#fatty liver grade 2

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Fatty liver disease occurs when excess fat builds up in liver cells, making it harder for the liver to function. Grade 2 fatty liver means that there’s moderate fat accumulation, typically between 30% and 60% of your liver cells, which can lead to inflammation or scarring if not treated.

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Fatty Liver Symptoms, Grade, Causes, Complications, Prevention

Fatty liver, also known as hepatic steatosis, is a condition in which there is an abnormal buildup of fat in the liver cells. This can lead to inflammation and damage to the liver. Fatty liver is a very common condition, affecting up to 30% of adults in the United States.

Grade 1 fatty liver

Grade 1 fatty liver is the mildest form of fatty liver. In this stage, the accumulation of fat in the liver cells is minimal and does not cause significant liver damage. However, it is important to address this condition promptly as it can progress to more severe stages of fatty liver disease, such as grade 2 or 3.

Grade 2 fatty liver

Grade 2 fatty liver is a moderate form of fatty liver. In this stage, there is a moderate buildup of fat in the liver cells. This can lead to inflammation and damage to the liver, and may also increase the risk of developing liver disease.

Causes of fatty liver

The exact cause of fatty liver is not fully understood, but it is thought to be caused by a combination of factors, including:

Obesity

Diabetes

High blood pressure

High cholesterol

Overconsumption of alcohol

Certain medications, such as steroids

Certain medical conditions, such as polycystic ovary syndrome (PCOS)

Symptoms of fatty liver

Most people with grade 1 or 2 fatty liver do not have any symptoms. However, some people may experience:

Fatigue

Right upper abdominal pain

Nausea

Weight loss

Yellowing of the skin (jaundice)

Diagnosis of fatty liver

Fatty liver can be diagnosed with a blood test, an ultrasound, or a liver biopsy.

Treatment for fatty liver

The goal of treatment for fatty liver is to reduce the amount of fat in the liver and prevent further damage. Treatment options include:

Weight loss

Exercise

A healthy diet

Medications to lower cholesterol and blood sugar

Quitting smoking

Prevention of fatty liver

The best way to prevent fatty liver is to maintain a healthy weight, eat a healthy diet, and exercise regularly.

If you are concerned that you may have fatty liver, talk to your doctor. Early diagnosis and treatment can help prevent the condition from progressing to more serious stages.

Here are some additional things you can do to help prevent fatty liver:

Limit your intake of sugary drinks and processed foods.

Choose lean protein sources, such as fish, chicken, and beans.

Eat plenty of fruits, vegetables, and whole grains.

Limit your intake of saturated and trans fats.

If you drink alcohol, do so in moderation.

By following these tips, you can help keep your liver healthy and reduce your risk of developing fatty liver.

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Excess weight and IgG food allergies

Inflammatory processes that promote obesity may be intensified by an IgG food allergy

Obesity is a chronic disease which is characterized by severe excess weight and a pathologically increased body fat percentage. It is very stressful for the body and often a psychological burden for the people affected. Excess weight and obesity may be due to metabolic disorders.

The exact mechanisms of the disease are not completely understood yet. However, it is clear that low-grade inflammatory processes play a role in obesity. An IgG food allergy may intensify such inflammatory processes. ImuPro helps you to detect such delayed food allergies and supports you with individual guidelines based on your test results. Learn more.

Obesity increases health risks for:Obesity and excess weight pose a major risk for serious diet-related chronic diseases.

If a person is obese or overweight, this means that he or she is carrying excess body fat. The balance between calories-in and calories-out differs for each person. Factors that might affect your weight include your genetic makeup, overeating, monotonous diet, excess carbohydrate and fat intake, and not being physically active.

Obesity increases health risks for:

Heart disease

Stroke

Arthritis

Some cancers

The diagnosis of obesity

Body mass index (BMI) is a simple index of weight-for-height that is commonly used to classify excess weight and obesity in adults. It is defined as a person’s weight in kilograms divided by the square of their height in meters (kg/m2). People with BMI > 25 kg/m2 meet the criterion for being overweight (BMI 25-29.9). If they have a BMI > 30, it indicates obesity. However, to get a complete clinical picture it is also important to analyse whether the weight gain negatively impacts the health of a patient.

The causes of obesity

While many factors may influence an individual’s weight, excess weight and obesity are due mainly to an imbalance of energy intake from the diet and energy expenditure (through physical activities and bodily functions). Genetic and environmental factors play a role, but attention to diet and physical activity is important not only for preventing weight gain, but also for weight loss and subsequent maintenance.

With obesity, systemic inflammatory activities are found. Inflammatory response to food induces the production of messenger proteins like TNF-alpha.

The messenger substance TNF-alpha blocks the receptors for insulin and so impedes the cell’s energy supply. The blood sugar present in the blood cannot or can only be transported to a restricted extent into the cell where it is urgently needed. This may activate two mechanisms:

1. Sugar becomes fat

Since the sugar remains in the blood, the blood sugar level remains elevated. The liver transforms the blood sugar increasingly into fatty acids which are then deposited in the fat cells. The blockage of the insulin receptors results in more and more insulin being released. The increased insulin concentration prevents the reconversion of fatty acids into blood sugar. The means that the energy stored in the fatty deposits is no longer available to the body.

