#long qt syndrome type 1

[aanews69]

Panera SETTLES wrongful death suit Update on The Ivy League student with heart condition died after consuming the highly caffeinated drink. #PaneraSettlement #SarahKatz #WrongfulDeathLawsuit #ChargedLemonade #CaffeineHealthRisk #Panera #paneralemonade Subscribe👇: https://vist.ly/3miamtv 👀👇: https://vist.ly/3miamtu

[meret118]

college student with a heart condition, Sarah Katz, died after drinking a heavily-caffeinated drink from Panera; her parents assert in the suit that Katz likely thought the beverage would be safe to drink because the chain’s marketing of the product does not reflect its high caffeine content.

The large size of Panera’s large Charged Lemonade, per the New York Times, has more caffeine than a 12-ounce Red Bull and a 16-ounce Monster Energy Drink combined. However, it is advertised as being “clean.” Katz had Long QT Type 1 Syndrome, a heart condition that necessitated avoiding highly caffeinated beverages. “If she didn’t know that this was an energy drink, it makes the family concerned about who else doesn’t know,” Elizabeth Crawford, the Katz fa

[blog-site-for-market-research]

Forecasting the Size of Long QT Syndrome Market

Market Overview –

The long QT syndrome market is anticipated to grow at a 5.2% CAGR from 2022 to 2030, or USD 3,933.02 million.

The Long QT Syndrome (LQTS) market encompasses medical interventions and devices designed to diagnose, treat, and manage this cardiac disorder characterized by abnormal heart rhythms. LQTS increases the risk of sudden cardiac arrest or arrhythmias, posing serious health threats. The market includes genetic testing, medications, implantable cardioverter-defibrillators (ICDs), and lifestyle modifications aimed at reducing the risk of life-threatening events.

The Long QT Syndrome market is characterized by advancements in understanding and managing this cardiac disorder. With a focus on long QT disorder, healthcare providers emphasize early diagnosis and personalized treatment approaches to mitigate the risk of life-threatening arrhythmias. As awareness grows, there's a rising demand for innovative therapies and genetic testing, driving market growth and research endeavors.

A key driver of the Long QT Syndrome market is the growing awareness of inherited cardiac disorders and advances in genetic testing technology. Early detection through genetic screening allows for timely interventions, such as medication adjustments or implantation of ICDs, to prevent cardiac events and improve patient outcomes.

Advancements in medical devices, such as miniaturized ICDs and wearable cardiac monitors, have revolutionized the management of Long QT Syndrome. These devices provide continuous monitoring and early detection of arrhythmias, offering peace of mind to patients and caregivers while improving overall safety.

The COVID-19 pandemic has impacted the Long QT Syndrome market, with disruptions in routine medical care, delayed diagnoses, and challenges in accessing specialized cardiac services. However, telemedicine and remote monitoring technologies have emerged as valuable tools in managing Long QT Syndrome, enabling patients to receive timely care and support from healthcare providers.

Segmentation –

The global long QT syndrome market is segmented on the basis of type, diagnosis, treatment, and end users. On the basis of the type, the market is segmented into long QT syndrome type 1, long QT syndrome type 2, long QT syndrome type 3, and others.  On the basis of the diagnosis, the market is categorized into tests, electrocardiogram (ECG), genetic testing, and others.

On the basis of the treatment, the market is segmented into medication, surgical procedures, and others.  On the basis of the end user, the market is segmented into hospitals & clinics, diagnostic labs, research organizations, and others.

Regional Analysis –

Regional analysis of the Long QT Syndrome (LQTS) market reveals variations in diagnosis rates, treatment options, and healthcare infrastructure across different regions. In developed regions like North America and Europe, where there is greater awareness of genetic disorders and access to specialized cardiac care, the market for LQTS diagnostics and management is well-established, with genetic testing, beta-blockers, and implantable cardioverter-defibrillators (ICDs) being standard of care.

Conversely, in developing regions with limited access to advanced cardiac diagnostics and therapies, such as parts of Africa and Asia-Pacific, the market for LQTS is still emerging, with challenges related to underdiagnosis and limited treatment options. Moreover, cultural attitudes towards genetic testing and preventive healthcare influence patient behaviors and healthcare-seeking patterns across different regions. As awareness of LQTS and its genetic basis increases globally, there is a growing opportunity for market expansion through education, advocacy, and investment in cardiac care infrastructure to improve outcomes for LQTS patients worldwide.

Key Players –

Long QT syndrome key players include Invitae Corporation (U.S.), GeneDx (U.S.), Asper Biogene (Estonia), Boston Scientific Corporation (U.S.), Laboratory Corporation of America Holdings (U.S.), Pfizer Inc. (U.S.), Zydus Pharmaceuticals, Inc. (U.S.), Aralez Pharmaceuticals Inc. (Canada), AstraZeneca (U.K.), Torrent Pharmaceuticals Limited (India), Lupin Pharmaceuticals, Inc. (U.S.), Cipla Inc. (India), Mylan N.V. (U.S.), Teva Pharmaceutical Industries Ltd. (Israel), and others.

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[drramjimehrotra]

(Dr Ramji Mehrotra) What are Inherited Cardiac Conditions?

Inherited cardiac conditions are a group of cardiovascular disorders that are caused by genetic mutations or abnormalities passed down from parents to their children. These conditions can affect the structure or function of the heart and may occur at various stages of life, from infancy to adulthood. Understanding the nature of inherited cardiac conditions is crucial for early detection, proper management, and preventive measures.

One prominent example of an inherited cardiac condition is hypertrophic cardiomyopathy (HCM). HCM is characterized by the thickening of the heart muscle, particularly in the left ventricle. This abnormal thickening can disrupt the heart's pumping ability and lead to symptoms such as chest pain, shortness of breath, and palpitations. In some cases, HCM can cause sudden cardiac arrest, especially during intense physical activity.

