#Nonclassical @nonclassical

[dhddmods]

Some intersex flags! (Part 2)

Part 2 of making flags for intersex traits and variations that were flagless! (Part one is here.) There will be a part 3 & 4 and then all the main intersex traits/variations will officially have flags.

Please read (and reblog) our intersex guide. It is so important to understand intersex people, their bodies, and their health!

Accessory Ovary

The flag is based on the uterus didelphys and polyorchidism flags. The A is meant to represent the name of this variation. The three circles are meant to represent ovaries. The plus symbol is meant to stand for people who have more than one accessory ovary (aka 3+ ovaries.)

Supernummary Ovary

The flag is based on the uterus didelphys and polyorchidism flags. The S is meant to represent the name of this variation. The three circles are meant to represent ovaries. The plus symbol is meant to stand for people who have more than one accessory ovary (aka 3+ ovaries.)

Cervical Duplication

The flag is based on the uterus didelphys flag. In the center of the flag, the shape is meant to vaguely resemble a uterus with two cervixes.

Cervical Duplication + Uterus Didelphys

The flag is based on the uterus didelphys flag, with the shapes being changed to vaguely resemble two uteruses with cervixes.

Cervical Agenesis

The flag is based on the MKRH Syndrome flag. The symbol is meant to represent a uterus with an X over where the cervix would be.

Cervical Hypoplasia

The flag is based on MKRH Syndrome flag. The symbol is meant to represent a uterus with a minus symbol over where the cervix would be, representing its smaller size.

Uterine Hypoplasia

The flag is based on MKRH Syndrome flag, with a minus with a minus symbol, representing its smaller size.

Anorchia

The flag is based on the ovarian agenesis flag. The circle with the X represents a lack of gonads.

Monorchidism

The flag is based on the ovarian agenesis flag. The circle in the symbol represents the testicle itself. The 1 represents how there is only one gonad, and the M represents the name of this variation.

Unilateral Ovarian Agenesis

The flag is based on the ovarian agenesis flag. The circle in the symbol represents the ovary itself. The 1 represents how there is only one gonad, and the U represents the name of this variation.

Aromatase Excess Syndrome (AEXS/AES)

The flag uses the hyperestrogenism symbol, as it is a form of hyperstrogenism. The shades of pink represent the feminizing effects of the variation. The rainbow and monochrome coloring represents the gender spectrum, and how any gender can have this variation.

Classic CAH (Simple-Virilizing)

The flag is based on the CAH flag. The SV represents the name of this variation.

Classic CAH (Salt-Wasting)

The flag is based on the CAH flag. The SW represents the name of this variation.

Nonclassic CAH

The flag is based on the CAH flag. The NC represents the name of this variation.

Leydig Cell Hypoplasia (LCH)

The shades of blue represent the masculinizing effects of testosterone. The grey represents hypoandrogenism, and low levels of androgens. The grey circle represents the testes, and the underdeveloped/absent leydig cells within them.

17-KSR Deficiency

The purple and yellow represents that this variation is a form of intersex. The shades of grey surrounding the blue represents the "barrier" preventing androstenedione from converting into testosterone. The Ks and 17 represents the name of this variation.

Aromatase Deficiency

The purple and yellow represents that this variation is a form of intersex. The shades of blue represents the hyperandrogenism, and the pink surrounded by grey represents hypoestrogenism. The AD represents the name of this variation.

Mild Androgen Insensitivity Syndrome (MAIS)

The flag is based on the AIS flag. The M represents the name of this variation.

Partial Androgen Insensitivity Syndrome (PAIS)

The flag is based on the AIS flag. The Ps represents the name of this variation.

Complete Androgen Insensitivity Syndrome (CAIS)

The flag is based on the AIS flag. The Cs represents the name of this variation.

[iamlisteningto]

Nonclassical’s nonclassical at 20

[hatsumishinogu]

Nonclassic School [Last Volume]

[bpd-shuichi-togo]

they're intersex now! bc it's likely a considerable portion of the cloned population was intersex, and i make all the rules 🤍

(they are also incredibly transgender, but that's unrelated.)