2. Energy consumption decreases

The Individual cells have less energy at their disposal. Therefore, the cell activity is reduced and the basal metabolic rate, i.e. the quantity of calories used at rest, diminishes.

In the case of an IgG food allergy the immune system produces IgG antibodies against certain food stuffs. IgG antibodies are created in increasing quantities, TNF alpha will be released in larger amounts and more receptors intended for insulin will be blocked. This means that the inflammatory processes may be intensified.

The scientific approach to obesity and IgG

The identification of foodstuffs that may trigger inflammatory conditions could offer a good possibility to exert a positive influence on the inflammation through a corresponding change in diet. IgG seems to be the best indicator for identifying a food as being pro-inflammatory. Every time a food comes in contact with a corresponding IgG antibody, an inflammatory response is launched. Studies have shown that an IgG guided diet has a positive effect on weight loss and stabilisation of the glucose https://imupro.com/glossary/inflammation/

.

For example, the study “IgG antibodies against food antigens are correlated with inflammation and intima media thickness in obese juveniles” Wilders-Truschnig et al. ECED published online 2007; DOI 10.1055/s-2007-993165showed a significant correlation between obesity, elevated IgG antibodies and inflammation. Thus, elevated IgG levels for food could be involved in the development of excess weight.

[susantaylor01]

How to Remove Gut Fat

Anatomy of the gut region

The abdominal or gut region generally consists of several muscles and layers of fat that help in the mobilization of the body and the protection of the important organs of the body. Gut fat mainly resides in the abdominal cavity and is known as visceral fat and it surrounds essential organs like the liver, intestine, and pancreas. The main muscles of the abdominal wall include:

Fig: Muscles of the Gut region

Rectus Abdominis: It is a muscle found in pairs that runs on each side of the anterior abdominal wall vertically. It has the popular name ‘six-pack’ and it works the spine flexion.

Transversus Abdominis: Lying beneath internal obliques, it is the innermost layer of the abdominal muscles. It works like a corset and compresses the abdomen to offer core support while also assisting in forceful expiration.

Internal and External Obliques: These muscles are located alongside the abdomen on the lateral walls. The external obliques are outside and the internal obliques come right below them thus making them the outer layer muscles of the abdominal wall. These muscles help in twisting and side-bending movements of the trunk.

Linea Alba: This is a connective tissue that lies along the midline of the abdomen and acts like a series of bundles of fibers. It is between the left and right rectus abdominis muscles and provides an origin for the two different muscles in the abdomen.

Subcutaneous Fat: This is the relatively subcutaneous fat or the fat found between the skin and the muscle layer. While it’s not as dangerous as visceral fat, excess subcutaneous fat causes poor muscle delineation and adds inches to a waistline.

Visceral Fat: This is the last of the fat that lies beneath the skin and encloses the vital organs of the body. It is different because it is metabolically active as well as poses a higher threat to health compared to subcutaneous fat.

Physiology of Gut Fat

As described before, the physiology of gut fat includes fat deposition and energy expenditure. Hormonal and metabolic processes are closely connected with visceral fat, especially with it.

Energy Storage: Calories in food above needs are deposited within the body in the form of fat. Weight gain causes the body to accumulate this energy in fat cells distributed all over the body including the visceral fat in the abdomen.

Endocrine Function: Visceral fat is arguably the most metabolically active of all fat deposits. It releases hormones and cytokines such as leptin which is involved in the regulation of appetite and adiponectin which is involved in glucose control. Excess of this fat in the abdomen can alter these hormones leading to such ailments as insulin resistance, type 2 diabetes and metabolic syndrome.

Inflammation: The visceral fat tissue synthesizes pro-inflammatory cytokines like the TNF-α and the IL-6 that cause chronic inflammation. This low-grade inflammation is linked with cardiovascular diseases, diabetes, and some kinds of cancer.

Liver Metabolism: The visceral fat is removed through the portal blood circulation which transports it to the liver and as such, free fatty acids from the visceral fat affect the liver resulting in diseases such as non-alcoholic fatty liver disease (NAFLD).

Hormonal Imbalance: Obesity, especially central obesity can change the cortisol level, a stress hormone as well as increase estrogen level. This hormonal imbalance also continues to lead to fat storage, especially around the belly area.

Targeted Exercises for Gut Muscles:

Plank (Targets: TVA, Rectus Abdominis): Keep a forearm plank position for up to one minute without including pads. This kind of exercise influences the deep core muscles and this will help in firming the abdominal area.

Bicycle Crunches (Targets: Rectus Abdominis, Obliques): Lie back with your back flat on the floor with your legs raised to the chest level, and knees bent. Move your elbow towards the opposite knee which will look like a cycling motion. This works the obliques and the rectus abdominis.

Leg Raises (Targets: Lower Rectus Abdominis): Lie down on your back, with your legs straight. Bring both legs up pointing to the ceiling, and then slowly lower them, without the feet touching the floor. This exercise concerns the lower part of the rectus abdominis muscle.