According to Dr. Ramji Mehrotra, leading cardiac surgeon in NCR region, genetic mutations in genes responsible for the structure of cardiac muscle proteins are often associated with HCM. Diagnosis typically involves a combination of clinical evaluation, imaging tests like echocardiography, and genetic testing to identify specific mutations. Management may include medications to alleviate symptoms, lifestyle modifications, and, in severe cases, surgical interventions.

Another inherited cardiac condition is Long QT syndrome (LQTS), which affects the electrical system of the heart. Individuals with LQTS have an abnormality in the ion channels responsible for regulating the heart's electrical activity. This can lead to irregular and potentially life-threatening arrhythmias, including a specific type known as Torsades de Pointes.

Symptoms of LQTS may include fainting, seizures, and sudden cardiac arrest. Genetic testing can identify mutations associated with LQTS, and electrocardiograms (ECGs) are used to evaluate the heart's electrical activity. Treatment typically involves medications to prevent arrhythmias, lifestyle modifications, and the avoidance of certain medications that can prolong the QT interval.

Arrhythmogenic right ventricular cardiomyopathy (ARVC) is an inherited cardiac condition that affects the structure and function of the heart's right ventricle. In ARVC, the normal muscle tissue in the right ventricle is progressively replaced by fatty or fibrous tissue, leading to abnormal heart rhythms and, in some cases, heart failure. Genetic mutations involving proteins responsible for maintaining the structural integrity of heart cells are often associated with ARVC. Diagnosis may involve a combination of clinical evaluation, imaging tests like cardiac MRI, and genetic testing. Treatment typically focuses on managing arrhythmias, preventing sudden cardiac death, and addressing heart failure symptoms through medications, implantable devices like defibrillators, and lifestyle modifications.

Marfan syndrome is an inherited connective tissue disorder that can also affect the cardiovascular system. It is caused by mutations in the FBN1 gene, which leads to abnormalities in the production of the protein fibrillin-1. Fibrillin-1 is a crucial component of connective tissue that provides strength and elasticity to various structures, including the heart's valves and aorta.

In individuals with Marfan syndrome, the aorta can be weakened and dilated, increasing the risk of aortic dissection or rupture. Diagnosis involves clinical evaluation, imaging tests like echocardiography, and genetic testing to confirm the presence of FBN1 mutations. Treatment may include medications to manage symptoms, regular monitoring of the aorta's size, and surgical interventions to repair or replace the affected valves or aorta if necessary.

Dr. Ramji Mehrotra says that understanding the genetic basis of inherited cardiac conditions, along with appropriate diagnostic strategies and management approaches, is crucial for early detection, prevention of complications, and optimal care.

[cpucsandtrains]

A medical youtuber named Medlife Crisis, who has a video about Long Qt Syndrome, (type 2), a heart disease that can literally make you die of fright, ends the video with a LOUD, and sudden, phone ring. Earlier on in the video, he straight up used someone getting a call from their mom and dying from it as an example. Additionally, he says that about 1 in 7000 people have Long Qt Syndrome.

That video currently has 68k views. While there does appear to be treatment for it, the fact that he would even do that in the first place could still mean that someone with it would’ve died FROM A VIDEO ABOUT IT.

Hell, someone with it could’ve looked up videos about their own condition, found this, and died because of it.

This is made even worse by the fact that, while I don’t know much in depth about the disease, is something that some people might dismiss as extreme anxiety. Even if I am wrong on that, it’s still something that legitimately might’ve caused a few deaths.

And in the comments, his only response, as far as I can tell, to people talking about how that could’ve actually killed someone, is this:

I don’t want to say that jump scares in general should be banned or anything, but when it’s on a video about how something like that could kill someone is when I think completely crosses the line. Note: While this is important, YOU ARE NOT A BAD PERSON IF YOU DON’T REBLOG IT. I want to make that very clear.

[falloutnewvegastransedmygender]

So they changed their tune after a wrongful death suit. There's actually a second one too, which is the one I heard about first. They also dropped the 30 oz to 237mg of Caffiene according to some stories but I'm not sure if that was after the first (October) suit or the second (December) suit.

Panera Bread is evidently taking a page out of the Cocaine Era of Coca-Cola

Caffeine In Beverages (See Google)

20 fl oz. Coke | 53 mg

8 fl. oz. Coffee | 95 mg

16 fl oz. Monster | 160 mg

20 fl. oz. Panera* | 260 mg

30 fl. oz. Panera* | 389 mg

*Panera Charged Lemonade Mango Yuzu Citrus

Maximum Recommended Daily Intake for a Healthy Adult 400 mg (Caffeine has a half life in the body of about 4-6 hours if what I saw online was correct.)

Companies with the American Beverage Association are not legally required to add this label (the FDA asked nicely so they could avoid expensive courts) and it looks as follows.

I should note the FDA does require labels to say if caffeine is present.

This is how the Charged Lemonades are served at Panera

Notice the labels only list the amount, absolutely zero further information. They are also only labeled as "Charged Lemonade" and not as Energy Drinks, which they actually contain significantly more caffeine than.

Be Informed and Be Aware

This info blew up over on TikTok after a woman made a video after having 4-5 of them without noticing the caffeine content. She had been doing this for several days it seems, going there and working on her laptop while drinking her Lemonade. When I say to you in the video she looked like her body was about to short circuit? Yeah. Video is linked below.

Now, back to my initial statement, why do I mention back when Coke had Coke? Well because caffeine is addictive, I'm no puritan about caffeine myself, but I at least know what I'm getting myself into with my tea, coke, and occasionally a Monster (about 1-3 a month). Furthermore it's a stimulant and can make you feel more awake and productive. Finally the larger sizes just barely skirt that max intake limit. I believe this is intentional, to bring customers back again and again.

https://www.tiktok.com/t/ZTRsaCEAk/

[nuedextainfo]

What You Need To Know About Nuedexta

Nuedexta for treatment of Pseudobulbar affect (PBA) and other uses. 

