#neurogenic simulation

[captaingimpy]

Exploring Mortality and the Human Experience in The Orville: Reflections on "The Mortality Paradox"

The Orville has always been more than just a science fiction series; it’s a philosophical exploration of the human condition, filled with deep questions about life, death, and everything in between. In Season 3, Episode 3, “The Mortality Paradox,” the show delves into the concept of mortality in a way that is both thought-provoking and deeply resonant. In this episode, the crew of the Orville…

[1-800-headmates]

welcome to 1-800-headmates!

we are another build a headmate blog!

requests are CLOSED!

we are anti-rad.queer and anti-trans.id. leave.

all info + dni under the cut ^^

this blog is run by two mods!

mod ideal is bodily 21 and has both a neurogenic monocon median system named the abstract system and an endogenic polycon system named the ideal system! please use plural they/them to refer to them unless members state otherwise! you can find them over on @idealism-world! their favorite aesthetics are weirdcore and webcore <3

mod sewers is bodily 22 and is a polyfrag DID system with mixed origins alters named the sewer system! please use collective he/it/any for mod sewers unless member state otherwise! its favorite aesthetics are scenecore and old webcore!

mod ideal and mod sewers are dating <333

this blog has nothing to do with selling headmates and does not believe in system hopping.

for your request, be as specific or as vague as you'd like! you can say "can i get a fictive of (so-and-so) please" or something like "can i get a 18 yr old split fictive of X and Y who has ghost and cat themed pronouns and genders please" both these are fine, no ask format needed!

we will not do factives (sorry!) but we will do fictives of media we know, which includes:

BUNGOU STRAY DOGS, danganronpa, persona series (especially 5), chainsaw man, needy streamer overload, homestuck, doki doki literature club, borderlands, creepypasta, slenderverse, marble hornets, regretevator, pet simulator 99, my little pony, the world ends with you, yugioh (specifically duel monsters or season 0/the toei anime), death note, percy jackson (pjo + hoo), steven universe, invader zim, boku no hero academia, pokemon (all pokemon, and we can try humans)

these are in no particular order and range from past and current hyperfixes to media we've consumed enough of to feel confident making headmates for, except bungou stray dogs, which is our mutual hyperfixation. request bungou stray dogs please 🙏🙏🙏

also we will not do god/above god related headmates for alterhuman reasons, sorry! fictives of god or godlike characters are fine. gods from mythology do not count as characters and we won't do those, but we can do inspired ones :>

here's our format!

* means you have to request these to be added, or else they'll be left out! you can request for any parts to be left out though!

Name(s):

Age:

Pronouns:

*Orientation:

Gender(s):

Likes:

Dislikes:

*Identity: (desirdae/dissomei/alter flags/etc!)

*Kins: (you can specify if you want fictionkins or otherkins, if you just say kins we'll do both!)

*Emoji(s):

*Typing quirk:

*Music: (could be bands or a few song!)

Faceclaim/Moodboard:

we won't assign headmates roles but you can ask for specific personalities that are similar! (ex. if you want a caregiver you can ask for someone caring and responsible)

mod sewers will sometimes do art for headmates!

any headmates requested here are okay to form by default! if you would like to ask that a requested headmate not be replicated outside your system, please add that in your original request ask!

if someone asks that, please don't actively try to form them! however please keep in mind that we have no way of enforcing this rule and also accidental formings happen and if you accidentally form someone requested to not be shared, that is alright :)

if you do form alters from this blog we greatly appreciate asks or reblogs letting us know and how it’s going and from the formed alters specifically are also very nice !!! :D

also if you do form sysmates from this blog feel free to use this userbox and/or @ us in posts about them! credit is completely optional (bc like. thats a person now and theyll obviously change a lot from original design) but i would request at least a like or reblog cuz they make us happy

do not interact

rad.queers, rad.queers neutrals, anti endos, proshippers, zionists, anti contridicatary labels, anti self-dx, anti mogai, people who believe in "trans.androphobia" and fakeclaimers.

