik it sounds like I'm just self diagnosing to make myself sound more pitiful but if I went through the shit I've been feeling it'd probably be more obvious. I'm sorry I'm not explaining in detail there either. I'm kinda half sleep tryna write 😭

Either way in my mind that is uh.. ig that's my consequence. I'm hoping it is just anemia from that day and not just the tip of the iceberg. Unless it's not even that heh. Then ima be mad

I feel like shit on and off. I swear it's so random. Sometimes I forget anything happened and I'll just be existing. But then like today it's like my entire body burns and aches. Once again, not unbearable. The burny feeling is moreso uncomfortable not painful. But the burning + the random dull aches everywhere is fucking annoying

I remember in the past being paranoid about what'd bring my lucky streak to an end. Ig this is it. I don't know if it's fair to call it that tho. I'm not hurting too bad honestly. It's nothing I can't handle with it not being constant. Did I get off lucky? I'm still here. My jaundice isn't bad. My stomach isn't horrible. My speech is mostly normal now that I've gotten used to not relying on guesstimating sm lol. Eh. I'll be seeing soon. Guess I really don't need to question it anymore

I'm going to sleep now im actually in the middle of this nonsense rn I'm so tired. My body hurt :[

[vaidyaslaboratory]

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Understanding Malaria: Symptoms, Treatment, and Diagnostic Excellence 

Symptoms of Malaria 

Malaria is a serious disease caused by Plasmodium parasites, transmitted through the bites of infected mosquitoes. Early recognition of symptoms is vital for effective treatment. The primary symptoms include: 

Fever and Chills: Sudden, high fever accompanied by severe chills. 

Headache: Intense and persistent headaches. 

Nausea and Vomiting: Common occurrences during infection. 

Muscle Pain and Fatigue: Generalized muscle pain and fatigue. 

Diarrhea: In some cases, patients may experience diarrhea. 

Signs of Malaria 

Recognizing the signs of malaria can aid in early diagnosis and treatment: 

Anemia: Due to the destruction of red blood cells. 

Jaundice: Yellowing of the skin and eyes, indicating liver involvement. 

Splenomegaly: Enlargement of the spleen as it filters infected blood cells. 

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Effective treatment of malaria involves: 

Antimalarial Medications: Chloroquine, artemisinin-based combination therapies (ACTs), and quinine are commonly used. 

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[navelgrazing]

Can she make it to more than 1 post??

I follow someone on tiktok/IG who speaks a lot about autism, how capitalism forces overwork and there really is no such thing as lazy- somewhat unrelated they had a TT about how to work the algorithms in your favor by flooding it with signals. so anything that’s even mildly interesting- like it, share it, comment, etc. they didn’t go into it much but it makes sense from my understanding of SM platforms from my perch in an adjacent field- fewer signals raise the importance or focus on those signals as they collate you into useful groupings to advertise to. but lots and lots and lots of signals, especially of unrelated topics or activities or whatever, yes, put you into the mix for more potential types of advertising but they also make the advertising you receive less intensely focused on purchasing stupid shit that you put into your cart once. 

All this to say I have started applying that to email newsletters (I have always pulled for email as a channel and, boy, is it funny to watch it become super relevant again at least in terms of content/thought distribution.) Also email is actually regulated through CAN-SPAM so you can actually select who you no longer want to be targeted by and there’s just less unchosen noise. Aaaanyways, all of that to say one of the newsletters I signed up for recently is called #1000wordsofsummer in which the organizing author/writer sends daily emails for 30 days to write a thousand words each day. In each day’s email, she includes inspiration/thoughts as well as letters and advice from contributors. I am MASSIVELY behind- but it did get me to start these meandering posts so there’s that. yesterday’s email had a letter from the author, kiese layman, who I follow and is very accomplished (they don’t just hand out those MacArthur genius grants I don’t think, not that we are wed to traditional or visible forms of accomplishment) and the visceral expression around how he feels about people now after all we’ve been through in the last few years (but also forever):

I’m so scared of people, Jami. I’m so disappointed in us. And I’m asking you if you feel this feeling much. I avoid people now at all costs. I’m just terrified of who we are, who I am, and I wonder how to do the work we have chosen while being terrified of people and petrified of their touch. This letter has far more to do with money and intimacy and feeling good than I’m comfortable writing, but if you want to write back, can you tell me if you still love people.  