The drug known as Nuedexta is made up of a combination of dextromethorphan hydrobromide/ quinidine sulfate which is prescribed for treating PBA (pseudobulbar affect). This medication was developed and manufactured by a company by the name of Avanir Pharmaceuticals. This company received authorization to market Nuedexta from the EC (European Commission) in June 2013, for treating the condition PBA. Avanir Pharmaceuticals obtained approval from the FDA (US Food and Drug Administration) for Nuedexta for treating PBA in the month of October 2010.

Symptoms Of Pseudobulbar Effect

PBA which is short for Pseudobulbar Affect is classified as a type of neurological disorder that emanates from a neurological condition like Parkinson's disease, amyotrophic lateral sclerosis, multiple sclerosis, stroke or a traumatic injury to the brain. This condition is the cause of uncontrolled and sudden episodes of laughing or crying involuntarily. This condition has been estimated to affect over 1 million people across the U.S.

The Mechanism Of Action Associated With Nuedexta

Nuedexta is made up of a mixture of two medications, which includes quinidine sulfate and dextromethorphan hydrobromide. Exact mechanisms of action associated with the drug is still not completely known, yet it is said that it works in the way of regulating the excitatory neurotransmissions that occur through the Sigma-1 Receptor agonist activity along with the NMDA receptor antagonist activity. This medication also works on slowing the breakdown of a chemical known as dextromethorphan found inside the brain. Nuedexta drug is in a class that includes opium alkaloids along with its derivatives.

Side Effects Of Nuedexta

To date, a number of side effects regarded as serious have been reported with this medication. Some of the common types of side effects linked to Nuedexta include:

- Vomiting

- Diarrhea

- Fainting/dizziness

- Stomach pain

- Difficulties with breathing

-Irregular or fast heartbeat

- Cough

- Fever

- Weakness

- Swelling of the lower legs, ankles, feet or hands

- Muscle spasms

- Painful, difficult or frequent urination

This list is not the complete version of Nuedexta side effects. It is advisable to ask for advice medically or call a doctor if you are experiencing side effects that are not going away.

Nuedexta Interactions

It is important that you let your physician know about any other non-prescription and prescription medications that you are taking, this also includes herbal supplements and vitamins. MAO inhibitors shouldn't be taken with or within 2 weeks after or before taking Nuedexta.  

Important Information

This medication should not be taken in patients who have experienced heart failure, a heart condition known as AV block, or you have a history associated with Long AT syndrome or heart rhythm disorders. You should also not take Nuedexta when you have used MAO inhibitors within the last 14 days. This can result in a dangerous interaction. The MAO inhibitors can include linezolid, isocarboxazid, phenelzine, methylene blue injection, rasagiline, phenelzine, tranylcypromine, selegiline, tranylcypromine among others. You should also refrain from using Nuedexta if you currently take quinine, mefloquine, or quinidine or you have in the past experienced allergic reactions or severe medical issues by taking one or more of these drugs.

Certain drugs are able to interact with Nuedexta, which is why they should never be used together. You need to disclose to your doctor about any medication that you use, along with those that you stop or start using over the course of your treatment with Nuedexta. This medication can be dangerous if you decide to use it while taking Quin-G, Lariam or Qualaquin, or when you have experienced allergic reactions.

Before You Take This Medicine

- Nuedexta should not be a medication that you should be using when you have already had an allergic reaction to quinidine or dextromethorphan. You should also not be using Nuedexta if you have experienced heart failure.

- A history linked to a life-threatening heart-rhythm disorder.

- A history linked to Long QT syndrome.

- You have been diagnosed with a heart condition known as AV Block unless you now have a pacemaker.

- If you are already taking quinine, quinidine or mefloquine.

Nuedexta usually starts with a 1 capsule dose every day at the same time for a period of 7 days. In the second week, you will start taking 1 capsule twice a day (every 12 hours).

[mcatmemoranda]

It’s hard to remember all the antiarrhythmic drugs because of this classification system. But there’s no other way. 😩 Drugs that prolong phase 0 in ventricular and atrial myocytes also prolong (widen) the QRS complex. Drugs that prolong phases 1 to 3 prolong the QT interval, which can lead to Torsades de Pointes. Drugs that slow conduction through the AV node (CCBs and beta blockers) can cause heart block (first degree or Mobitz type 1/Wenckebach). AV nodal block = increased PR interval (it’s taking longer for the impulse to go through the AV node).

Blocking Na+ channels prolongs phase 0, which widens the QRS complex. Blocking K+ channels prolongs phases 1-3, which prolongs the QT interval. Slowing AV node conduction = prolonged PR interval.

Class Ic only affects the Na+ channels. Class Ia blocks Na+ and K+ channels. Class Ib shrinks action potential duration and effective refractory period, but doesn’t affect QRS complex.

Class Ib binds Na+ channels better when the HR is fast because these drugs bind better to Na+ channels in depolarized tissue. Faster HR means depolarization is happening faster, so more drug can bind and thus you get increased toxicity. So class Ib drugs have increased toxicity if the HR is fast. All the class I antiarrhythmics exhibit use dependence, meaning the drugs bind better to Na+ channels that are in use (open or inactivated). In ischemic tissue, the myocytes are more depolarized because they can't maintain the -85 mV membrane potential. Thus, class Ib drugs work best for ischemic tachycardia.

Amiodarone has the lowest risk of causing torsades de pointes of all the class III drugs. It also has class I, II, and IV effects, so it’s very effective. It has the most effects of all the antiarrhythmics. It is lipid soluble, so it takes a long time to be eliminated from the body. It has a lot of adverse effects--the one I remember most and the reason pts can die from amiodarone is pulmonary fibrosis. It has iodine in it, so it can cause hypothyroidism or hyperthyroidism. It has worse side effects the longer you use it, so it’s not given to young pts. It also causes photosensitivity, increased LFTs, blue man syndrome (blue face), corneal deposits (look like cat whiskers; benign effect). So you need to do baseline LFTs, PFTs, thyroid tests, CXR before starting pts on amiodarone.

Sotalol and dofetilide especially cause QT interval prolongation. Sotalol has beta block effects too. Sotalol and dofetilide work best when K+ channels are at rest, which occurs during bradycardia. This is reverse use dependence. So sotalol and dofetilide work best in pts with bradycardia; that also means increased risk of toxicity in such pts. Ibutilide may be given IV for chemical cardioversion but it has lots of side effects.