Russian Twists (Targets: Obliques, TVA): Sit on the floor with knees bent and the feet off the floor. Rotate your upper body from side to side while using a weight. This movement helps in the contraction of the obliques and the TVA.

Mountain Climbers (Targets: Rectus Abdominis, Obliques, and Cardiovascular endurance) : Starts with the plank pose. Quickly change sides and pull each knee towards the chest. It targets every part of the abdomen, increasing your heart beats per minute which is effective in burning fat.

Role of Nutrition in Gut Fat Loss

Although exercise is very important, diet has the most potential of helping to lose visceral fat. Eating less refined sugars and trans fats and taking more fiber, lean proteins, healthy fats and whole grains can decrease the formation of fat in the gut.

Newly Discovered ‘Mountain Tea’ awakens metabolism

Conclusion

Losing gut fat thus demands knowledge of the structure of muscles in the abdominal area and specific exercises with planks, sit-ups, or mountain climber activities, among others in addition to exercising the heart. With a proper diet, all these can do a lot in enhancing the core muscle strength and thus diminishing the visceral fat hence improving the general wellbeing of an individual.

[moleculardepot]

Cod Liver Oil Fatty Acid Methyl esters

Cod Liver Oil Fatty Acid Methyl esters Catalog number: B2018118 Lot number: Batch Dependent Expiration Date: Batch dependent Amount: 1 g Molecular Weight or Concentration: N/A Supplied as: Solution Applications: a molecular tool for various biochemical applications Storage: 2-8℃ Keywords: N/A Grade: Biotechnology grade. All products are highly pure. All solutions are made with Type I ultrapure…

[rnomics]

Nutrients, Vol. 16, Pages 1929: Gut Dysbiosis Shaped by Cocoa Butter-Based Sucrose-Free HFD Leads to Steatohepatitis, and Insulin Resistance in Mice

Background: High-fat diets cause gut dysbiosis and promote triglyceride accumulation, obesity, gut permeability changes, inflammation, and insulin resistance. Both cocoa butter and fish oil are considered to be a part of healthy diets. However, their differential effects on gut microbiome perturbations in mice fed high concentrations of these fats, in the absence of sucrose, remains to be elucidated. The aim of the study was to test whether the sucrose-free cocoa butter-based high-fat diet (C-HFD) feeding in mice leads to gut dysbiosis that associates with a pathologic phenotype marked by hepatic steatosis, low-grade inflammation, perturbed glucose homeostasis, and insulin resistance, compared with control mice fed the fish oil based high-fat diet (F-HFD). Results: C57BL/6 mice (5–6 mice/group) were fed two types of high fat diets (C-HFD and F-HFD) for 24 weeks. No significant difference was found in the liver weight or total body weight between the two groups. The 16S #rRNA sequencing of gut bacterial samples displayed gut dysbiosis in C-HFD group, with differentially-altered microbial diversity or relative abundances. Bacteroidetes, Firmicutes, and Proteobacteria were highly abundant in C-HFD group, while the Verrucomicrobia, Saccharibacteria (TM7), Actinobacteria, and Tenericutes were more abundant in F-HFD group. Other taxa in C-HFD group included the Bacteroides, Odoribacter, Sutterella, Firmicutes bacterium (AF12), Anaeroplasma, Roseburia, and Parabacteroides distasonis. An increased Firmicutes/Bacteroidetes (F/B) ratio in C-HFD group, compared with F-HFD group, indicated the gut dysbiosis. These gut bacterial changes in C-HFD group had predicted associations with fatty liver disease and with lipogenic, inflammatory, glucose metabolic, and insulin signaling pathways. Consistent with its microbiome shift, the C-HFD group showed hepatic inflammation and steatosis, high fasting blood glucose, insulin resistance, increased hepatic de novo lipogenesis (Acetyl CoA carboxylases 1 (Acaca), Fatty acid synthase (Fasn), Stearoyl-CoA desaturase-1 (Scd1), Elongation of long-chain fatty acids family member 6 (Elovl6), Peroxisome proliferator-activated receptor-gamma (Pparg) and cholesterol synthesis (β-(hydroxy β-methylglutaryl-CoA reductase (Hmgcr). Non-significant differences were observed regarding fatty acid uptake (Cluster of differentiation 36 (CD36), Fatty acid binding protein-1 (Fabp1) and efflux (ATP-binding cassette G1 (Abcg1), Microsomal TG transfer protein (Mttp) in C-HFD group, compared with F-HFD group. The C-HFD group also displayed increased gene expression of inflammatory markers including Tumor necrosis factor alpha (Tnfa), C-C motif chemokine ligand 2 (Ccl2), and Interleukin-12 (Il12), as well as a tendency for liver fibrosis. Conclusion: These findings suggest that the sucrose-free C-HFD feeding in mice induces gut dysbiosis which associates with liver inflammation, steatosis, glucose intolerance and insulin resistance. https://www.mdpi.com/2072-6643/16/12/1929?utm_source=dlvr.it&utm_medium=tumblr

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Reversing Fatty Liver Disease (Grade 1): A Physician's Guide to Management

As a physician, I frequently encounter patients concerned about fatty liver disease, particularly in its earlier stages. The encouraging news is that fatty liver disease, especially Grade 1, is highly treatable and often reversible with a structured approach. This article empowers you to understand and manage Grade 1 fatty liver disease, providing a clear path towards optimal liver health.

