#the Neurodegenerative Disease

[hrsychannel]

the Neurodegenerative Disease

A Comprehensive Guide to the Neurodegenerative Disease that Affects Behavior, Personality, and Language

Frontotemporal Dementia // Pixabay

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Frontotemporal dementia (FTD) 

is a neurodegenerative disease that affects the frontal and temporal lobes of the brain. It is a progressive condition that gradually impairs a person's cognitive and language function, as well as their behavior and personality. FTD is a relatively rare form of dementia, accounting for only about 5-10% of all dementia cases, but it can have a significant impact on individuals and their families.

FTD is actually a group of diseases that share similar clinical features, including changes in personality and behavior, loss of empathy, and language difficulties. There are three main subtypes of FTD, which are distinguished by their symptoms and the parts of the brain that are most affected.

There are three main subtypes of FTD, which are distinguished by their symptoms and the parts of the brain that are most affected:

The first subtype is behavioral variant FTD (bvFTD), which is characterized by changes in behavior and personality. People with bvFTD may experience apathy, loss of empathy, impulsivity, and inappropriate social behavior. They may also exhibit a lack of inhibition, including making inappropriate comments or gestures, or acting in socially unacceptable ways. Changes in eating habits, including overeating, food cravings, and a preference for sweet foods, are also common. Difficulty with planning, organization, and decision-making are also hallmark features of bvFTD.

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The second subtype is semantic variant primary progressive aphasia (svPPA), which is characterized by progressive difficulty in understanding and using language. People with svPPA may have difficulty recognizing familiar objects and faces, loss of knowledge of common facts and concepts, and may exhibit changes in behavior and personality, including apathy and social withdrawal.

The third subtype is nonfluent variant primary progressive aphasia (nfvPPA), which is characterized by difficulty with speech production. People with nfvPPA may experience stuttering, word-finding difficulty, and halting speech. They may also have difficulty understanding complex sentences and following conversations, and they may experience a loss of ability to write or read fluently. Like svPPA, people with nfvPPA may also exhibit changes in behavior and personality, including apathy and social withdrawal.

FTD typically affects people between the ages of 40 and 65, and it is more common in men than women. Unfortunately, there is currently no cure for FTD, and treatment is focused on managing symptoms and improving quality of life. Medications such as antidepressants, antipsychotics, and mood stabilizers may be used to manage behavioral symptoms such as agitation and aggression. Speech therapy and occupational therapy may be used to help maintain communication skills and improve functional abilities.

FTD can be challenging for both the person with the disease and their family members. The changes in behavior and personality can be particularly difficult to deal with, and it can be hard to adjust to the loss of language and communication skills. However, there are many resources available to help people cope with FTD, including support groups, counseling, and respite care.

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In conclusion, frontotemporal dementia is a complex and challenging disease that affects cognitive, behavioral, and language function. It is important to be aware of the symptoms of FTD and to seek a proper diagnosis if you or a loved one is experiencing any of them. While there is currently no cure for FTD, there are treatments available to manage symptoms and improve quality of life. With the right support and care, people with FTD can maintain a good quality of life for many years.

[newsfromstolenland]

There’s a renewed call for a public inquiry into why so many New Brunswickers have come down with neurodegenerative symptoms and illnesses.

When an initial cluster of over 40 people presented with similar symptoms – muscle spasms, atrophy and progressive dementia – it was thought to be Creutzfeldt-Jakob disease, until they tested negative.

That’s when a Moncton-based neurologist thought they could be experiencing an unknown brain disease. The cluster was initially focused in the Moncton and Acadian Peninsula areas of the province.

But after over a year of confusing back-and-forth for patients and their families, the province of New Brunswick halted further investigation by its federal counterparts, saying there was an explanation for each patients’ illness.

This week, New York Times Magazine released an article that calls that into question, citing that neurologist Dr. Alier Marrero believes the number of people seeking help with similar symptoms is now over 400.

It has sparked Green Party MLA Megan Mitton to call for a public inquiry into what happened internally between the province and Ottawa, as well as further testing, including environmental, to try and find patients some answers.

Full article

Tagging: @allthecanadianpolitics

[laureala]

AMELIA SHEPHERD

— in Grey's Anatomy 20.01 - We've Only Just Begun

You told me to say whatever's in my- I'm sorry! It's making me think that maybe Meredith isn't insane.

[mindblowingscience]

A spectrum of neurodegenerative diseases, including frontotemporal dementia (FTD), progressive supranuclear palsy (PSP), and corticobasal degeneration (CBD) are due to the accumulation of abnormal, misfolded tau proteins in the brain. A team of researchers has found potential ways to interrupt this process by targeting “sticky” sites along the long form of mutated tau, preventing the misfolding and spreading of the neurofibrillary tangles.

Continue Reading.