[tahkannibal]

Tahkawin / Kevin / Vampyr / WTVR U WANNA CALL ME !!!!!!!!!!!!!

CERTIFIED GOONER !!!!!!

☆ General warning for my acc : gore, references to horror, mentions of personal trauma, violence and other stuff

☆ He/xey/it, intersex afab, bigender 2spirit boygirl lesbian leatherdyke ^_^

☆ I am autistic and apart of a traumagenic/neurogenic system, I also have BPD, STPD and ADHD.

☆ I like Metalocalypse, China IL, Postal, Hatred, Smiling Friends, Resident Evil, Rob Zombie media, View Askew, Dolls, MLP, South Park, Kevin Spencer, Pinky and Pepper Forever, Lupin III, Layers of Fear, simulator games, Kurtis Conner, Lolcow drama, true crime (NOT TCC, TCC KYS) ect. BIG OL GOOFBALL !!!

☆ I am autistic (as previously mentioned) my special interests are Resident Evil, Adult Swim Media and Care Bears.

☆ I selfship with multiple characters, I am fine with doubles EXCEPT for Chop-Top Sawyer. I'm sorry this is really stupid but it upsets me, I'm not one of those crazy ass "he's MINE!!! ONLY I CAN LIKE HIM!!!" kinda ppl doubles just upset me.

☆ Please refrain from mentioning the name Kass, topics of eating disorders of self harm, jokes about my f/o's being problematic (even if they are it still upsets me lolz ...) and disturbing (problematic) paraphilias (specifically necrophilia and pedophilia, they upset me a lot due to past experiences.) Also people who actively search for real life gore can fuck off I think u suck BIG TIME

☆ Do not interact if you are not pro-Palestinian or "have no opinion" on the "war" (genocide.) Please dni if you're anti-SJW/anti-PLUR, heavy right wing or what people consider bigotted.

☆ I am a white passing Ojibwe native. I grew up within the community and culture. I may say slurs but all of them are reclaimed.

My Flagz !!

Tagz + Linkz !!

Artblog : @tahdrawin

My carrd :

(Carrd is being reworked so sorry)

[nobantucelemyot]

From Theory to practice: first week of fieldwork

Time is an invaluable asset, especially for an occupational therapy student on their first day of fieldwork practical’s. The limited amount of time given is usually expended by the initial interview when getting to know the client and building up a picture of the client as a whole, screening to observe their  cognitive and perceptual capacity, identifying psychosocial issues  to be able to refer them to appropriate multidisciplinary team members for further assessment and intervention, providing information for setting goals, and providing  information from which a baseline for intervention can be formed. This becomes a challenge when working with a client who has a communication disorder.

On my initial encounter with the client who sustained a left cerebrovascular accident, I instantly became aware of their speech impairments since most if not all responses they gave were the same and unreliable, they were nodding their head and replied with a yes to all questions including those that required descriptions or a single word, only one word was produced whenever speech was attempted.  When it was realized that my client has expressive aphasia, which is an acquired communication disorder caused by brain damage to the language Centre in the dominant side of the brain usually in a right-handed client(Sarno,1994), a new strategy had to be formulated and had to identify communication methods that can be used to assist both my client and I to achieve both our goals.

The methods used for communication were initially gestures, body language and facial expressions along with speech to emphasize and demonstrate what was required. Adaptations to assessments were made to suit the client’s condition. To assess cognition ,pictures were utilized where the client had to point as a response, she was to identify shapes, colours, types of weather conditions and appropriate clothing for  an appropriate weather,  time of day in pictures where a moon or the sun was out,  her memory and attention were also assessed with the use of pictures . In the assessment of activities of daily living such as toothbrushing and dressing, the student therapist had to simulate the task with the use of hands and available items as an indication to the client on what to do.