Klinefelter Syndrome is a genetic condition in those who would otherwise be assigned male, characterized by an additional X-chromosome (sometimes more than one). Read about signs and symptoms at Mayo Clinic!

Congenital adrenal hyperplasia is a group of genetic disorders that affect the adrenal glands, which produce various hormones, including androgens like testosterone. "Nonclassic" CAH is a milder form that may only become apparent in the later stages of childhood or early adulthood, and may even be asymptomatic, whereas "classic" CAH is more severe, and is usually detected at birth or in early infancy. Read about signs and symptoms at Mayo Clinic!

[intersexfairy]

I’m tired of people saying pcos is intersex, just shut the fuck up dude, no one takes it seriously and no one will start if you are spreading fucking lies about it. Pcos can be caused by 4 things, adrenal glands, insulin resistance, hormone imbalance, or hereditary. You see a woman with a beard and you think they must be intersex. You’re a fucking disgrace and should be ashamed

I was gonna not post this to not spread negativity, until I realized you literally made the case for it being intersex in your own ask but are too intersexist to realize it. So now I just think it's really funny - if anyone shouldn't be taken seriously, it's you.

"Adrenal" "Hereditary" "Woman with a beard" ...Hey what medically recognized intersex variation (that's virutally indistinguable to PCOS in its nonclassic form by symptoms alone) does that sound like? Congenital adrenal hyperplasia, anyone?

Also InterAct Advocates, an intersex activist organization, recognizes PCOS as intersex so like. To any intersex people with PCOS reading this, you are intersex and there are so fucking many of us who know it. We see you and we accept you with open arms. And we'll be fighting alongside you to get to recognition and rights we all deserve!

[258punkweight]

i have hyperandrogenism but i think it's more likely cah than pcos.

pcos is more common i think. the symptoms are as follows: irregular/absent/heavy periods, excess body hair, acne, pelvic pain, infertility, patches of darker skin, ovarian cysts, hyperandrogenism, weight gain, metabolic syndrome, and alopecia.

i only experience the menstrual irregularity, body hair, acne, and hyperandrogenism. i'm not sure abt the infertility bc i don't want kids and never have, and i was supposed to get scanned for the cysts but it never happened. also 80% of ppl with pcos are obese and i have never been.

the symptoms of cah (more specifically, the nonclassic/late onset variant; the others would've been detected at birth): acne, hirstutism, rapid childhood growth, early or delayed puberty, menstrual irregularity, infertility, and a couple things that make me uncomfortable to mention here

i experience one of 3 unmentioned symptoms. like i said, idk abt infertility bc i don't want kids. but i experience literally everything else i listed.

it's 4/11 pcos symptoms, vs 7/9 cah symptoms

[intersex-support]

Hey, I have some questions rated to NCAH. I was AFAB and as far as I know don't have genital variations, but I had an early puberty and symptoms of hyperandrogenism such as acne and excess body and facial hair (noticeable but not severe). I had issues with my periods but I was diagnosed with endometriosis so I don't think they're connected. I did hormone testing and despite everything, my testosterone is average. However, I have high prolactin and 17-alpha-hidroxi-progesterone, which I know is associated with NCAH. Is it enough for a diagnosis though? I've done research on it but I'm not sure. Sadly I don't think my doctor will help in this regard because she already glossed over this part of the test results completely when I received them and wasn't open to the idea of me maybe having hyperandrogenism anyways

Hi anon! I'm sorry that your doctor hasn't been helpful in interpreting your test results. that's really frustrating that you aren't able to easily access medical support. Standard disclaimer that we can't diagnose people or provide medical advice on here, but I'm happy to share some resources for understanding your test results and explaining the CAH diagnosis criteria.

So, you're absolutely right that 17 alpha hidroxi progesterone (17 OHP) is associated with CAH. High levels of 17 OHP is one of biggest signs of CAH. I think it was really, really shitty and irresponsible of your doctor to refuse to test more because 17 OHP plus signs of hyperandrogenism are like textbook signs of CAH.

I'm going to share a chart explaining 17 OHP levels and CAH, sourced from myendoconsult. (Here's another medical journal with more information about diagnosis as well.)

