#Mavrikakis

[scienza-magia]

Catalisi chimica a temperatura ambiente per risparmiare energia

Scoperta a livello atomico può cambiare il mondo del petrolio e della chimica. Gli ingegneri chimici dell’Università del Wisconsin-Madison hanno sviluppato un modello di funzionamento delle reazioni catalitiche su scala atomica. Si tratta di un progresso considerato una svolta nella ricerca sulla chimica computazionale. La comprensione potrebbe consentire a ingegneri e chimici di sviluppare catalizzatori più efficienti e di mettere a punto i processi industriali – potenzialmente con enormi risparmi energetici, dato che il 90% dei prodotti che incontriamo nella nostra vita sono prodotti, almeno in parte, attraverso la catalisi. Il team ha pubblicato la notizia del loro progresso sulla rivista Science. I materiali catalitici accelerano le reazioni chimiche senza subire essi stessi cambiamenti. Sono fondamentali per la raffinazione dei prodotti petroliferi e per la produzione di prodotti farmaceutici, plastiche, additivi alimentari, fertilizzanti, carburanti verdi, prodotti chimici industriali e molto altro. Due brevi filmati sono disponibili in un file zip a questo link della pagina Science abstract.

Scienziati e ingegneri hanno trascorso decenni a mettere a punto le reazioni catalitiche; però, poiché attualmente non è possibile osservare direttamente tali reazioni alle temperature e alle pressioni estreme spesso coinvolte nella catalisi su scala industriale, non si sa esattamente cosa avvenga su scala nanometrica e atomica. Si sa cosa entra, cosa esce, ma non esattamente le singole operazioni a livello atomico. Questa nuova ricerca aiuta a svelare questo mistero, con potenziali importanti ramificazioni per l’industria. Infatti, solo tre reazioni catalitiche – il reforming del metano a vapore per produrre idrogeno, la sintesi dell’ammoniaca per produrre fertilizzanti e la sintesi del metanolo – utilizzano quasi il 10% dell’energia mondiale. Manos Mavrikakis, professore di ingegneria chimica e biologica presso l’UW-Madison, che ha guidato la ricerca, ha dichiarato: “Se si riducono di pochi gradi le temperature a cui si svolgono queste reazioni, si ottiene un’enorme diminuzione della domanda di energia che oggi l’umanità deve affrontare. Diminuendo il fabbisogno energetico per far funzionare tutti questi processi, si riduce anche la loro impronta ambientale”. Mavrikakis e i ricercatori post-dottorato Lang Xu e Konstantinos G. Papanikolaou, insieme alla studentessa Lisa Je, hanno sviluppato e utilizzato potenti tecniche di modellazione per simulare le reazioni catalitiche su scala atomica. Per questo studio, hanno esaminato le reazioni che coinvolgono i catalizzatori di metalli di transizione in forma di nanoparticelle, che includono elementi come il platino, il palladio, il rodio, il rame, il nichel e altri importanti per l’industria e l’energia verde. Secondo l’attuale modello di superficie rigida della catalisi, gli atomi strettamente impacchettati dei catalizzatori di metalli di transizione forniscono una superficie 2D a cui i reagenti chimici aderiscono e partecipano alle reazioni. Quando si applica una pressione e un calore o un’elettricità sufficienti, i legami tra gli atomi dei reagenti chimici si rompono, permettendo ai frammenti di ricombinarsi in nuovi prodotti chimici. Mavrikakis ha spiegato: “L’ipotesi prevalente è che questi atomi metallici siano fortemente legati tra loro e forniscano semplicemente dei ‘punti di atterraggio�� per i reagenti. L’ipotesi di tutti è che i legami metallo-metallo rimangano intatti durante le reazioni che catalizzano. Per la prima volta, quindi, ci siamo posti la domanda: “L’energia necessaria per rompere i legami nei reagenti potrebbe essere di entità simile all’energia necessaria per rompere i legami all’interno del catalizzatore?””. Secondo la modellazione di Mavrikakis, la risposta è sì. L’energia necessaria per lo svolgimento di molti processi catalitici è sufficiente per rompere i legami e consentire a singoli atomi di metallo (noti come adatomi) di liberarsi e iniziare a viaggiare sulla superficie del catalizzatore. Questi adatomi si combinano in cluster, che servono come siti sul catalizzatore dove le reazioni chimiche possono avvenire molto più facilmente rispetto alla superficie rigida originale del catalizzatore. Utilizzando una serie di calcoli speciali, il team ha esaminato le interazioni di importanza industriale tra otto catalizzatori di metalli di transizione e 18 reagenti, identificando i livelli di energia e le temperature che possono formare questi piccoli cluster metallici, nonché il numero di atomi in ogni cluster, che può anche influenzare drasticamente i tassi di reazione. I loro collaboratori sperimentali dell’Università della California, Berkeley, hanno utilizzato la microscopia a scansione tunneling a risoluzione atomica per osservare l’assorbimento del monossido di carbonio sul nichel (111), una forma cristallina stabile di nichel utile nella catalisi. I loro esperimenti hanno confermato i modelli che mostravano che vari difetti nella struttura del catalizzatore possono anche influenzare il modo in cui i singoli atomi di metallo si liberano e la formazione dei siti di reazione. Mavrikakis afferma che il nuovo quadro sta mettendo in discussione le fondamenta del modo in cui i ricercatori comprendono la catalisi e il modo in cui si svolge. Potrebbe applicarsi anche ad altri catalizzatori non metallici, cosa che studierà in futuro. È anche rilevante per la comprensione di altri fenomeni importanti, come la corrosione e la tribologia, o l’interazione delle superfici in movimento. “Stiamo rivedendo alcuni presupposti ben consolidati per capire come funzionano i catalizzatori e, più in generale, come le molecole interagiscono con i solidi”, ha detto Mavrikakis. Il successo di questa ricerca può rivoluzionare tutti i processi chimici, perché permette di calcolare con precisione l’energia da utilizzare e di ottimizzarlo. Chi riuscirà a sfruttare meglio questa conoscenza avrà un vantaggio competitivo notevole, mentre si prospetta un calo significativo del costo di un insieme di prodotti chimici di sintesi.  Read the full article