Understanding Fatty Liver Disease

The liver, a vital organ, performs a multitude of functions, including detoxification, protein synthesis, and metabolic regulation. Fatty liver disease arises when excess fat accumulates within liver cells, potentially compromising their function. This condition manifests in two primary forms: - Nonalcoholic Fatty Liver Disease (NAFLD) (ICD-10 Code: K75.0): The most prevalent form, not caused by excessive alcohol consumption. - Alcoholic Fatty Liver Disease: Develops due to heavy alcohol use. This article focuses on NAFLD, particularly Grade 1, which denotes a mild degree of fat accumulation.

Clinical Presentation of Fatty Liver Disease

Early stages of NAFLD are frequently asymptomatic. However, as the condition progresses, you might experience: - Fatigue - Unexplained weight loss - Right upper abdominal discomfort or pain

Diagnosis of Fatty Liver Disease

If you suspect fatty liver disease, consulting your physician is paramount. Diagnosis typically involves: - Comprehensive history and physical examination: Discussing risk factors and any presenting symptoms. - Laboratory evaluation: Blood tests to assess liver function and identify potential abnormalities. - Imaging studies (optional): Ultrasound, CT scan, or MRI to visualize the liver's condition.

Treatment of Fatty Liver Disease (Grade 1): A Roadmap to Recovery

The excellent news: Grade 1 NAFLD is highly reversible! Here's a comprehensive strategy to achieve optimal liver health: - Dietary Modifications: - Prioritize whole foods: Emphasize fruits, vegetables, whole grains, lean protein sources (fish, poultry, legumes), and healthy fats (olive oil, nuts, avocados). - Limit added sugars and refined carbohydrates: Reduce sugary beverages, processed foods, white bread, and pastries. These contribute to excessive fat storage in the liver. - Moderate sodium intake: Excessive sodium intake can negatively impact liver health, so limit processed foods and high-sodium condiments. - Weight Management: - Safe and gradual weight loss: Aim for a weight loss of 1-2 pounds per week through a combination of diet and exercise. Rapid weight loss can be detrimental to your health. - Calorie tracking: Consider tracking your calorie intake to ensure a sustainable calorie deficit for weight loss. - Exercise Prescription: - Regular physical activity: Aim for at least 150 minutes of moderate-intensity exercise (brisk walking, swimming) or 75 minutes of vigorous-intensity exercise (running, cycling) per week. - Strength training: Incorporate strength training exercises two to three times a week to build muscle mass, which aids in burning calories and improving overall metabolic health. - Alcohol Consumption: - Abstain or limit alcohol intake: Alcohol can further exacerbate fatty liver disease. Ideally, abstain from alcohol altogether. If you choose to drink, limit it to one drink per day for women and two drinks per day for men.

Additional Considerations:

- Management of underlying conditions: If you have diabetes, high blood pressure, or high cholesterol, effectively managing these conditions can significantly improve your liver health. - Ayurvedic Medicine (for Grade 2+): While research is ongoing, some studies suggest certain Ayurvedic herbs might be helpful in managing NAFLD (Grade 2 and above). However, it's crucial to consult your physician before starting any herbal remedies. - Supplements: Be wary of supplements claiming to cure fatty liver disease. There's no magic pill, and some supplements can even harm your liver. Always consult your physician before taking any supplements. - Regular Follow-up: Schedule regular check-ups with your physician to monitor your progress and adjust your treatment plan as needed.

Foods to Avoid with Fatty Liver Disease:

- Sugary drinks and foods: Soda, juices, processed snacks, pastries, candies - Refined carbohydrates: White bread, pasta, white rice - Unhealthy fats: Saturated and trans fats found in fried foods, processed meats, and commercially baked goods - Red meat: Limit red meat intake, opting for lean protein sources like fish, chicken, or legumes.

Papaya and Fatty Liver Disease

There's limited scientific evidence to definitively claim papaya cures fatty liver disease. However, papaya is a healthy fruit rich in antioxidants and fiber, both beneficial for overall health. While it might not be a cure Read the full article

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Adipose Signals Regulating Distal Organ Health and Disease

Excessive adiposity in obesity is a significant risk factor for development of type 2 diabetes (T2D), nonalcoholic fatty liver disease, and other cardiometabolic diseases. An unhealthy expansion of adipose tissue (AT) results in reduced adipogenesis, increased adipocyte hypertrophy, adipocyte hypoxia, chronic low-grade inflammation, increased macrophage infiltration, and insulin resistance. This…

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What is eicosapentaenoic acid ethyl ester (EPA)?

Eicosapentaenoic acid ethyl ester(EPA), known for its cardiovascular health benefits, plays a crucial role in promoting heart well-being. Pharmaceutical-grade omega-3 fatty acid supplements produced from eicosapentaenoic acid ethyl ester, such as Vascepa, have become the preferred therapy for reducing triglyceride levels (TG).