Class II (beta blockers) work on myocytes in the SA and AV nodes. So they can slow the HR. Recall that the action potential in pacemaker cells looks different from the one for ventricular or atrial myocytes. The action potential for pacemaker cells has phase 4, phase 0, and phase 3. Beta blockers prolong phase 4 (resting potential, funny current). Beta blockers make the cell take longer to depolarize. So they decrease HR and conduction through AV node. Decreased conduction through AV node = PR prolongation, which can cause AV nodal block. Unlike other antiarrhythmics, beta blockers improve mortality.

Class IV = Calcium Channel Blockers (CCBs). Block Ca2+ channels in phase 0-> slow HR & AV node conduction. Also prolong PR interval. Same effect as beta blockers.

Adenosine binds to purinergic receptors in the AV node and vascular smooth muscle. In AV node, it activates K+ channels, hyperpolarizing myocytes. It also blocks Ca2+ influx. Slows AV node conduction. Treats SVT (like AVNRT). Theophylline and caffeine block adenosine receptors. In the vascular smooth muscle, adenosine causes vasodilation. So it's normal for adenosine to cause flushing, dyspnea, and CP, which resolves rapidly.

Magnesium can help manages torsades because it blocks Ca2+ influx into cells. Ca2+ influx into myocytes causes early afterdepolarizations, which cause torsades. So blocking Ca2+ stops the early afterdepolarizations.

Atropine is a muscarinic receptor antagonist. I always remember that it's used to block the parasympathetic nervous system; so people who have been poisoned by nerve gas have overactivation of the parasympathetic nervous system (lacrimation, diarrhea) and atropine blocks that. Atropine blocks the parasympathetic nervous system--> increased conduction through AV node, so it treats bradycardia due to AV block by increasing HR and impulse conduction through AV node. Since it blocks PSNS, AEs include dry mouth, constipation, urinary retention, and confusion in elderly pts.

Digoxin blocks the Na+/K+ ATPase. This stops the pumping of K+ into the cell and Na+ out of the cell. The increased Na+ in the cell will make it so that the Na+/Ca2+ exchanger will stop pumping Na+ into the cell in exchange for Ca2+ out of the cell. Thus there will be more Ca2+ in the cell--> increased contractility. Digoxin increases parasympathetic tone--> suppressed AV node conduction. So digoxin increases contractility and slows conduction through AV node. Digoxin competes with K+ for binding to the Na+/K+ ATPase, so when you have hypokalemia, there's less K+ for digoxin to compete with and thus increased effects of digoxin--> increased toxicity. So loop diuretics like furosemide, which cause K+ loss would increase digoxin toxicity. Digoxin AEs: N/V, abdominal pain, lethargy, fatigue, scotomas, loss of color vision, blindness, cardiac arrhythmias. Digoxin causes more Na+ to be inside myocytes, so their membrane potential is more positive and they can spontaneously depolarize (automaticity)--> extra beats. Digoxin can cause hyperkalemia since it can poison the Na+/K+ ATPase--> too much extracellular K+. Digibind is the antidote; it's an antibody (digoxin antigen binding fragment, Fab). Digibind treats the hyperkalemia.

[chronic-confessions]

Confession #4,511

Hi, i have Long QT syndrome type 1 and i was diagnosed at age 13 despite it being genetic. is it bad that i think i’m lucky that i’m asymptomatic? But then Like i love my friends and going out with them and doing stupid things or being stupid with them but then i remember the faulty ticker and say sorry guys i’m gonna sit out for a bit and get eye rolls in response or i get asked if i want kids and then get judgemental looks for saying no because the heart conditions genetic but i don’t show it

[lisanealblog-blog]

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Before taking this medicine 

You ought not to use Levitra in the event that you are susceptible to vardenafil, or on the off chance that you take different medications to treat pneumonic blood vessel hypertension, such as riociguat.

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Long QT syndrome

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Levitra side effects 

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Global Cardiovascular Partnering 2014-2021: Deal trends, players and financials

Bharat Book Bureau Provides the Trending Market Research Report on “Global Cardiovascular Partnering 2014-2021: Deal trends, players and financials” under  Life Sciences Category. The report offers a collection of superior market research, market analysis, competitive intelligence and Market reports.

Global Cardiovascular Partnering 2014 to 2021 provides the full collection of Cardiovascular disease deals signed between the world’s pharmaceutical and biotechnology companies since 2014.

Trends in Cardiovascular partnering deals Financial deal terms for headline, upfront and royalty by stage of development Cardiovascular partnering agreement structure Cardiovascular partnering contract documents Top Cardiovascular deals by value Most active Cardiovascular dealmakers

Most of the deals included within the report occur when a licensee obtains a right or an option right to license a licensor’s product or technology. More often these days these deals tend to be multi-component including both a collaborative R&D and a commercialization of outcomes element.

The report takes readers through the comprehensive Cardiovascular disease deal trends, key players and top deal values allowing the understanding of how, why and under what terms companies are currently entering Cardiovascular deals.

The report presents financial deal terms values for Cardiovascular deals, where available listing by overall headline values, upfront payments, milestones and royalties enabling readers to analyse and benchmark the value of current deals.

The initial chapters of this report provide an orientation of Cardiovascular dealmaking trends.

Chapter 1 provides an introduction to the report.

Chapter 2 provides an overview of the trends in Cardiovascular dealmaking since 2014 covering trends by year, deal type, stage of development, technology type and therapeutic indication.

Chapter 3 includes an analysis of financial deal terms covering headline value, upfront payment, milestone payments and royalty rates.

Chapter 4 provides a review of the leading Cardiovascular deals since 2014. Deals are listed by headline value. The chapter includes the top 25 most active Cardiovascular dealmakers, together with a full listing of deals to which they are a party. Where the deal has an agreement contract published at the SEC a link provides online access to the contract.