[crepuscule-pourpre]

[noahl-art]

14, 27, 34 :3

HEYA ! ✨️

14 - What’s your favorite color?: mmmh if I say black does it sound very emo or... but also dark royal teal (paired with gold ✨️)

27 - What’s your favorite book? Or just one you’ve read a few times?: That's a hard one! I would say either The Tenant of Wildfell Hall by Anne Brontë or Deuils Cannibales et Mélancoliques by Catherine Mavrikakis

34 - What’s your favorite flower? Buttercups, daisies and dandelions 🌼

[colonna-durruti]

Ma ci sono dei momenti nella vita che non succede niente, che hai l'impressione che il destino o quel bastardo di un Dio ci abbia dimenticato... Non mi sento più eletto, mi sembra di essere stato dimenticato.

Gli ultimi giorni di Smokey Nelson, Mavrikakis

[longhaulerbear]

Severe COVID-19 is linked to changes in the brain that mirror those seen in old age, according to an analysis of dozens of post-mortem brain samples.

The analysis revealed brain changes in gene activity that were more extensive in people who had severe SARS-CoV-2 infections than in uninfected people who had been in an intensive care unit (ICU) or had been put on ventilators to assist their breathing — treatments used in many people with serious COVID-19.

The study, published on 5 December in Nature Aging, joins a bevy of publications cataloguing the effects of COVID-19 on the brain. “It opens a plethora of questions that are important, not only for understanding the disease, but to prepare society for what the consequences of the pandemic might be,” says neuropathologist Marianna Bugiani at Amsterdam University Medical Centers. “And these consequences might not be clear for years.”

COVID on the brain

Maria Mavrikaki, a neurobiologist at the Beth Israel Deaconess Medical Center in Boston, Massachusetts, embarked on the study about two years ago, after seeing a preprint, later published as a paper2, that described cognitive decline after COVID-19. She decided to follow up to see whether she could find changes in the brain that might trigger the effects.

What triggers severe COVID? Infected immune cells hold clues

She and her colleagues studied samples taken from the frontal cortex — a region of the brain closely tied to cognition — of 21 people who had severe COVID-19 when they died and one person with an asymptomatic SARS-CoV-2 infection at death. The team compared these with samples from 22 people with no known history of SARS-CoV-2 infection. Another control group comprised nine people who had no known history of infection but had spent time on a ventilator or in an ICU — interventions that can cause serious side effects.