Now let's explore eicosapentaenoic acid ethyl ester together and focus on cardiovascular health.

n-3 polyunsaturated fatty acids.

What is eicosapentaenoic acid ethyl ester (EPA)?

Eicosapentaenoic acid ethyl ester, also referred to as EPA ethyl ester or simply EPA ester, is a derivative of eicosapentaenoic acid (EPA), an omega-3 fatty acid. Eicosapentaenoic acid is a polyunsaturated fatty acid found in certain fish oils, especially in cold-water fish like salmon, mackerel, and sardines.

 The "ethyl ester" part indicates that it is an ethyl derivative commonly used in pharmaceutical formulations. This modification involves attaching an ethyl group to the carboxylic acid end of the fatty acid chain, creating a compound more suitable for certain medical applications.

 EPA ethyl ester is frequently used in the production of pharmaceutical-grade omega-3 fatty acid supplements, including the medication Vascepa. Vascepa is specifically formulated to provide concentrated, purified eicosapentaenoic acid and is approved by regulatory agencies for treating elevated triglyceride levels in adults.

What is eicosapentaenoic acid ethyl ester (EPA) used for?

1.   Supporting Cardiovascular Health

Eicosapentaenoic acid ethyl ester (EPA) is a derivative of omega-3 fatty acids and effectively lowers plasma triglyceride levels. Elevated triglycerides are a risk factor for cardiovascular disease. By promoting the reduction of triglycerides, EPA makes a significant contribution to maintaining healthy lipid profiles.

The mechanisms by which EPA ethyl ester reduces triglyceride levels include:

 Lowering the synthesis of very low-density lipoprotein (VLDL) in the liver, weakening gluconeogenesis, enhancing fatty acid β-oxidation, and reducing the amount of non-esterified fatty acids transported to the liver, thereby inhibiting triglyceride synthesis from the source.

Decreasing the enzymatic activity required for triglyceride synthesis.

Increasing the production of phospholipids in the liver.

2.   Controlling and Improving Inflammation

Inflammation is a key driver of many cardiovascular diseases. EPA has potent anti-inflammatory properties, countering excessive inflammatory responses, stimulating anti-inflammatory mechanisms, regulating leukocyte transport, and promoting the phagocytosis of apoptotic neutrophils by macrophages.

The mechanisms by which EPA ethyl ester affects the inflammatory response include:

 Altering the lipid composition of cell membranes affects their fluidity and structural integrity, influences cell signal transduction, and regulates expression, thereby altering cell activity and receptor activity.

Modulating intercellular signaling mechanisms, influencing the activation of transcription factors and gene expression.

 Improving the product spectrum of lipid-regulating factors to exert anti-inflammatory effects.

3.   Skin Health

EPA's anti-inflammatory properties extend to skin health, potentially aiding in the treatment of skin conditions such as psoriasis and eczema. Additionally, the EPA's contribution to maintaining overall cell health can promote a radiant and healthy complexion.

What is eicosapentaenoic acid ethyl ester (EPA)?

In the realms of health and nutrition, eicosapentaenoic acid ethyl ester proves to be a versatile and effective ally. Embracing the power of this compound, supported by scientific insights and centuries of wisdom, can elevate your health to new levels.

We provide EPA in various purities, enabling you to choose a specification that aligns with your specific needs. You can contact us for various purities EPA. We enforce rigorous purity controls on the crucial impurity DHAee to guarantee its appropriateness for pharmaceutical formulations. While our suggested purity is 70%, we also provide EPAee with purity levels of 80%, 90%, and 98%.

[miracle-drinks]

If you're struggling with fatty liver, you need to know about these hidden foods for fatty liver ! In this video, we're going to share with you the foods that are known to treat your fatty liver, and what you can eat to prevent it. Today, we're tackling the ever-popular miracle foods for fatty liver. Because, apparently, eating your way out of years of unhealthy habits is a thing now. Let's dive into what foods are said to cure your liver, and I'll sprinkle in some harsh truths along the way. If you're experiencing any of the following symptoms, it's time to get to know your diet better: excessive weight gain, fatigue, persistent difficulty focusing, and excessive urination. The content discusses dietary recommendations for individuals with fatty liver grade 1 and grade 2. It emphasizes the importance of avoiding excessive salt and water intake, as well as certain food items. The content also mentions the benefits of prebiotics, the consumption of specific juices, and a flax-based diet. It provides guidelines for preparing and consuming fish and chicken and suggests a remedy for cold, cough, and fever using spices. Contact Us : 📞808-69043800 Visit Our Website : https://miracledrinks.in/ Re-commanded Diet and Foods for Fatty Liver. [🥦] Avoid consuming more than 5 grams of salt per day and limit water intake to 1 liter, especially if edema or ascites is present. [🚫] Exclude all forms of oils and fats except for TKA or Rising. Dairy-based products like ice cream should also be avoided, except for curd or yogurt. [🍪] Do not consume bakery foods like cookies and biscuits, and avoid alcohol, aerated beverages, and packed juices. [🍹] Fresh juices can be consumed in limited quantities. [🌱] Incorporate prebiotics like garlic, beans, ginger, and fenugreek seeds into the diet to support gut health. [🍌] Consider drinking banana stem juice twice a day, except for those with edema or arthritis. [🥕] Consume raw vegetable juice as a supplement, which can serve as a blood thinner and support gut microbes and cells. [🌾] Include flaxseed in the diet as a natural remedy for various health issues, such as cancer prevention and heart health. [🐟] Fish can be eaten, but it should not exceed 20% of the diet and should be prepared with specific herbs without frying. [🍗] Chicken can be included in the diet if baked, not fried, and should be consumed with limited salt. [☕] For cold, cough, or fever, prepare a spice tea with ginger, black pepper, cinnamon, and camel and consume it as a remedy. 📣 **Stay Connected!** For more insights and updates, follow us on our social media platforms: - Facebook: https://www.facebook.com/miracledrinksin/ - Instagram: https://www.instagram.com/miracledrinksin/ - Twitter: https://twitter.com/miracledrinksin Youtube : https://youtu.be/nIbkkwPIbIM