Im not scared of my people but I am scared for them. and yes, everyone else, I have lost what little faith I had in things like goodness and fairness. Truly, this jaundiced feeling is not new, it has the texture of familiar cloth, but the all encompassing nature of it’s harsh truth- that is newly crystallized. Money, intimacy and feeling good. It was hard to write when my daily realities were perhaps harder but I shared them more completely with others, it is even harder to compound those feelings of failure when the whole wide world seems to be falling off a cliff. I used to be able to point to something- trump, COVID, whatever but now it just feels like the grey texture of reality. maybe the challenge then is to write that- the truth of what I'm living through now. A line from one of my favorite movies, end of the affair, rings so true- Pain is easy to write. In pain we're all drabbly individual. Now what can one write about happiness?

[kmp78]

Yeh I also thought on his IG Live he looked tanned 🧐 That wasn't from the sun because he has tons of makeup on his face and used a damn bad filter...he looks like he has jaundice...🤔🤭😂😂😂🤷🏼‍♀️

Excellent! He already whined about his "bad back" (which never seems to bother him when he's CLIMBING ON ROCKS but fpr some reason always flares up when he has to perform on stage in front of his Mother Hens... 🤔) in one interview so a new illness brings a nice twist to his sob stories! 🙌

[flyonthewallmedstudent]

Hydatid Cysts & Echinococcosis

Parasites are fun.

It's a tapeworm that forms cysts.

Cold open with a very short case report

30F in Mexico presents with RUQ pain and nausea

on examination of her abdo, authors felt a RUQ mass

She received an MRI of her abdo, revealing a cystic mass

on histopatho following a cystectomy, they found Echinococcosis cysts, she also had positive IgG serology to echinococcosis granulosus

Microbiology

a tapeworm that forms cysts that are usually found in the liver or lungs (if not filtered out by the liver), rarely found elsewhere but possible (numerous case reports out there reported in the brain, bones, spleen, kidneys, muscles etc.)

in humans, these cysts can remain in a person's body for years, causing no harm and no symptoms

they can grow by 1 cm per year in the liver

Cyst image from wiki

The tapeworm itself is incredibly eerie to look out, as per CDC website:

4 species of concern in people:

E. granulosus = cystic echinococcosis, or hydatid cysts

E mlutilocularis = alveolar

E. vogeli & oligarthrus = polycystic

Hosts:

generally dogs

intermediates: sheep, goats, pigs

humans are 'incidental or accidental hosts" via contact with a reservoir animal - that is to say, the parasite relies on the above 2 hosts for it's life cycle (not necessarily humans)

transmission: faecal oral, ingestion of eggs in from host faeces

larvae from the eggs penetrate the intestine and is transported in blood/lymphatics to liver (and if through liver to lungs), sometimes other organs

cysts = layers with a fibrous capsule, daughter cysts can bud off, can grow up to multiple litres and can septate and have daughter cysts over time, so may not remain unilocular. hosts may develop a granulomatous reaction

Clinical presentation

can be asymptoamtic for decades, as per 1 case report in a South Korean journal, cysts in one patient caused no issues for 30-40 years (after exposure during the Vietnam war)

become symptomatic when large enough to cause them (if in liver = nausea, vomiting, RUQ pain as per the case report, on exam: abdo distension, palpable mass and hepatomegaly, obstructive jaundice)

in cystic liver disease, it's considered a hydatid cyst and usually assiocaited with cystic echinococcosis

cysts can also eventually rupture and the fluid can cause extreme reactions such as anaphylaxis (IgE mediated Type 1 hypersensitivity reaction), leaking into the peritoneum = hydatidosis