The team found that genes associated with inflammation and stress were more active in the brains of people who had had severe COVID-19 than in the brains of people in the control group. Conversely, genes linked to cognition and the formation of connections between brain cells were less active.

The scientists also analysed brain tissue from 20 further uninfected people: 10 who were 38 years old or younger at death, and 10 who were 71 or older. A comparison revealed that people in the older group had brain changes that were similar to those seen in people with severe COVID-19.

The work is preliminary and will need to be confirmed using complementary approaches, says Daniel Martins-de-Souza, head of proteomics at the University of Campinas in Brazil. But it is an informative study, he says, and such research could ultimately guide treatment for people who have lingering cognitive difficulties after COVID-19.

Inflammatory effect

Mavrikaki suspects that COVID-19’s effects on gene activity are caused indirectly, by inflammation, rather than by viral infiltration of the brain. Supporting this interpretation, she and her colleagues found that exposing laboratory-cultured neurons to proteins that promote inflammation affected the activity of a subset of the aging-related genes.

But it’s possible that this response might also be triggered by other infections, she says. And the study could not fully control for obesity or other conditions that might both increase a person’s chances of developing severe COVID-19 and generate an inflammatory state that affects gene expression in the brain.

Another key question is whether the changes in gene expression are associated only with severe cases of COVID-19, or if milder disease can also cause them, says Bugiani. In March, a study3 of hundreds of brain images collected by the UK Biobank found that even mild disease could cause changes in the brain, including damage to the regions involved in smell and taste.

It will take time to determine whether the changes observed in the study are transient or are there to stay, Bugiani says. “The duration of the pandemic has now been long enough to see these things, but not long enough to establish if they are permanent,” she says. “We don’t yet know what their real consequences will be.”

[curiositesvoltairiennes]

«Le dieu de Pearl n’était pas le même que celui de ces gens et il ne demandait pas grand-chose aux humains. Il suffisait de cultiver son propre jardin pour l’apaiser» (Catherine Mavrikakis, les Derniers Jours de Smokey Nelson, Montréal, Héliotrope, 2011, 303 p., p. 141).

Les derniers mots de Candide sont «Cela est bien dit, répondit Candide, mais il faut cultiver notre jardin.»

[already-14]

Valentine Penrose

1898 — MONT-DE-MARSAN, FRANCE | 1978 — CHIDDINGLY, ROYAUME-UNI

Portrait de l’artiste en jeune femme surréaliste Valentine Penrose, Écrits d’une jeune femme surréaliste, Édition (introduction, chronologie et bibliographie) établie par Georgiana M. M. Colvilie, préface d’Antony Penrose, Éd. Joëlle Losfeld, 290 p. Catherine Mavrikakis

awarewomenartists.com

[themedicalstate]

Brain plasticity in drug addiction: Burden and benefit

Contributor: Maria Mavrikaki, PhD

The human brain is the most complex organ in our body, and is characterized by a unique ability called neuroplasticity. Neuroplasticity refers to our brain’s ability to change and adapt in its structural and functional levels in response to experience. Neuroplasticity makes it possible for us to learn new languages, solve complex mathematical problems, acquire technical skills, and perform challenging athletic skills, which are all positive and advantageous for us. However, neuroplasticity is not beneficial if we develop non-advantageous learned behaviors. One example of non-advantageous learning is habitual drug misuse that can lead to addiction.

Our brain learns to respond to drugs of abuse

Our first decision to use a drug may be triggered by curiosity, circumstances, personality, and stressful life events. This first drug exposure increases the release of a molecule (neurotransmitter) called dopamine, which conveys the feeling of reward. The increased changes in dopamine levels in the brain reward system can lead to further neuroplasticity following repeated exposure to drugs of abuse; these neuroplasticity changes are also fundamental characteristics of learning. Experience-dependent learning, including repeated drug use, might increase or decrease the transmission of signals between neurons. Neuroplasticity in the brain’s reward system following repeated drug use leads to more habitual and (in vulnerable people) more compulsive drug use, where people ignore the negative consequences. Thus, repeated exposure to drugs of abuse creates experience-dependent learning and related brain changes, which can lead to maladaptive patterns of drug use.