The first treatment session involved teaching the client a hemiplegic method of dressing the UL using a shirt, in her case using a dress with buttons. The session did not go according to plan, few adaptations had to be made, as the student therapist had not previously considered the client’s current sitting balance, instead of using a plinth the client performed the tasked while seated in her wheelchair for safety purposes. Initially, gestures were used to teach the method, as she failed to perform the student therapist used another dress, and did the task simultaneously with the client, thus her performance started to improve. The supervisor suggested that it would have been more successful or less complex for the client if I was sitting side by side with the client while doing the task, the client would have been less confused with the use of left and right hand in the task. Another issue was weight bearing on the feet, the wheelchair had only one foot-rest, one foot was not supported, using a substitute item to compensate for the missing footrest was suggested.

Receiving feedback whether positive or negative will improve my clinical reasoning as well as my performance in implementing therapy for my clients, I believe there’s still a lot to learn about clients with Aphasia, and with time and dedication, a great therapist in me will surface.

References

American Heart Association. (1994). Caring for the person with aphasia. Brochure. Dallas: American Heart Association.

Sarno, M. T. (1994). Neurogenic disorders of speech and language. In S.B. O’Sullivan & T. J. Schmitz (Eds.), Physical rehabilitation: Assessment and treatment (3rd ed., pp. 633–653). Philadelphia: Davis.

[erectiledysfunc]

tyrosine erectile dysfunction

Contents

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Public health problem

Million people worldwide

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Determination of amino acid in 10 patients showed high levels of glycine, low levels of tyrosine, and a low Fischer ratio. Two patients did not receive chemotherapy and died. Twenty-two out of 25.

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The introduction of novel tyrosine kinase inhibitors into clinical practice has rendered CML a manageable disease for which the prognosis changed significantly in the last 10 years. The efficacy.

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Nelson Textbook of Pediatrics, 19th ed. Philadelphia, PA: Saunders Elsevier; 2011. Mahoney CR, Castellani J, Kramer FM, Young A, Lieberman HR. Tyrosine .

L-Tyrosine May Reverse Erectile Dysfunction. A study published in 2015 showed that L-tyrosine may reverse neurological ED symptoms in mice.

Erectile Dysfunction John P. Mulhall is Director of the Male Sexual and Reproductive Medicine Program, which is devoted entirely to the care of men who have suffered sexual difficulties or fertility.

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Additional abstracts include findings from a study of the pharmacokinetics of lucitanib, an oral, potent inhibitor of tyrosine kinase activity, in a simulated patient population to inform.

This report provides strong evidence that a neurotrophic tyrosine kinase receptor type 1 monoclonal antibody (TrkA-mAb) inhibits sympathetic nerve.

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[healthtimetaylor]

Protective role of berberine on ulcerative colitis through modulating enteric glial cells-intestinal epithelial cells-immune cells interactions.

PMID:  Acta Pharm Sin B. 2020 Mar ;10(3):447-461. Epub 2019 Sep 5. PMID: 32140391 Abstract Title:  Protective role of berberine on ulcerative colitis through modulating enteric glial cells-intestinal epithelial cells-immune cells interactions. Abstract:  Ulcerative colitis (UC) manifests as an etiologically complicated and relapsing gastrointestinal disease. The enteric nervous system (ENS) plays a pivotal role in rectifying and orchestrating the inflammatory responses in gut tract. Berberine, an isoquinoline alkaloid, is known as its anti-inflammatory and therapeutic effects in experimental colitis. However, little research focused on its regulatory function on ENS. Therefore, we set out to explore the pathological role of neurogenic inflammation in UC and the modulating effects of berberine on neuro-immune interactions. Functional defects of enteric glial cells (EGCs), with decreased glial fibrillary acidic protein (GFAP) and increased substance P expression, were observed in DSS-induced murine UC. Administration of berberine can obviously ameliorate the disease severity and restore the mucosal barrier homeostasis of UC, closely accompanying by maintaining the residence of EGCs and attenuating inflammatory infiltrations and immune cells overactivation., berberine showed direct protective effects on monoculture of EGCs, bone marrow-derived dendritic cells (BMDCs), T cells, and intestinal epithelial cells (IECs) in the simulated inflammatory conditions. Furthermore, berberine could modulate gut EGCs-IECs-immune cell interactions in the co-culture systems. In summary, our study indicated the EGCs-IECs-immune cell interactions might function as a crucial paradigm in mucosal inflammation and provided an infusive mechanism of berberine in regulating enteric neurogenic inflammation.