65%: liver, 25% lungs, but 20-40% of patients can have multiorgan involvement

often acquired in childhood but present later in life (excpetion: eye and brain)

in lung: SOB, chest pain, chronic cough, haemoptysis

Rare (from stat pearls)

heart: tamponade

brain = raised ICP, seizures

spinal cord = compression

kidneys = haematuria, flank pain

bones = pathologic fractures

INcreased risk

a/w endemic areas - South America (as per case report above), former soviet union (kinda random), middle east, eastern med, Western China (I suppose these areas are all geographically connected), but also East Africa and Australia

however, alveolar echinococcosis is endemic only to Russia, China, North America and Europe

rural/grazing areas (not surprisingly given the hosts are dogs and sheep), communities needing dogs for herding

30-40 yos more likely to have hydatid cysts while > 50 are a/w alveolar

elderly and immunocompromised are more vulnerable

look for these RFs above (esp contact with dogs + cattle)

Investigations

imaging is definitive, look for the cysts, pretty big if symptomatic

nonspecific, but clues on bloods: mild eosinophilia, leukopaenia and thrombocytopaenia, deranged LFTs in hydatid cysts (abnormal in only 40% of patients, ALP usually high the rest can be normal)

imaging: USS, CT are sufficient, MRI great but no real advantage over CT, useful in the seronegative

serology is useful both in diagnosis and in follow-up after treatment, but many do not have an immune response depending on cyst wall intactness

avoids: aspiration, unless USS/CT guided given the risk of the fluid causing anaphylaxis

in the event the biliary tree is affected, await an ERCP

Stages determine management

WHO informal working group stages (IWGE) based on USS:

CE 1: simple cyst, double line sign

CE2: mlutiseptated, honeycomb/rosette

CE3A: watery lily sign - fluid collection with a detached membrane

CE3B: daughter cysts ina. solid matrix

CE4: heterogenous, hyperechoic, matrix, no duaghter cysts

CE5: solid cystic wall

1&2: active, 3 = transitional (cyst compromised), 4 and 5= inactive

ring like calcifications can be present in all stages

CT over USS = complications, like rupture, infection and biliary/vascular involvement

Treatment

albendazole - antiparasite medication is the foundation

generally depends on the size and number of cysts and there's whole WHO staging and guidelines for this

medical therapy can be anywhere from 1-3 months up to 6, but are futile if the sole treatment cysts > 5 cm or daughter cysts involved

Stage 4&5: watch and wait, USS regularly, these are uncomplicated

CE1 and CE3a: cysts < 5 cm in liver and lung, but inoperable and >2 organs involved = albendazole (CI: pregnancy and cysts vulnerable to rupture), also used following surgery and perc drainage (CT/USS guided) to prevent recurrence

surgery in: complicated cysts for removal of large stage 2 and 3b cysts or cysts that can rupture (superficial), infected, biliary tree communication and pressure on organ. Inovlves excision of cyst and possibly liver resection, contamination with daughter cysts is possible so adj albendazole and praziquantel often done (i.e. 1-4 weeks prior then 4 weeks post. has lower recurrence rates

PAIR = puncture, aspiration, inj with 20% hypertonic saline and re aspiration = USS guided in the inoperable or relapsing cases or failing to respond. usually in cysts > 5 cm with albendazole for 4 weeks, stage 1 only/single compartment cysts (PAIR ineffective in higher stages = multiple compartments)

post treatment: USS 3-6 monthly then yearly

lungs: 2 years of treatment and surgery, simple cysts may just require medical therapy

Differentials

straight from stat pearls - abscess, simple benign cysts, budd chiari, biliary colic, cirrhosis, TB, carcinoma, haemagiomas

Sources:

wikipaedia

statpearls

CDC guidelines

WHO

Australian guidelines (behind a paywall)

[helliontherapscallion]

Axolotl names in my mcpe world that just make sense: Glizzybobizzymomizzy (cyan), Egg (cyan), Mushu (pink), Jaundice (gold), and Mac n Cheese (gold)

[doctorfoxtor]

the way it makes me feel like, the way it makes me feel i never seem to act so stupid... oh here we go