Views on addiction: Learning and disease

A recent learning model proposed by Dr. Marc Lewis in New England Journal of Medicine highlights the evidence of brain changes in drug addiction, and explains those changes as normal, habitual learning without referring to pathology or disease. This learning model accepts that drug addiction is disadvantageous, but believes it is a natural and context-sensitive response to challenging environmental circumstances. Dr. Nora Volkow, director of the National Institute on Drug Abuse (NIDA), and many addiction researchers and clinicians, view addiction as a brain disease triggered by many genetic, environmental, and social factors. NIDA uses the term “addiction” to describe the most severe and chronic form of substance use disorder that is characterized by changes in the brain’s reward, stress, and self-control systems. Importantly, both learning and brain disease models accept that addiction is treatable, as our brain is plastic.

We can adapt to new learned behaviors

Our brain’s plastic nature suggests that we can change our behaviors throughout our lives by learning new skills and habits. Learning models support that overcoming addiction can be facilitated by adopting new cognitive modifications. Learning models suggest pursing counseling or psychotherapy, including approaches such as cognitive behavioral therapy (CBT), which can help a person modify their habits. NIDA suggests that, for some people, medications (also called medication-assisted treatment or MAT) can help people manage symptoms to a level that helps them pursue recovery via strategies such as counseling and behavioral therapies, including CBT. Many people use a combination approach of medications, behavioral therapies, and support groups to maintain recovery from addition.

Neuroplasticity can help us modify behaviors relevant to addiction

CBT is an example of a learning-based therapeutic intervention; thus, it utilizes neuroplasticity. Scientific evidence suggests that CBT, alone or in combination with other treatment strategies, can be effective intervention for substance use disorders. CBT teaches a person to recognize, avoid, and learn to handle situations when they would be likely to use drugs. Another example of evidence-based behavioral therapy that has been shown to be effective for substance use disorders is contingency management. Contingency management provides a reward (such as vouchers redeemable for goods or movie passes) to individuals undergoing addiction treatment, to reinforce positive behaviors such as abstinence. This approach is based on operant conditioning theory, a form of learning, where a behavior that is positively reinforced tends to be repeated. Overall, multiple evidence-based approaches are used for the treatment of substance use disorders that require learning and utilize neuroplasticity.

The bottom line

Our brain is plastic, and this trait helps us learn new skills and retrain our brain. As the brain can change in a negative way as observed in drug addiction, the brain can also change in a positive way when we adopt skills learned in therapy and form new, healthier habits.

Source: Harvard Health Blog at Harvard Medical School

[weirdletter]

Canadian Science Fiction, Fantasy, and Horror: Bridging the Solitudes (Studies in Global Science Fiction), edited by Amy J. Ransom and Dominick Grace, Palgrave Macmillan, 2019. Cover art: Oxygen/Getty Image, info. palgrave.com.

Canadian Science Fiction, Fantasy, and Horror: Bridging the Solitudes exposes the limitations of the solitudes concept so often applied uncritically to the Canadian experience. This volume examines Canadian and Québécois literature of the fantastic across its genres—such as science fiction, fantasy, horror, indigenous futurism, and others—and considers how its interrogation of colonialism, nationalism, race, and gender works to bridge multiple solitudes. Utilizing a transnational lens, this volume reveals how the fantastic is ready-made for exploring, in non-literal terms, the complex and problematic nature of intercultural engagement.