Chapter 5 provides comprehensive access to Cardiovascular deals since 2014 where a deal contract is available, providing the user with direct access to contracts as filed with the SEC regulatory authorities. Each deal title links via Weblink to an online version of the deal record contract document, providing easy access to each contract document on demand.

Chapter 6 provides a comprehensive directory of all Cardiovascular partnering deals by specific Cardiovascular target announced since 2014. The chapter is organized by specific Cardiovascular therapeutic target. Each deal title links via Weblink to an online version of the deal record and where available, the contract document, providing easy access to each contract document on demand.

In addition, a comprehensive appendix is provided with each report of all Cardiovascular partnering deals signed and announced since 2014. The appendices are organized by company A-Z, stage of development at signing, deal type (collaborative R&D, co-promotion, licensing etc) and technology type. Each deal title links via Weblink to an online version of the deal record and where available, the contract document, providing easy access to each contract document on demand.

The report also includes numerous tables and figures that illustrate the trends and activities in Cardiovascular partnering and dealmaking since 2014.

In conclusion, this report provides everything a prospective dealmaker needs to know about partnering in the research, development and commercialization of Cardiovascular technologies and products.

Report scope

Global Cardiovascular Partnering 2014-2021: Deal trends, players and financials is intended to provide the reader with an in-depth understanding and access to cardiovascular trends and structure of deals entered into by leading companies worldwide.

Global Cardiovascular Partnering 2014-2021: Deal trends, players and financials includes:

Trends in cardiovascular dealmaking in the biopharma industry since 2014 Access to headline, upfront, milestone and royalty data Access to over 850 cardiovascular deal records The leading cardiovascular deals by value since 2014

The report includes deals for the following indications: Abdominal aortic aneurysm, Angina, Arrhythmia, Atrial fibrillation, Long QT syndrome, Supraventricular Tachycardia, Ventricular fibrillation, Ventricular tachycardia, Atherosclerosis, Breathlessness, Cardiogenic shock, Cardiomyopathy (heart muscle disease), Chest pain, Congenital heart disease, Congestive heart failure, Coronary artery disease, Ductus arteriosus, Fatigue, Hypercholesterolemia, Hypertension, Intermittent claudication, Ischemic heart disease, Limb ischemia, Marfan's Syndrome, Myocardial Infarction, Oedema (excess fluid), Palpitations, Peripheral arterial disease, Thrombus (blood clot), Valvular heart disease, Aortic stenosis, Restenosis, Varicose veins, plus other cardiovascular indications.

In Global Cardiovascular Partnering 2014-2021: Deal trends, players and financials, the available deals are listed by:

Company A-Z Headline value Stage of development at signing Deal component type Specific therapy target

Each deal title links via Weblink to an online version of the deal record and where available, the contract document, providing easy access to each contract document on demand.

The Global Cardiovascular Partnering 2014-2021: Deal trends, players and financials report provides comprehensive access to available deals and contract documents for over 850 cardiovascular deals.

Browse our full report with Table of Content : https://www.bharatbook.com/report/897789/global-cardiovascular-partnering-deal-trends-players-and-financials About Bharat Book Bureau: Bharat Book is Your One-Stop-Shop with an exhaustive coverage of 4,80,000 reports and insights that includes latest Market Study, Market Trends & Analysis, Forecasts Customized Intelligence, Newsletters and Online Databases. Overall a comprehensive coverage of major industries with a further segmentation of 100+ subsectors.

Contact us at: Bharat Book Bureau Tel: +91 22 27810772 / 27810773 Email: poonam@bharatbook.com Website: www.bharatbook.com

[techscimarketresearch]

United States Pacemaker Market to Register Healthy Growth through 2025 | TechSci Research

Increasing cases of cardiovascular diseases and technological advancements in healthcare to drive United States pacemaker market.

According to TechSci Research report, “United States Pacemaker Market By Type (MRI Compatible Pacemakers, Conventional Pacemakers), By Product Type (Implantable Pacemaker, External Pacemaker), By Application (Heart Block, Arrhythmia, Atrial Fibrillation, Bradycardia, Tachycardia,  Long QT Syndrome), By Technology (Single Chamber, Dual Chamber, CRT-P/Biventricular Pacemaker), By End User (Hospitals & Cardiac Centers, Ambulatory Surgical Centers), By Region, Forecast & Opportunities, 2025”, the market is anticipated to grow at a significant CAGR during the forecast period. Key factors propelling the growth of United States pacemaker market include increasing prevalence of cardiovascular diseases and the spread of COVID-19, which has led to high spread of CVDs among patients suffering from coronavirus. The increasing demand for minimally invasive procedures is also creating high demand for pacemakers in United States market. Furthermore, technological advancements and launch of new products such as leadless pacemakers and MRI safe pacemakers are expected to propel the market growth in coming years. Also, favorable government policies and increasing expenditure on healthcare sector are supporting the growth of United States pacemaker market.

Additionally, favorable reimbursement procedures such as Medicare system coupled with rise in manufacturing high-end pacemakers, which caters to customized care are fueling the growth of United States pacemaker market. Along with this, growing geriatric population who are more vulnerable to cardiovascular diseases is also acting as a major growth driver for this market. The advent of battery free pacemakers has brough revolutionary change in the market and is augmenting the market growth. With increasing FDA approval for novel pacemakers, the market is expected to witness substantial growth through 2025. However, pacemaker devices cost two to six times more in the United States than in European countries such as Germany. This might hinder the United States pacemaker market growth.

Browse XX market data Tables and XX Figures spread through XX Pages and an in-depth TOC on "United States Pacemaker Market"

https://www.techsciresearch.com/report/united-states-pacemaker-market/5173.html

The United States pacemaker market is segmented based on type, product type, application, technology, end user and region. Based on application, the market can be segmented into heart block, arrhythmia, atrial fibrillation, bradycardia, tachycardia, and long QT syndrome. Among them, atrial fibrillation is expected to hold a significant share in United States pacemaker market during the forecast period. According to CDC, around 2.7–6.1 million people in the United States have atrial fibrillation, which is the most common type of heart arrhythmia. The number is expected to increase over the coming years.