[mcatmemoranda]

General measures – The following general measures apply to patients with nonalcoholic fatty liver disease (NAFLD):

•For patients without serologic evidence of immunity, vaccination for hepatitis A virus and hepatitis B virus.

•Standard, age-appropriate immunizations.

•For patients with hyperlipidemia, lipid-lowering therapy.

•For patients with diabetes, optimizing blood glucose control.

●Weight management – We recommend weight loss for patients with obesity (Grade 1B). Weight loss has been associated with histologic improvement in patients with NAFLD. We advise patients to lose a minimum of five to seven percent of body weight at a rate of 0.5 to 1.0 kg per week (1 to 2 lb per week) through lifestyle modifications, including dietary therapy and exercise.

●Avoiding alcohol – For patients with NAFLD, we recommend refraining from heavy alcohol consumption (Grade 1A) and suggest abstinence from alcohol (Grade 2C). Heavy alcohol use is associated with alcohol-related liver disease and other adverse consequences, including cancers of the mouth and esophagus. In patients with or at risk for NAFLD, heavy alcohol use is associated with hepatic steatosis, hepatic injury, and fibrosis progression. Whether light to moderate alcohol consumption is harmful remains somewhat uncertain as data are mixed. In the absence of definitive data, we suggest abstinence from alcohol for patients with NAFLD.

●Medical therapy

•Patients with NASH but without diabetes mellitus – For patients with biopsy-proven nonalcoholic steatohepatitis (NASH) and fibrosis stage ≥2 but without diabetes, we suggest using vitamin E (800 international units per day) (Grade 2C). Limited evidence supports a benefit of vitamin E in patients without diabetes, but some observational studies suggest a possible increase in all-cause mortality with higher dose vitamin E. As a result, we discuss the risks and benefits with the patient before starting treatment.

•Patients with NASH and diabetes mellitus – For patients with NASH and diabetes mellitus, the presence of NASH can inform the choice of glucose-lowering therapy. Although initial therapy for type 2 diabetes mellitus is typically with metformin, the beneficial impact on liver histology with certain other insulin-sensitizing agents could be a consideration when choosing a second-line agent for patients with NASH who cannot take metformin or need additional glucose-lowering therapy. In this setting, pioglitazone and GLP-1 receptor agonists (eg, liraglutide, semaglutide) are reasonable options.

●Laboratory monitoring – We obtain serum aminotransferases (alanine aminotransferase and aspartate aminotransferase) three and six months after patients with NAFLD implement lifestyle interventions for weight loss. If the aminotransferases do not return to normal levels with weight loss or if they increase, we evaluate for an alternative cause of liver disease.

●Monitoring for fibrosis – For patients with biopsy-proven NASH, we obtain a noninvasive assessment for advanced fibrosis at a time interval determined by their clinical course:

•For patients who have not been able to lose at least five to seven percent of their body weight and/or have elevated serum aminotransferases, we obtain a noninvasive assessment every three years.

•For patients who achieve their weight loss goals and have normal serum aminotransferases, we obtain a noninvasive assessment every four years.

[aurvedicproductsindia]

Smartveda Liv Amrit | Smartveda Products

This powerful formula, enriched with Bhumi Amla, Nagarmotha, Kasni, and more, targets Fatty Liver Grades 1 and 2. Bhumi Amla extract stands out, aiding detoxification and supporting liver wellness. Embrace the natural choice for optimal liver function. https://smartveda.co/product/liv-amrit/

[healthcare-industry]

Liver Cancer Treatment in India

Liver cancer, also known as hepatocellular carcinoma (HCC), is a serious condition that requires prompt and effective treatment. In India, liver cancer treatments are conducted by highly skilled oncologists and multidisciplinary teams in specialized cancer centers equipped with advanced technologies. In this detailed point-wise exploration, we will delve into the causes, symptoms, diagnosis, treatments, and costs associated with liver cancer treatment in India.