read more

[ianmkeenan]

Do Your Research - Search for Articles on Cannabinoids and CBD

At That's Natural, we believe it is your natural and constitutional right to consume plant-based medicines that have no known negative side effects.  As a consumer, it is up to YOU to do your due diligence and research and decide what is best for you!

Here is a list of some research that we have compiled, you can also go to the National Institutes of Health or Google Scholar and search through thousands of research articles on cannabinoids like CBD as well as the Endocannabinoid System that exists in each of us!

Anxiety

1- The anxiolytic effect of cannabidiol on chronically stressed mice depends on hippocampal neurogenesis: involvement of the endocannabinoid system

2- The anxiolytic-like effects of cannabidiol injected into the bed nucleus of the stria terminalis are mediated by 5-HT1A receptors

3- Plant-based medicines for anxiety disorders, part 2: A review of clinical studies with supporting preclinical evidence

4- Effects of cannabidiol (CBD) on regional cerebral blood flow

5- Central anandamide deficiency predicts stress-induced anxiety: behavioral reversal through endocannabinoid augmentation

6- Cannabidiol reduces the anxiety induced by simulated public speaking in treatment-naïve social phobia patients

7- Neural basis of anxiolytic effects of cannabidiol (CBD) in generalized social anxiety disorder

8- Cannabidiol, a Cannabis sativa constituent, as an anxiolytic drug

9- Antidepressant-Like and Anxiolytic-Like Effects of Cannabidiol: A Chemical Compound of Cannabis Sativa 

10- Cannabidiol induces rapid-acting antidepressant-like effects and enhances cortical 5-HT/glutamate neurotransmission: role of 5-HT1A receptors

11- Cannabidiol: Pharmacology and potential therapeutic role in epilepsy and other neuropsychiatric disorders

Alzheimer’s & Dementia

1- Endocannabinoid signalling in Alzheimer’s disease

2- A molecular link between the active component of marijuana and Alzheimer’s disease pathology

3- Neuroprotective effect of cannabidiol, a non-psychoactive component from Cannabis sativa, on beta-amyloid-induced toxicity in PC12 cells

4- The Role of Endocannabinoid Signaling in the Molecular Mechanisms of Neurodegeneration in Alzheimer’s Disease

5- Cannabinoids for the treatment of dementia

6- Cannabidiol in vivo blunts beta-amyloid induced neuroinflammation by suppressing IL-1beta and iNOS expression

7- Cannabidiol: A promising drug for neurodegenerative disorders?

8- Cannabinoid receptor 1 deficiency in a mouse model of Alzheimer’s disease leads to enhanced cognitive impairment despite of a reduction in amyloid deposition

9- The role of the endocannabinoid system in Alzheimer’s disease: facts and hypotheses

10- The role of phytochemicals in the treatment and prevention of dementia

11- Cannabinoids for the treatment of dementia

12- Cannabidiol Promotes Amyloid Precursor Protein Ubiquitination and Reduction of Beta Amyloid Expression in SHSY5YAPP+ Cells Through PPARγ Involvement

13- Cannabinoids for treatment of Alzheimer’s disease: moving toward the clinic

14- In vivo Evidence for Therapeutic Properties of Cannabidiol (CBD) for Alzheimer’s Disease

Arthritis & Anti-Inflammatory

1- The nonpsychoactive cannabis constituent cannabidiol is an oral anti-arthritic therapeutic in murine collagen-induced arthritis

2- Involvement of the endocannabinoid system in osteoarthritis

3- The abnormal cannabidiol analogue O-1602 reduces nociception in a rat model of acute arthritis via the putative cannabinoid receptor GPR55

4- Cannabinoid-mediated antinociception is enhanced in rat osteoarthritic knees

5- Preliminary assessment of the efficacy, tolerability and safety of a cannabis-based medicine (Sativex) in the treatment of pain caused by rheumatoid arthritis

6- Cannabidiol (CBD) and its analogs: a review of their effects on inflammation.