Based on technology, United States pacemaker market can be segmented into single chamber, dual chamber, and CRT-P/biventricular pacemakers. Among them, dual-chamber pacemakers held significant share in United States market in 2019, owing to reduced post-surgical complications and presence of two leads which assure normal physiology of the heart. However, CRT-P, also known as biventricular pacemakers, are expected to register fastest growth in United States pacemaker market during the forecast period as they are increasingly being used to treat people with arrhythmias caused by advanced heart failure. These pacemakers contain three leads which are connected to the right atrium and both ventricles.

Major players operating in the United States pacemaker market include Biotronik, Inc., Boston Scientific Corporation, Abbot Inc., Zoll Medical Corporation, Oscor Inc., Medtronic, Inc., Cordis, Inc., Livanova PLC, Medico Inc., Oscor Inc. and Osypka Medical Inc. Leading companies are undertaking growth strategies such as new product launches, mergers & acquisitions, and partnerships.

Download Sample Report @ https://www.techsciresearch.com/sample-report.aspx?cid=5173

Customers can also request for 10% free customization on this report.

“The introduction of U.S. Food and Drug Administration (FDA)-cleared MRI-conditional pacemaker models are a growing trend in United States. Around 20% of pacemaker patients need an MRI within the first two years of implant. Pacemakers which do not have MRI-conditional use technology, generally prevent patients from being able to get an MRI. With increasing cases of cardiovascular diseases in the country, more manufacturers are coming up with advanced products and are using MRI-conditional use technology,” said Mr. Karan Chechi, Research Director with TechSci Research, a research based global management consulting firm.

“United States Pacemaker Market By Type (MRI Compatible Pacemakers, Conventional Pacemakers), By Product Type (Implantable Pacemaker, External Pacemaker), By Application (Heart Block, Arrhythmia, Atrial Fibrillation, Bradycardia, Tachycardia,  Long QT Syndrome), By Technology (Single Chamber, Dual Chamber, CRT-P/Biventricular Pacemaker), By End User (Hospitals & Cardiac Centers, Ambulatory Surgical Centers), By Region, Forecast & Opportunities, 2025” has evaluated the future growth potential of United States pacemaker market and provides statistics & information on market size, structure and future market growth. The report intends to provide cutting-edge market intelligence and help decision makers take sound investment decisions. Besides, the report also identifies and analyzes the emerging trends along with essential drivers, challenges, and opportunities in United States pacemaker market.

Contact

Mr. Ken Mathews

708 Third Avenue,

Manhattan, NY,

New York – 10017

Tel: +1-646-360-1656

Email: sales@techsciresearch.com

Website: https://www.techsciresearch.com/

For More Market Research Blogs Visit: https://techsciblog.com/

[techsciresearch]

United States Pacemaker Market to Register Healthy Growth through 2025 | TechSci Research

Increasing cases of cardiovascular diseases and technological advancements in healthcare to drive United States pacemaker market.

According to TechSci Research report, “United States Pacemaker Market By Type (MRI Compatible Pacemakers, Conventional Pacemakers), By Product Type (Implantable Pacemaker, External Pacemaker), By Application (Heart Block, Arrhythmia, Atrial Fibrillation, Bradycardia, Tachycardia,  Long QT Syndrome), By Technology (Single Chamber, Dual Chamber, CRT-P/Biventricular Pacemaker), By End User (Hospitals & Cardiac Centers, Ambulatory Surgical Centers), By Region, Forecast & Opportunities, 2025”, the market is anticipated to grow at a significant CAGR during the forecast period. Key factors propelling the growth of United States pacemaker market include increasing prevalence of cardiovascular diseases and the spread of COVID-19, which has led to high spread of CVDs among patients suffering from coronavirus. The increasing demand for minimally invasive procedures is also creating high demand for pacemakers in United States market. Furthermore, technological advancements and launch of new products such as leadless pacemakers and MRI safe pacemakers are expected to propel the market growth in coming years. Also, favorable government policies and increasing expenditure on healthcare sector are supporting the growth of United States pacemaker market.

Additionally, favorable reimbursement procedures such as Medicare system coupled with rise in manufacturing high-end pacemakers, which caters to customized care are fueling the growth of United States pacemaker market. Along with this, growing geriatric population who are more vulnerable to cardiovascular diseases is also acting as a major growth driver for this market. The advent of battery free pacemakers has brough revolutionary change in the market and is augmenting the market growth. With increasing FDA approval for novel pacemakers, the market is expected to witness substantial growth through 2025. However, pacemaker devices cost two to six times more in the United States than in European countries such as Germany. This might hinder the United States pacemaker market growth.

Browse XX market data Tables and XX Figures spread through XX Pages and an in-depth TOC on "United States Pacemaker Market"

https://www.techsciresearch.com/report/united-states-pacemaker-market/5173.html

The United States pacemaker market is segmented based on type, product type, application, technology, end user and region. Based on application, the market can be segmented into heart block, arrhythmia, atrial fibrillation, bradycardia, tachycardia, and long QT syndrome. Among them, atrial fibrillation is expected to hold a significant share in United States pacemaker market during the forecast period. According to CDC, around 2.7–6.1 million people in the United States have atrial fibrillation, which is the most common type of heart arrhythmia. The number is expected to increase over the coming years.

Based on technology, United States pacemaker market can be segmented into single chamber, dual chamber, and CRT-P/biventricular pacemakers. Among them, dual-chamber pacemakers held significant share in United States market in 2019, owing to reduced post-surgical complications and presence of two leads which assure normal physiology of the heart. However, CRT-P, also known as biventricular pacemakers, are expected to register fastest growth in United States pacemaker market during the forecast period as they are increasingly being used to treat people with arrhythmias caused by advanced heart failure. These pacemakers contain three leads which are connected to the right atrium and both ventricles.