1. Causes of Liver Cancer:

Liver cancer typically develops due to a combination of risk factors, including:

  a. Chronic Hepatitis B or C Infection: Long-term infections with hepatitis B or C viruses significantly increase the risk of developing liver cancer.

  b. Chronic Alcohol Abuse: Heavy and prolonged alcohol consumption can lead to liver cirrhosis, which increases the risk of liver cancer.

  c. Non-Alcoholic Fatty Liver Disease (NAFLD): The accumulation of fat in the liver can progress to liver inflammation and cirrhosis, increasing the risk of liver cancer.

  d. Cirrhosis: Liver cirrhosis resulting from various causes, such as viral hepatitis, alcohol abuse, or fatty liver disease, can lead to liver cancer.

  e. Genetic Factors: Inherited genetic conditions like hereditary hemochromatosis and certain metabolic disorders can increase the risk of liver cancer.

2. Symptoms of Liver Cancer:

The symptoms of liver cancer may vary depending on the stage of the disease. Common symptoms include:

  a. Abdominal Pain: Persistent pain or discomfort in the upper abdomen.

  b. Unexplained Weight Loss: Sudden and unexplained weight loss without any dietary or lifestyle changes.

  c. Jaundice: Yellowing of the skin and eyes due to impaired liver function.

  d. Loss of Appetite: A significant decrease in appetite and a feeling of fullness even after consuming small amounts of food.

  e. Fatigue and Weakness: Persistent fatigue and weakness, often accompanied by a decline in physical stamina.

3. Diagnosis and Staging:

The diagnosis of liver cancer involves a series of tests and evaluations to determine the extent of the disease. Diagnostic procedures may include:

  a. Imaging Tests: CT scans, MRI, and ultrasound are used to visualize the liver and identify any tumors or abnormalities.

  b. Blood Tests: Blood tests are performed to assess liver function, identify tumor markers, and detect viral infections.

  c. Biopsy: A tissue sample is obtained from the liver and examined under a microscope to confirm the presence of cancer cells and determine the tumor type and grade.

  d. Staging: Once liver cancer is confirmed, further imaging and tests are conducted to determine the stage of the disease, which helps guide treatment decisions.

4. Liver Cancer Treatments:

The treatment of liver cancer in India depends on various factors, including the stage of the disease, the overall health of the patient, and the extent of liver involvement. Treatment options may include:

  a. Surgery: Surgical procedures, such as liver resection (removal of the tumor) or liver transplant, may be recommended for early-stage liver cancer.

  b. Ablation Therapies: Techniques like radiofrequency ablation (RFA), microwave ablation, and cryoablation are used to destroy cancer cells in small tumors.

  c. Transarterial Chemoembolization (TACE): This procedure involves delivering chemotherapy drugs directly into the blood vessels feeding the tumor, followed by blocking the blood flow to the tumor.

  d. Targeted Therapy: Targeted drugs are used to inhibit specific molecules involved in the growth of cancer cells, often in cases where surgery is not feasible.

  e. Radiation Therapy: High-energy X-rays or other radiation sources are used to kill cancer cells and shrink tumors.

  f. Systemic Therapy: Chemotherapy or immunotherapy drugs may be prescribed to destroy cancer cells or enhance the body's immune response against cancer.

5. Cost of Liver Cancer Treatment in India:

The cost of liver cancer treatment in India can vary depending on several factors, including the stage of the disease, the chosen treatment modality, the duration of treatment, the hospital or cancer center, and additional services required. It is advisable to consult with an oncologist or a healthcare provider to get a personalized cost estimate based on individual circumstances.

Liver cancer treatment in India offers a comprehensive approach to diagnosing and treat liver cancer, providing patients with optimal outcomes and improved quality of life. With the expertise of highly skilled oncologists, state-of-the-art medical facilities, and a multidisciplinary approach, patients can receive effective treatment and personalized care in their journey toward combating liver cancer.

[susantaylor01]

Introduction 

Removing Belly fat  is about more than cosmetic purposes; it is essential for one’s health. Fat located in the abdomen is known to increase the risk for many severe diseases such as heart disease, diabetes and metabolic syndrome. Some of the practical ways of belly fat reduction include a healthy diet, exercise, stress relief and good sleep. By tackling those areas there would be sweetens the external look, but also prolongs the wholesome health.

What is Belly Fat and its Types ? 

Belly fat is simply defined as body fat that gets accumulated in the belly. Here two of the most commonly seen fatty deposits are:

Subcutaneous Fat: This is also known as dermal fat, or cutaneous fat, and protects the deeper body structures as it sets a layer beneath the skin. It is pain free and can be pinched by fingers, and is less dangerous than the visceral fat.

Visceral Fat: This is the fat located deep in the abdomen surrounding the internal organs such as the stomach intestines, liver and pancreas etc. This form of fat accumulation is regarded as harmful because it increases the risk of many health risks especially heart disease, type 2 diabetes and metabolic syndrome.

It is necessary to get rid of belly fat not only for aesthetics but also for health reasons.

What are the Risks Associated With Excess Belly Fat ? 