ID: [A flowchart titled "Cosyntropin (ACTH) stimulation test. The first row of the flowchart shows the possible options for 17 OHP levels. Text of option 1 reads "Heterozygotes/Unaffected. 17 OHP less than 200 ng/dL or less than 6 nmol/L." Option 2 reads "Indeterminate. 17 OHP is 200 to 10,000 ng/dL or 6 to 300 nmol/L." The third option reads "Classic 21OH deficiency (CAH). 17 OHP is greater than 10,000 ng/dL or greater than 300 nmol/L." The next section of the flowchart has different options for what to do next if the results are indeterminate, and shares the three options for ACTH stim test results. The first ACTH stim test result option is less than 1000 ng/dL or less than 30 nmol/L, which would be heterozygotes/unaffected. The next category of ACTH stim test result is 1000 to 10,000 ng/dL or 31 to 300 nmol/L, which is Nonclassic CAH (NCAH). The third category of ACTH stim test result is greater than 10,000 ng/dL or greater than 300 nmol/L, which is Classic CAH. ]

This flowchart shows the role that 17 OHP plays in CAH diagnosis. First, doctors just test your 17 OHP levels and see if they can make a diagnosis just from that. If your 17 OHP levels were less than 200 ng/dL, they can see that you don't have CAH. If your 17 OHP levels were over 10,000, they can diagnose you with Classic CAH. However, if your 17 OHP levels are higher than normal but still below 10,000 CAH, that usually means they need to do more testing to figure it out.

The usual way they test this is through an ACTH stimulation test, where they inject you with ACTH and then measure your 17 OHP levels an hour afterwards. For people with NCAH or CAH, their 17 OHP levels will jump even higher, with a change of at least 1000 ng/dL or 30 nmol/L required for diagnosis.

So, long story short, one way you could try to figure out if your 17 OHP levels mean that you have CAH is by comparing your lab results to the flowchart above. If your 17 OHP levels are in the indeterminate range, this is something that you could bring back in to your doctor and say "Hey, best practices mean that since my 17 OHP levels are high enough to be in this range that you need to do an ACTH stimulation test for me so I can get actual results."

High prolactin can also be associated with CAH. Here's one study talking about high prolactin levels. In fact, here's another study specifically talking about high prolactin levels and normal androgen levels in a subset of people with CAH.

So, long story short, high levels of 17 OHP and high prolactin can be signs of CAH. To confirm a diagnosis, you'd probably need to look specifically at your records to figure out your 17 OHP levels, and potentially try to get an ACTH stim test depending on your results, although I know that can be difficult or impossible when doctors refuse to cooperate.

I hope this helps, and please feel free to send in another ask if you have any other questions!

[77ngiez-archive]

pride headcanons for katya pafl?:]

KATYUSHA :((( I THINK OF HER...

shes an aroacespec lesbian! she isnt interested jn traditional dating but she loves to be in qprs. she's intersex [unrelated to her chimerism for the record], specifically she has nonclassic cah. it hasnt affected her too much yet at the time of canon and nobody knows abt it yet bcs of that. annnd shes fem-aligned but doesnt rlly care how she gets gendered. she asks her friends to switch it up a little for her every now and then. for fun <3

[the-intersex-experience]

Can someone be considered intersex if they have non-salt-wasting nonclassical congenital adrenal hyperplasia (NCAH)?If they had non consensual surgery as an infant, but were raised as one gender and made to believe they are perisex?

I’m struggling with some recent life discoveries and trying to understand myself a bit better… any insight is very much appreciated!

I'm not super educated on the first words. But Google said it can cause intersex variations + if you had surgery against your will in order to better "fit" their idea of a sex, that's an intersex experience. Others who know more / have advice to help you figure things out feel free to reply

[hatsumishinogu]

Nonclassic School [First Volume]

[trans-axolotl]

do you know anything about late-onset congenital adrenal hyperplasia? i just found it on my file while looking for something else. i have an unrelated surgery coming up and i'm trying to figure out if this could have any effect on how my body handles that, especially wrt cortisol. but i can't find much on it at all, particularly how it affects ppl beyond infertility and sex characteristics. my impression is that at least the classic variant of it sounds like something intersex-related so i thought you might have heard of it. i remember asking my doctors for more information and they were basically like "don't worry about it unless you wanna get pregnant :)"

Hey anon.