Contents: Notes on Contributors Introduction: Bridging the Solitudes as a Critical Metaphor – Amy J. Ransom and Dominick Grace Colonial Visions: The British Empire in Early Anglophone and Francophone Canadian Science Fiction and Fantasy – Allan Weiss Nevermind the Gap: Judith Merril Challenges the Status Quo – Ritch Calvin Two Solitudes, Two Cultures: Building and Burning Bridges in Peter Watts’s Novels – Michele Braun The Affinity for Utopia: Erecting Walls and Building Bridges in Robert Charles Wilson’s The Affinities – Graham J. Murphy The Art of Not Dying: Station Eleven by Emily St. John Mandel and Oscar De Profundis by Catherine Mavrikakis – Patrick Bergeron When Are We Ever at Home? Exile and Nostalgia in the Work of Guy Gavriel Kay – Susan Johnston Reconciliation, Resistance, and Biskaabiiyang: Re-imagining Canadian Residential Schools in Indigenous Speculative Fictions – Judith Leggatt Indigenous Futurist Film: Speculation and Resistance in Jeff Barnaby’s Rhymes for Young Ghouls and File Under Miscellaneous – Kristina Baudemann Building Hope Through Community in Élisabeth Vonarburg’s The Maerlande Chronicles – Caroline Mosser Cruising Canadian SF’s Queer Futurity: Hiromi Goto’s The Kappa Child and Larissa Lai’s Salt Fish Girl – Wendy Gay Pearson 12. Crossing the (Trans)Gender Bridge: Exploring Intersex and Trans Bodies in Canadian Speculative Fiction – Evelyn Deshane A Maelstrom of Replication: Peter Watts’s Glitching Textual Source Codes – Ben Eldridge The Missing Link: Bridging the Species Divide in Margaret Atwood’s MaddAddam Trilogy – Dunja M. Mohr “I Can’t Believe This Is Happening!”: Bear Horror, the Species Divide, and the Canadian Fight for Survival in a Time of Climate Change – Michael Fuchs Interacting with Humans, Aliens, and Others in Science Fiction from Québec – Isabelle Fournier Holes Within and Bridges Beyond: The Transfictions of Élisabeth Vonarburg and Michel Tremblay – Sylvie Bérard Tropes Crossing: On Some Québec SF Writers from the Mainstream – Sophie Beaulé Transculture, Transgenre: Stanley Péan’s Fantastic Detective Fiction – Kathleen Kellett Excerpts from A Glossary of Non-essential Forms and Genres in English-Canadian Literature – Jordan Bolay Index

[billscheft]

Okay, here’s the full Paul Shaffer walk-on at the Cutting Room (minus my intro and the huge ovation he got walking up). Lovingly shot by my longtime Letterman colleague Kathy Mavrikakis....

[skillstopallmedia]

niagara | Catherine Mavrikakis speaks to her dead (7/10)

niagara | Catherine Mavrikakis speaks to her dead (7/10)

“It was in Niagara that I contracted the disease of death at three years old and I have never recovered from it”, writes Catherine Mavrikakis in a new novel which sheds light on all of her work, whose death has always been the main character and for whom his only shadow annihilates any possibility of serenity. Posted at 5:30 p.m. In 1964, Catherine Mavrikakis’ father took a photo of his wife…

[madeleinisms]

L'Amérique est notre sépulture. Le ciel, une belle ordure.

Le ciel de Bay City, Catherine Mavrikakis

[alexmotamots]

L'absente de tous bouquets - Catherine MAVRIKAKIS

L'absente de tous bouquets de Catherine MAVRIKAKIS : un deuil difficile

J’avais entendu une interview de l’auteure sur France Culture il y a quelques semaines qui m’avait intéressé. Un article dans Lire m’a convaincu de découvrir ce texte. Je dois dire que j’ai été dubitative en lisant ce journal de deuil. D’abord parce qu’il a parfois résonné en moi, mais surtout parce que je ne vis pas le deuil de la même façon que l’auteure. Je me suis parfois sentie très éloignée…

[mook-mookie]

Tagged by @eungi-hyung​  Thanks again hehe 😊😊 !! 

Name: Emily, one of the most common names ever 

Nickname(s): Emoly...my friends call me like that because of the mole i have on my face...oh well 

Average hours of sleep: 8 nowadays cuz im trying to get more sleep lol (im currently learning about sleep and dreams in my psychology class so it kinda made me want to change my sleep habits)

Coffee or Tea: Depends on my mood, really. I don’t drink any of those on a daily basis...but if i really gotta choose i’ll go with tea?

Why I made a tumblr: I have no idea...pure instincts?!

Reasons for my URL: It’s similar to my first name x’D 

Favorite Color(s) to wear: Black and red 

Lipstick or Chapstick: Lipstick, I want people to look at my lips cuz its the only decent feature of my face x’D 

Favorite Hobbies: Drawing, reading smut  and discovering new music artists! 

Last song I listened to: Instupendo - Boy

Last movie and tv show: Movie: Coco Tv show: Blue Planet II

Top three shows: Stranger Things, One Punch Man and One Piece lol

Relationship status: Single 

Favorite colors: Red. Seriously, you can’t go wrong with red 

Three bands/artists: Seventeen, sasakure.UK and Gallant

Currently reading: A freaking bunch of BL mangas/webcomics *sweats* im not even going to name them all cuz the list is long :’D, 1984 by George Orwell and Oscar De Profundis by Catherine Mavrikakis 

tagging: @ploceidaee, @k-i-s-m-e-t, @sinner-chan, @but-what-if-i-fly @mental-llama :D

[curiositesvoltairiennes]

«Le dieu de Pearl n’était pas le même que celui de ces gens et il ne demandait pas grand-chose aux humains. Il suffisait de cultiver son propre jardin pour l’apaiser.»