An excess in the amount of belly fat, mainly visceral fat comes with various health risks, these include the following: 

Heart Disease: Subcutaneous adipose tissue presents more than just wrinkles in terms of appearance. It is linked to certain cardiovascular diseases due to associated chronic-low-grade inflammation or other metabolic changes that can occur in patients with visceral fat. 

Type 2 Diabetes: Individuals will exhibit greater degrees of insulin resistance and likely a higher basal blood glucose level, therefore there is a high possibility that one will develop type 2 diabetes. 

Metabolic Syndrome: Metabolic syndrome as defined by the WHO is the constellation of clinical and laboratory signs which include abdominal obesity: hyperlipidemia: hypertension; diabetes – all of which are shown to increase the risk of coronary heart disease and diabetes. 

Inflammation: Toxins which ripen in the visceral fat may also stimulate the immune system through the hypoxic inflammation and angiogenesis process. 

Certain Cancers: Apparently obesity and being overweight are factors which increase the chances of some cancers especially: breast cancer; colon cancer; and cancer of the pancreas.

Dietary Changes Helpful To Remove Belly Fat 

Dietary changes that could help reduce belly fat include the following:

Increase Intake of Fibre: Foods containing soluble fibre, such as oats, beans, and vegetables, can help reduce visceral fat by creating a fuller feeling in the stomach and by improving digestion.

Lean Proteins: Inclusion of lean protein foods such as chicken, fish, tofu, and legumes promotes an increase in metabolism and building of muscles.

Limit Sugar-Containing Food and Drinks: Reduce sugar-sweetened beverages, candies, and baked goods, as high intake of sugar is associated with greater accumulation of fat, particularly in the abdomen.

Whole Grains: Eat whole grains instead of refined grains. Brown rice, quinoa, and whole-wheat bread are more nutritious and friendlier to weight management.

Healthy Fats: Avocados, nuts, seeds, and olive oil should be consumed, as these healthful fats may actually contribute to a reduction of overall fat without encouraging belly fat.

Limit Processed Foods: Limit processed foods and high-sodium foods; these will surely increase weight and cause bloating.

Hydrate: Drink plenty of water and limit high-calorie beverages. Proper hydration supports metabolism and digestion.

Control Portions: Pay greater attention to the size of portions so that overeating may not occur and the intake of calories can be better controlled.

Excersises Helpful To Remove Belly Fat 

A combination of strength training and cardiovascular workouts makes effective exercises to help reduce belly fat. Here are some key exercises:

Cardio Exercises: Activities such as running, swimming, cycling and brisk walking burn calories thereby making it possible for you to lose belly fat and overall body weight as well.

High-Intensity Interval Training (HIIT): Brief rest periods followed by intense workouts help one to effectively burn fat within a short time span because it creates a high demand for energy during exercise. Sprint intervals or circuit training are examples of HIIT.

Strength Training: Engaging in weightlifting or resistance band exercises together with bodyweight workouts including squats or lunges leads to building up muscle mass which boosts metabolism consequently reducing the amount of fat stored within the body

Core Exercises: Although they may not have direct impacts on belly fat reduction, these exercises strengthen core muscles resulting in better toned bodies and improved postures.

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Conclusion 

Implementing a multimodal strategy which includes dietary changes, exercise and some lifestyle changes is necessary to remove the bulge from the stomach. You can specifically fight against too much fat in your abdomen by having a balanced meal with lots of fibre, rich in low-fat protein and healthy fats as well as engaging in some cardiovascular activities and strength training. In addition, it is important to manage stress and to sleep enough for good health so as to help reduce body fat. These methods not only help to lose weight from the stomach but they also improve overall health and reduce the chances of developing other related diseases.

[drpathikparikh]

Evaluation of Fatty Liver – Beyond sonography and SGPT - Dr. Pathik Parikh

Evaluation of Fatty Liver – Beyond sonography and SGPT

How many of you have fatty liver on sonography?

How did you come to know that you have fatty Liver?

What tests did you carry out to evaluate fatty liver?

Fatty Liver is most diagnosed incidentally when patients get evaluated for abdominal discomfort or as a routine health checkup. Sometimes it gets diagnosed when altered liver tests are noticed. It affects nearly one quarter of our population.

Whenever there is fatty liver, the pragmatic next step is to start evaluating for metabolic syndrome like diabetes, hypertension, abnormal cholesterol levels, obesity, thyroid abnormalities, sleep apnea. The next step is to exclude other liver disease like viral hepatitis, autoimmune hepatitis, and metabolic liver disease. Once fatty liver is diagnosed, we need to evaluate the stage of fatty liver disease. There is a misconception that ultrasound gives you the stage of liver disease. It does not give stage of liver disease but just grade of fat in the liver. The sonography just mentions the degree of fat in the liver as grade 1, 2 or 3. It is insufficient to tell you the stage of liver disease. Also, SGPT or ALT is inadequate to give you the stage of liver disease. It may be falsely normal in more than 50 percent patients despite abnormal liver function.

More Information: https://www.drpathikparikh.com/evaluation-of-fatty-liver-beyond-sonography-and-sgpt/