So, late onset congenital adrenal hyperplasia, also called nonclassic congenital adrenal hyperplasia, is considered an intersex variation. (If you search NCAH on my blog or on @intersex-support, you might find some other posts about it). Because of the way it affects our testosterone levels and secondary sex characteristics outside of what is typically considered "female", late onset CAH is intersex, even if late onset CAH hasn't caused changes in genitalia.

Of course, there's no pressure to instantly start referring to yourself as intersex, or start participating in intersex community, but know that you are welcome here, and there are a lot of people who understand what it's like. I also have CAH, and I'm always happy to talk about it with anyone else. I know that it can be sort of shocking or overwhelming to get that information for the first time. Any feelings you have about it are completely valid. You can take as much time as you need to process. The community is here for you whenever you're ready.

In terms of surgery and CAH, there are some important considerations. For people with NCAH, there's going to be a range for how each individual makes and processes cortisol and aldosterone. Some people need daily glucocorticoid replacement therapy while others do not. However, even if you do not need daily steroid replacement, some people with NCAH need extra "stress dosing" of glucocorticoids when their body is under physical stress, such as during illness, injury, or surgery. It would be really important to talk to your surgeon, an anesthesiologist, and probably an endocrinologist before the surgery to make sure you know what your cortisol and aldosterone production is and make a plan if you need stress dosing during surgery or surgery recovery.

One book I would recommend for people with CAH is the Parent's Guide to CAH. In a lot of ways this book kind of sucks--it's directed towards parents, uses a lot of cissexist language, and uses a very medicalized model, but has some of the clearest nonacademic writing about what CAH is, how it works, treatment options, and talks about different considerations like surgery, illness, etc.

Please feel free to reach back out with any other questions, whether it's about CAH, intersex stuff, or if you want suggestions on any other resources. Thinking of you, anon, and hope your surgery goes well! 💜💛💜

[pensandsliverswords]

∞💙

whelp first nonclassical/non-song-on-a-playlist-i-havent-listened-to-yet, ‘Jingle Jangle Jingle’

“ I got spurs that Jingle Jangle Jingle as I go riding merrily along and they say oh ain’t you glad you’re single and that song it’s so very far wrong”

I do love me some cowboy songs even if there absurd at times

[dushume]

Disruptive Frequencies Live @ Kings Place - 15th July 23.

Beautiful moments captured by Stephen Akinyemi @Bare_Clips on Insta.

Disruptive Frequencies Vinyl/Digital Out Now - Purchase Here

[mcatmemoranda]

BMI: body mass index; E2: estradiol; FSH: follicle-stimulating hormone; hCG: human chorionic gonadotropin; MRI: magnetic resonance imaging; PCOS: polycystic ovary syndrome; PRL: prolactin; T: testosterone; TSH: thyroid-stimulating hormone.

* Many clinicians also measure serum 17-hydroxyprogesterone at the initial visit to rule out nonclassic 21-hydroxylase deficiency. Some also measure serum dehydroepiandrosterone sulfate (DHEAS).

¶ Mild hyperprolactinemia can sometimes be seen with hypothyroidism. Euthyroidism should be confirmed before performing MRI.Δ Pituitary MRI not required in those with clear explanation for their hypogonadotropic amenorrhea, eg, eating disorder, excessive exercise, celiac disease, or type 1 diabetes mellitus.

[doberbutts]

I just saw your post about how testosterone has helped your disability tremendously, which if I remember correctly is hypermobile Ehlers-Danlos syndrome (but maybe I’m wrong). I’m agender and have been considering early menopause to help with my abundance of menstrual health issues, but if t helped your hEDS too that makes me really want to consider it further. If this is too private you absolutely don’t have to answer it but I’d love to know more as a fellow disabled person.

It's POTS actually :) though POTS is highly comorbid with hEDS and I *am* hypermobile, to a degree that I made my physical therapist wince with how easily I can assume positions that should noooooot be possible without pain when I was in recovery due to my car accident. She actually asked me if I have EDS and I said well I have POTS so... not officially but is it possible sure I guess.