100 days of productivity

day 46 + 47

CVS/RS

vitamin A oversupplementation is actually assoc w/ ↑risk of ca lung

MTX lung tox occurs within 4 months of starting, but rheumatoid organising pneumonia can occur at any point in the disease

inhaled steroids have one major systemic side effect and it is skin changes (thinning, fragility, easy bruising)

transbronchial biopsy is useless in IPF; only fibrosis w/ nothing to suggest aetiology will be seen, so take a surgical biopsy if needed

severe mitral stenosis → percutaneous mitral commissurotomy (if not suitable, transcatheter replacement)

CNS/Ophthal/Psych

low CSF pressure headache: LP, but also minor trauma to meninges, idiopathic (insidious); orthostatic headache, N/V, ↔ diplopia, ataxia/vertigo, stiff neck, back pain, VF abnormalities; ↓CSF opening pressure; MRI: diffuse pachymeningeal enhancement, tonsilar descent, post fossa crowding; conservative → epidural blood patch

vitreous haemorrhage in DM is an indication for photocoagulation and a hallmark of proliferative disease

vitamin E def → cerebellar ataxis, dysarthria!, ± myopathy

fluctuating eyesight in DM → fluctuating ocular glucose leading to variable osmotic pressures → fluctuating lens hydration → fluctuating lens shape

schizophrenia spectrum: if tx is started, it is effective if there is marked improvement within 2 wks and complete remission within 4-6 wks

Rheum/Derm/Immuno

lichen sclerosus → atrophy, 'cigarette paper' texture of indurated plaques, marked hypopigmentation

morphoea = 'localised scleroderma' → indurated annular plaques w/ advancing inflammatory edge and central atrophy

erythema nodosum causing drugs: omeprazole, sulphonamides, halides (iodides/bromides), gold (aurothiocyanate), HBV vaccine

Igs by serum concentration: G 75% > A 15% > M 10% > D 1% > E <0.1% (all approx)

GIT

vitamin B6 and D are assoc w/ reduced ca colon

autoimmune hepatitis: type 1 = nonspecific progressive fatigue w/ ↑LFTs, anti-smooth muscle; type 2 = acute episodes of jaundice, anti-LKM1

acute flare of IBD not responding to steroids: cyclosporin, infliximab take rapid effect (AZP/6-MP take weeks)

Onc/Haem

myeloma: thalidomide → bortezomib → lenalidomide; melphalan in very frail pts; cyclophosphamide as adjunct

immune thrombocytopaenia (idiopathic and lupus): platelet transfusion only if active bleeding AND plt <30k/mcl, otherwise can manage medically (steroids for ITP, HCQS → AZP/6-MP/ciclosporin for SLE)

Renal/Biochem

allopurinol actually has a role in reducing progression to diabetic nephropathy in diabetics w/ gout

vitamin E oversupplementation is assoc w/ ↑risk of ca prostate

ACEi+ARB together show benefit in only one condition: nephrotic-range proteinuria; otherwise, increased risk of renal damage and no clear benefit

Pharm/Toxo/Misc

specifically, slow acetylator status is assoc w/ N-acetyltransferase-2

incidentally, fast- status is assoc w/ INH-hepatits, characterised by microvascular fatty infiltration and necrosis

AZP can be continued in pregnancy; it is assoc w/ very light LBW but it is not otherwise life threatening or teratogenic (but withdrawing tx will be problematic for mother)

DNA polymorphism vs mutation: 1% in population is cutoff

[duggardata]

Baby Layla is Back In The Hospital

According to Carlin’s IG Story, Layla had to return to the hospital last night. Based on the photos, it looks like she (Layla) is receiving phototherapy—and thus, is likely suffering from infant jaundice. Jaundice is a pretty common issue for babies, especially pre–term babies. (Note—Despite arriving early, Layla was not pre–term.) Jaundice can cause brain damage in bad cases, but with prompt treatment—like Layla seems to be receiving—there’s usually no long–term effect.

Sending good thoughts to Carlin + Evan and Layla.