Major players operating in the United States pacemaker market include Biotronik, Inc., Boston Scientific Corporation, Abbot Inc., Zoll Medical Corporation, Oscor Inc., Medtronic, Inc., Cordis, Inc., Livanova PLC, Medico Inc., Oscor Inc. and Osypka Medical Inc. Leading companies are undertaking growth strategies such as new product launches, mergers & acquisitions, and partnerships.

Download Sample Report @ https://www.techsciresearch.com/sample-report.aspx?cid=5173

Customers can also request for 10% free customization on this report.

“The introduction of U.S. Food and Drug Administration (FDA)-cleared MRI-conditional pacemaker models are a growing trend in United States. Around 20% of pacemaker patients need an MRI within the first two years of implant. Pacemakers which do not have MRI-conditional use technology, generally prevent patients from being able to get an MRI. With increasing cases of cardiovascular diseases in the country, more manufacturers are coming up with advanced products and are using MRI-conditional use technology,” said Mr. Karan Chechi, Research Director with TechSci Research, a research based global management consulting firm.

“United States Pacemaker Market By Type (MRI Compatible Pacemakers, Conventional Pacemakers), By Product Type (Implantable Pacemaker, External Pacemaker), By Application (Heart Block, Arrhythmia, Atrial Fibrillation, Bradycardia, Tachycardia,  Long QT Syndrome), By Technology (Single Chamber, Dual Chamber, CRT-P/Biventricular Pacemaker), By End User (Hospitals & Cardiac Centers, Ambulatory Surgical Centers), By Region, Forecast & Opportunities, 2025” has evaluated the future growth potential of United States pacemaker market and provides statistics & information on market size, structure and future market growth. The report intends to provide cutting-edge market intelligence and help decision makers take sound investment decisions. Besides, the report also identifies and analyzes the emerging trends along with essential drivers, challenges, and opportunities in United States pacemaker market.

Contact

Mr. Ken Mathews

708 Third Avenue,

Manhattan, NY,

New York – 10017

Tel: +1-646-360-1656

Email: sales@techsciresearch.com

Website: https://www.techsciresearch.com/

For More Market Research Blogs Visit: https://techsciblog.com/

[biomedgrid]

Biomed Grid | The Role of Hyperaldosteronism in the Pathogenesis of Neuroleptic Cardiomyopathy

Introduction

Neuroleptic cardiomyopathy (NCMP) is a little-studied iatrogenic pathology of the heart due to the side cardiotoxic effects of antipsychotic (neuroleptic) drugs [1-4]. Along with the fact that the etiology of the disease is quite clearly defined, its pathogenesis remains not fully studied. Analysis of the literature data on the development of different types of cardiomyopathies allows us to make some assumptions about the pathogenesis of NCMP. One of the possibilities of heart damage with long-term use of antipsychotics is their indirect effect through the influence of these drugs on the overall metabolism in the body. So, it is well known that one of the most serious side effects of neuroleptic therapy is metabolic syndrome [5-9]. One of the links of neuroendocrine dysfunction in metabolic syndrome is hypercorticism, that is, an increase in the formation and excretion of all steroid hormones of the adrenal cortex [10], including aldosterone, which plays a certain and, presumably, a significant role in the pathogenesis of NCMP [11]. This brief review of the literature is devoted to this issue.

First, it is necessary to highlight several pathophysiological aspects related to this hormone. Thus, aldosterone is a mineralocorticoid produced mainly in the glomerular zone of the adrenal cortex and in smaller quantities - in the brain, myocardium, vascular endothelium [12-14]. The main activator of the synthesis of aldosterone is the renin - angiotensin system. Inhibit the formation and secretion of the hormone primarily atrial and brain natriuretic peptides [13]. The main effects of aldosterone are an increase in sodium reabsorption in the distal tubules of the kidneys and the associated fluid retention; the consequence of reducing natriuresis is an increase in potassium excretion in the urine [11-16]. Aldosterone also increases the severity of local intravascular inflammation, causes damage to the endothelium of peripheral vessels, increases the number of receptors for angiotensin II in vessels, accelerates apoptosis of cardiomyocytes and potentiates the effects of reninangiotensin system [11,13,17,18]. In this regard, it is important to note that, in addition to the distal renal tubules, aldosterone receptors are present in endothelial cells, cardiomyocytes and fibroblasts of the heart muscle [11,13,19]. It was found that the interaction of aldosterone with these receptors directly affects the state of the extracellular matrix of the myocardium, causes an acceleration of fibroblast proliferation, as well as an increase in the synthesis and accumulation of collagen types I and III, resulting in myocardial fibrosis [19,20-27], which plays a leading role in the appearance of diastolic dysfunction induced by the action of this hormone [21]. It should be emphasized that the development of fibrosis of the heart muscle under the action of aldosterone is not mediated by sodium and fluid retention but is a consequence of direct stimulation of myocardial receptors [28]. This effect is realized primarily by increasing the synthesis of aldosterone not in the adrenal glands, but in the myocardium [29,30], in the aorta and in the coronary sinus [30]. It is also believed that aldosterone which is namely locally synthesized and fixed to the membrane receptors of the heart plays an important role in the processes of ventricular remodeling [31,32]. The triggering effect of aldosterone in relation to fibrosis of the heart muscle is likely determined by the influence of this hormone on the activity of collagenase.