So no, I do not have hEDS. But also yes I might have hEDS. Schroedinger's diagnosis.

No, testosterone helped my POTS symptoms disappear to almost nothing. This is at least partially because POTS does not get along with estrogen ans menstrual cycles, and taking testosterone lowered the estrogen in my body and also stopped my menstural cycle. Don't get me wrong, I still have some symptoms, but they are dramatically improved. I can do all sorts of things I used to not be able to.

There is... a bit of debate whether I have POTS at all. But I do have MCAS and, as said, I'm hypermobile, so very likely yes POTS is very likely. But with my NCAH diagnosis, it's equally possible that I *do not* have POTS, because NCAH also causes weird changes to your vasovagal response and your autonomic nervous system. In other words, do I have POTS AND NCAH... or do I just have NCAH?

Personally I don't really care because adding testosterone fixed the symptoms way better than anything the POTS meds were doing so w/e, if it works it works.

It's "makes you intersex and sometimes gay disease but also you faint a lot and your body *really* sucks at handling stress disease". To be fair the classic form of CAH can just outright kill you if not well controlled. Nonclassic CAH by comparison sometimes still tries really hard but is less likely to do more than give you some serious medical trauma.

So like. Am I fainting because my body doesn't know how to handle stress which triggers a response of my autonomic nerve and because my adrenal glands are too busy making androgens to give me some fucking cortisol to communicate with my autonomic nerve, the thing guesses wrong and I hit the ground as my body attempts the biology version of "have you turned it off and then back on again"? Or am I the unluckiest fucker on the planet and I have two annoyingly underdiagnosed but much more common than we thought disorders which both hate estrogen and make me faint a lot?

Or, bonus, since POTS joins PCOS in the "disorder named for a symptom that's not even diagnostuc criteria and no one actually knows what causes it" family, is what we're calling "POTS" actually a group of symptoms that has a wide range of causes and my cause for my own symptoms IS NCAH? Who knows 🤷‍♂️

[hiiragi7]

Frustrated about how inaccessible and inaccurate so much info about CAH is.

Annoyed with how many people call CAH a condition which only affects females, when it isn't.

Common CAH variations may still show signs in male patients (but are usually deemed "socially acceptable"), and there are CAH variations such as 3bHSD2D where *both sexes may have ambiguous genitals at birth*.

Sick especially of medical resources doing this shit because apparently all they know is what they read off of google about 21-OHD and stopped there. This is just one example of all the shitty misinfo about CAH and other intersex variations online and why it took so long to know basic shit about our body. Even our doctors did not know basic information on CAH and it was pure luck we got moved to an endocrinologist who knew there was more types of CAH than just 21-OHD and tested for them.

Want to know another fun fact? One I did not myself know until very recently despite having diagnosed CAH? Classic CAH is not synonymous with ambiguous genitals at all. In fact, with the type of CAH I have, most female infants are born with mild or no virilization at all and almost all have very low prader scale scores.

And yet ambiguous genitals as a classic CAH symptom are made out to be like it's the biggest sign of classic vs nonclassic CAH even by doctors. Nobody reads the actual fucking literature on this shit.

We've been under the assumption for years that we could not have classic CAH because our genital ambiguity grew over childhood and puberty rather than being born with super obviously ambiguous genitals and that it would've been obvious at birth if it were classic and we would've been diagnosed as an infant. It turns out with the type of CAH we were diagnosed with our experience is actually extremely fucking textbook for classic-type!!

We also assumed that we would've been caught by our country's mandatory CAH screenings as an infant if it were classic - Nope, our CAH variant in the way it presents in us does not cause elevated 17-OHP levels and the infant screenings only test for 21-OHD. It's why as an adult we were missed the first time by CAH screening as well until we ended up with previously mentioned endo who tested for CAH variations that aren't necessarily related to 17-OHP.

I am. So so tired of how nobody seems to know shit about anything intersex-related. I just want to know basic information about my own body like anybody else. I just want my friends to be able to understand themselves without going through this slow, horrible, agonizing process of constantly wondering if you're making it all up in your head or why the doctors are so fucked up or why your body looks and functions the way it does and having no answers.

This information should be so much more easily accessible and spread than it is.