[chicago-geniza]

Did you know. Even if you stop drinking alcohol. You can still drink so much non-alcoholic beer you throw up from acid reflux. Anyway I'm too old to kin fictional characters but some part of me...some part of me does admittedly feel a deep and abiding kinship with Daniel Molloy

[ohno-the-sun]

Woot woot more mad scientist au doodles! 

Credit to @oobbbear​ 

Another doodle under the cut (its my favorite but its gory so be warned)

[swinging-stars-from-satellites]

having a hypochondriac moment pray for me babes

[idontmindifuforgetme]

My forever struggle is that I’m in love with both neuroscience and medicinal/organic chemistry and I’m just insane enough to be double dipping in both constantly

[bpod-bpod]

Painting Proteins

To understand a huge ecosystem like a rainforest, you might need to watch a few individual species closely: which animal eats which, or how individual trees affect the surrounding area. At the cellular level, countless individual proteins interact in the same way, but these are even harder to track than animals in the jungle. A new protein-labelling approach, called SUM-PAINT, makes it possible, and researchers have created an atlas of brain cells at the level of individual molecules, tracking 30 different types of protein and unravelling the complexity of 900 synapses – junctions between cells. Looking at the whole system and individual brain cells (pictured, with snapshots of 30 proteins overlaid to show the cumulative complexity) the team discovered a new type of synapse, demonstrating a tool that they hope will help reveal hidden details of cell biology, and lead to new treatment approaches for neurodegenerative diseases.

Written by Anthony Lewis

Image from work by Eduard M. Unterauer, Sayedali Shetab Boushehri and Kristina Jevdokimenko, and colleagues

Max Planck Institute of Biochemistry, Planegg, Germany

Image originally published with a Creative Commons Attribution 4.0 International (CC BY 4.0)

Published in Cell, March 2024

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[nightmaretour]

If you're going to call out specific groups of people who aren't allowed to relate to your posts (instead of just, I don't know... ignoring people who don't respond to your posts the way you'd like?), can you at least get your facts straight? ADHD absolutely can cause severe memory issues. Real, actual, severe, debilitating memory problems. You're letting your (understandable!) frustration with certain people who do talk over other people with more severe disabilities turn into actual intracommunity ableism, and it sucks.

Lmao clearly you don't understand what "severe" means. Your ADHD does not cause you memory issues on par with anterograde amnesia from a moderate-severe brain injury. It does not cause you memory issues on par with dementia. You assume you can relate because you think you're the most disabledest ever and nobody can possibly suffer more than you, but you really can't. Some people are more disabled than you, that isn't an attack on you personally. It isn't ableism to tell you that something isn't about you, which it isn't.

I wouldn't have to add things like that to posts if people just like you didn't CONSTANTLY insist on making posts by people with more severe disabilities than you all about yourselves and derailing them because you find more severely disabled people invalidating. THAT'S ableism. You try dealing with that on almost every single post you make, no, every time you try to talk about your experiences, then you can talk to me about "just ignoring it". Sometimes things just aren't about you, learn to deal with it and respect other disabled people's boundaries, or leave the rest of us the fuck alone.

[evolve-rat]

We’ve moved past people misusing the word twink and people are now calling old Daniel Malloy a bear. Day ruined

[floofyfluff]

we're in this phase III trial of this thing that is soooooooo cool and i want to talk about it sooooo bad but 1. no. 2. no one will understand me if i do. 3. no.

but its so wild to watch disease processes go from totally untreatable to like. one time novel solution. in half of a lifetime. like from "not only do we not know why this is happening or how to stop it but i can tell you that you're just going to go blind," to "well if you come in and get this done every x weeks actually you will preserve most of your vision" to "actually maybe we can just do this one procedure and the thing that robbed 25% of your family of the ability to read by age 75 will simply not be a problem for you"

[thermometermercury]

*watching Hannibal saw will’s head open with the intent to eat his brain* oh no girl! :0 Watch out for those prions!!!

[lornteeleaf]

Loosing a parent to a neurodegenerative disorder feels like watching them die over months and years.

We're almost 7 years in at this point. I'm still shocked every time there is a noticeable loss of function. I should be used to it by now but I'm not.

I still get frustrated and angry. With the situation, not with them. But at the end it's my parent I lash out against.

I'm still so sad and lost. It's been almost a year in therapy, I found my ways to grieve now and have plans how to grieve after they're gone. But I still cry myself to sleep sometimes.

[chicago-geniza]

Love when you put on the armored-tank exoskeleton neck brace and the brain fog so impenetrable you couldn't hold a thought for more than 10 seconds immediately clears up. Like I'm sure my cervical spine's structural integrity is fine and there is nothing to worry about. I'm sure the years of cervical dystonia and being unable to support my head are just because I'm out of shape

[devybobevy]

I hate that I spent my entire childhood promising that I'd never be anything like my mother just to find out that she is the way she is because of Huntington's disease and that whether or not I end up like her is in the hands of a genetic coin toss that happened at my conception