Catherine Mavrikakis, les Derniers Jours de Smokey Nelson, Montréal, Héliotrope, 2011, 303 p., p. 141.

[schoolenthusiast]

83 reasons why I’m doing the #readingfromhome challenge

@theaspenreader and I decided to challenge ourselves in 2018 and read only from our TBR pile (required readings for school are an exception), so I decided to imitate @abreathofair and list the unread books I own to motivate myself. I need this challenge!

My goals for 2018 is to read at least 50 books.

Books in French :

Green River de Tim Willocks

Le carnet noir d’Arthur Machen

Cantique des plaines de Nancy Huston

Poétique d’Aristote

Rhétorique d’Aristote

Second début de Francine Pelletier

Contes et légendes inachevés de Tolkien

La bête humain d’Émile Zola

L’homme révolté d’Albert Camus

Homo Sapienne de Niviaq Korneliussen

La langue affranchie d’Anne-Marie Beaudoin-Bégin

L’influence d’un livre de Philippe Aubert de Gaspé

Ubu Roi d’Alfred Jarry

Essais de Montaigne

La quatrième dimension de Robert Bloch

Les meilleurs récits de Planet Stories

L’empereur de Paris de Richardson

Le ciel de Bay City de Catherine Mavrikakis

Les rois maudits tome 1 de Maurice Druon

La machine ultime d’A.E. Van Vogt

La poire sur un plateau de Jonathan Latimer

Hyperion 2 de Dan Simmons

La petite marchande de prose de Daniel Pennac

Caligula d’Albert Camus

La classe de madame Valérie de Francis Blais

La chartreuse de Parme de Stendhal

Les voies d’Anubis de Tim Powers

Le pavillon d’or de Mishima

L’assommoir d’Émile Zola

Le pendule de Foucault d’Umberto Eco

La maison éternelle d’A.E. Van Vogt

Le deuxième gant de Natasha Beaulieu

Pars vite et reviens tard de Fred Vargas

Des fleurs pour Algernon de Daniel Keyes

Le prisonnier de Thomas Disch

Le clan des Otori de Lian Hearn

Le livre noir du Canada anglais de Normand Lester

La nuit qui ne finit pas d’Agatha Christie

Books in English :

In Paradise by Peter Matthiessen

A Virtual Soul by Kevin Texeira

Murder in retrospect by Agatha Christie

Crooked House by Agatha Christie

Ender in Exile by Orson Scott Card

The Age of Innocence by Edith Wharton

The Blind Assassin by Margaret Atwood

One Flew Over the Cuckoo’s Nest by Ken Kesey

The Sirens of Titan by Kurt Vonnegut

Dear Life by Alice Munro

The Sound and the Fury by William Faulkner

Great Expectations by Charles Dickens

The Rude Story of English by Tom Howell

The Wise Man’s Fear by Patrick Rothfuss

Heart of Darkness by Joseph Conrad

The Singing Sword by White

Dreamsongs 1 by George R.R. Martin

The Vampire Armand by Anne Rice

Frankenstein by Mary Shelley

Orlando by Virginia Woolf

Blood and Gold by Anne Rice

Pandora by Anne Rice

Dawn of Prophecy by David Eddings

Child’s Play by Andrew Niederman

Foreigner by Robert J. Sawyer (last book before I’ve read them all!!)

Volumes 5, 6, 7, 8, 9, 10, 11, 12 and 13 of A series of unfortunate events by Lemony Snicket

The Merchant of Venice by William Shakespeare

Pericles by William Shakespeare

Nonzero by Robert Wright

Promethea book 2 by Alan Moore

Scenes from a Marriage by Ingmar Bergman

The Soul of a New Machine by Tracy Kidder

The Players of Null-A by A.E. Van Vogt

Virtual Light by William Gibson

Young Lust by Kathy Acker

Mindreading and false belief by Eva-Maria Ehrhardt

Theory of Games and Economic Behavior by John von Neumann and Morgenstern