According to CG Brilla et al. [33], aldosterone enhances the expression of type III collagen gene in cardiac fibroblasts by inhibiting the activity of this enzyme. In addition, many authors have revealed the direct influence of hyperaldosteronism on the processes of cardiac remodeling and hypertrophy [34-37] and demonstrated a direct correlation of aldosterone level with myocardial mass [38,39]. One of the mechanisms of aldosterone participation in cardiac remodeling is its induction of generalized inflammation and oxidative stress [24,25,40], as well as increased activity of matrix metalloproteinases [22&40]. All this is accompanied by an increase in the ultrasonic density of the myocardium, an increase in the stiffness of the walls of the left ventricle, violations of its filling and the appearance of diastolic dysfunction due to the development of extensive fibrosis [22,41,42]. These phenomena accelerate the progression of heart failure, increase the electrical heterogeneity of the myocardium, which underlies the mechanisms of re-entry and reduce the threshold for the occurrence of life-threatening ventricular arrhythmias [22]. The elongation of the QT interval is observed on the electrocardiogram [41,42]. It is appropriate to recall that this electrophysiological phenomenon is a very characteristic feature of the cardiotoxic action of neuroleptics and developing NCMP [43-48]. Diastolic dysfunction induced by the action of aldosterone has been found by many researchers [35,37,42,49,50]. Myocardial fibrosis plays a leading role in its development [50,51]. Hyperaldosteronism entails a decrease in the number of contractile elements per unit volume of the myocardium, the development of tissue hypoxia and disruption of the synchronous operation of cardiomyocytes [22]. At the same time, one of the mechanisms of the negative effect of aldosterone on the structure of the heart muscle is its ability to activate cardiomyocyte apoptosis [22,52]. This process is known to be of importance in the pathogenesis of cardiac remodeling [53-58]. Aldosterone can influence on the development of cardiovascular diseases, including NCMP, through the formation of endothelial dysfunction [22,59]. The development of aldosterone-induced endothelial dysfunction is associated with decreased bioavailability of NO and activity of NO-synthase [60]. At the cellular level, aldosterone alters signals from the transcriptional nuclear factor NF-kB, induces oxidative stress and enhances the penetration of reactive oxygen species into the vascular wall, which is a serious importance in the genesis of endothelial dysfunction [25,61].

Thus, in addition to the direct cardiotoxic side effect of neuroleptic drugs, their indirect effect on the overall metabolism of the body, the development of neuroendocrine dysfunction and metabolic syndrome, an important component of which is hyperaldosteronism, is essential in the pathogenesis of NCMP. The diverse damaging effect of aldosterone on the heart plays an important role in the development of NCMP [11].

Read More About this Article: https://biomedgrid.com/fulltext/volume5/the-role-of-hyperaldosteronism-in-the-pathogenesis-of-neuroleptic-cardiomyopathy.000916.php

For more about: Journals on Biomedical Science :Biomed Grid | Current Issue

[market-research-future]

Long QT Syndrome Market Overview, Economic Impact, Dynamics and SWOT Analysis To 2027

Market Highlights

The market of Long QT syndrome (LQTS) is driven by factors like the prevalence of cardiovascular diseases & diabetes, and growing geriatric population. Moreover, rising per capita healthcare expenditure and increasing R&D expenses by the key players is fuelling the market growth over the forecast period. However, lack of awareness and stringent FDA approvals followed by the high cost of the surgical therapeutics pertaining to this disorder are restraining the market growth.

The global long QT syndrome market is expected to grow at a CAGR of 8.50% during the forecast period.

Segmentation

The global long QT syndrome market is segmented on the basis of type, diagnosis, treatment, and end users. On the basis of the type, the market is segmented into long QT syndrome type 1, long QT syndrome type 2, long QT syndrome type 3, and others.  On the basis of the diagnosis, the market is categorized into tests, electrocardiogram (ECG), genetic testing, and others.

On the basis of the treatment, the market is segmented into medication, surgical procedures, and others.  On the basis of the end user, the market is segmented into hospitals & clinics, diagnostic labs, research organizations, and others.

Regional Analysis

The Americas dominate the global long QT syndrome market. A well-developed healthcare sector, high per capita healthcare expenditures, increasing number of patients suffering from cardiovascular diseases and diabetes are the major drivers for long QT syndrome market in the Americas. Moreover, the presence of developed economies like the U.S. and Canada boosts the market growth within this region.

Europe holds the second largest market share. Availability of funds for research, huge patient population, and government support for research & development will drive the market. Regionally, Europe is divided into Western Europe and Eastern Europe. The Western Europe leads the market within the Europe. However, Eastern Europe is estimated to be the fastest growing region.

Browse Full Report @ https://www.marketresearchfuture.com/reports/long-qt-syndrome-market-5303 .

Asia Pacific is the fastest growing market. Increasing prevalence of diabetes and growing geriatric population are the major driving factors for the market growth within the region. Moreover, the presence of developing healthcare sector and developing economies within the region are boosting the market growth.

The Middle East & Africa holds the least share of the market. Presence of poor economies and stringent government policies especially in the Africa region are the restraining factors for market growth within the region. The Middle East holds a major share of the market within the region. This can be attributed due to huge healthcare expenditures by the developed economies of the region.

Key Players

The key players for the global long QT syndrome market are Invitae Corporation (U.S.), GeneDx. (U.S.), Asper Biogene (Estonia), Boston Scientific Corporation (U.S.), Laboratory Corporation of America Holdings (U.S.), Pfizer Inc. (U.S.), Zydus Pharmaceuticals, Inc. (U.S.), Aralez Pharmaceuticals Inc. (Canada), AstraZeneca (U.K), Torrent Pharmaceuticals Limited (India), Lupin Pharmaceuticals, Inc. (U.S.), Cipla Inc. (India), Mylan N.V. (U.S.), Teva Pharmaceutical Industries Ltd. (Israel), and others.

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[hobisexually]

You definitely don't have to answer if you don't want to but what heartproblem do you have? : /

not a problem love! been talking about it for twenty years so might as well.

it’s an arrhythmia disorder called lqts (long QT syndrome) and it basically means that there’s a faulty electric wiring in my genes that makes my heartbeat irregular. your heartbeat has a QT interval, and mine is prolonged. you have different types of it (one type only occurs when you’re in physical extortion/straining yourself, the second with extreme emotions, third in rest).

we discovered I had this when I was seven through a story I won’t bore you with and I’ve been taking beta blockers ever since (which people with anxiety also get prescribed sometimes, double cheers fnfnf). I have type 1, which means that with working out I have to be careful, with sharp differences between hot and cold, and especially with working out in heat. (This sounds like I’m an omega, Jesus) swimming can be dangerous in case it goes wrong so I’m never allowed to go alone.

It’s fine, I just have to be careful with holidays and sports in the summer and it means I’ll never be a professional athlete but I never really had that dream anyway 😂