7- Cannabidiol exerts sebostatic and antiinflammatory effects on human sebocytes. 

8- Cannabidiol-transdermal delivery and anti-inflammatory effect in a murine model.

9- HU-444, a Novel, Potent Anti-Inflammatory, Nonpsychotropic Cannabinoid.

10- HU-446 and HU-465, Derivatives of the Non-psychoactive Cannabinoid Cannabidiol, Decrease the Activation of Encephalitogenic T Cells. 

11- Pathways and gene networks mediating the regulatory effects of cannabidiol, a nonpsychoactive cannabinoid, in autoimmune T cells

Asthma & Allergies

1- Activation of cannabinoid receptors prevents antigen-induced asthma-like reaction in guinea pigs

2- The role of cannabinoids in inflammatory modulation of allergic respiratory disorders, inflammatory pain and ischemic stroke

3- Endogenous cannabinoid receptor agonists inhibit neurogenic inflammations in guinea pig airways

Autism

1- Mutations found in individuals with autism interfere with endocannabinoid signaling in the brain

2- Cannabinoid receptor type 2, but not type 1, is up-regulated in peripheral blood mononuclear cells of children affected by autistic disorders

3- A novel approach to the symptomatic treatment of autism

4- Consequences of cannabinoid and monoaminergic system disruption in a mouse model of autism spectrum disorders

5- Targeting the endocannabinoid system in the treatment of fragile X syndrome

Cancer & Tumors

1- Cannabidiol inhibits growth and induces programmed cell death in kaposi sarcoma-associated herpes virus-infected endothelium

2- Cannabidiol as potential anticancer drug

3- Cannabidiol inhibits cancer cell invasion via upregulation of tissue inhibitor of matrix metalloproteinases-1

4- Cannabidiol inhibits angiogenesis by multiple mechanisms

5- The inhibitory effects of cannabidiol on systemic malignant tumors

6- HU-331, a novel cannabinoid-based anticancer topoisomerase II inhibitor.

7- Cannabidiol as potential anticancer drug.

Cancer-Related Pain

1- Multicenter, double-blind, randomized, placebo-controlled, parallel-group study of the efficacy, safety, and tolerability of THC:CBD extract and THC extract in patients with intractable cancer-related pain

2- Nabiximols for opioid-treated cancer patients with poorly-controlled chronic pain: a randomized, placebo-controlled, graded-dose trial

Bladder Cancer

1- TRPV2 activation induces apoptotic cell death in human T24 bladder cancer cells: a potential therapeutic target for bladder cancer

Brain Tumors

1- Antitumor effects of cannabidiol, a nonpsychoactive cannabinoid, on human glioma cell lines

2- Cannabidiol enhances the inhibitory effects of delta9-tetrahydrocannabinol on human glioblastoma cell proliferation and survival

3- Systematic review of the literature on clinical and experimental trials on the antitumor effects of cannabinoids in gliomas

4- Cannabidiol inhibits human glioma cell migration through a cannabinoid receptor-independent mechanism

5- Id-1 is a key transcriptional regulator of glioblastoma aggressiveness and a novel therapeutic target

6- Local delivery of cannabinoid-loaded microparticles inhibits tumor growth in a murine xenograft model of glioblastoma multiforme

7- Cannabidiol, a non-psychoactive cannabinoid compound, inhibits proliferation and invasion in U87-MG and T98G glioma cells through a multitarget effect

8- Triggering of the TRPV2 channel by cannabidiol sensitizes gli