#Meningioma Surgery

[hmsdesk]

(via Meningioma Surgery Cost in India)

[kondavanelos]

Μηνιγγίωμα εγκεφάλου.

[medical-23]

Illuminating the Path: Innovations in Meningioma Treatment

Meningiomas, the most common primary brain tumors, require careful consideration and personalized treatment approaches. These tumors arise from the meninges, the protective membranes surrounding the brain and spinal cord. The landscape of meningioma treatment has undergone remarkable advancements, providing hope and improved outcomes for affected individuals. This creative article explores the intricacies of meningioma treatment, delving into innovative surgical techniques, targeted radiation therapy, emerging molecular therapies, and the importance of multidisciplinary care. By illuminating the path of meningioma treatment, we strive to empower patients and healthcare professionals alike.

Understanding Meningiomas

Meningiomas are tumors that develop in the meninges, predominantly affecting the outer layer of the brain. While most meningiomas are benign, some can be aggressive and exhibit malignant behavior. These tumors can cause a variety of symptoms depending on their location and size, including headaches, seizures, vision problems, and neurological deficits.

Surgical Innovations: Maximizing Resection and Minimizing Risk

Surgery is often the primary treatment for meningiomas, with the goal of achieving maximum tumor resection while minimizing the risk of complications. Innovative surgical techniques, such as skull base approaches, neuroendoscopy, and intraoperative imaging, have revolutionized meningioma surgery. These advancements enable surgeons to access and remove tumors in challenging locations while preserving critical brain structures and minimizing the risk of postoperative complications.

Targeted Radiation Therapy: Precision and Efficacy

Radiation therapy plays a crucial role in meningioma treatment, particularly for tumors that cannot be completely resected or are recurrent. Recent advancements in radiation therapy techniques, such as stereotactic radiosurgery (SRS) and proton therapy, have significantly improved treatment outcomes. These modalities deliver precise and targeted radiation to the tumor while sparing surrounding healthy tissue, reducing the risk of side effects and promoting tumor control.

Molecular Therapies: Unlocking New Possibilities

Emerging molecular therapies are reshaping the landscape of meningioma treatment. Through the identification of specific genetic mutations and molecular pathways, targeted therapies, such as receptor tyrosine kinase inhibitors and hormone-based therapies, are being explored to disrupt tumor growth and improve patient outcomes. Clinical trials and ongoing research aim to uncover novel therapeutic targets and advance personalized treatment options for meningiomas.

Multidisciplinary Care: A Collaborative Approach

The management of meningiomas necessitates a multidisciplinary approach, involving a team of neurosurgeons, radiation oncologists, medical oncologists, pathologists, and other specialists. Collaborative tumor boards, where experts from different disciplines discuss individual cases, ensure comprehensive treatment planning and personalized care. This multidisciplinary approach facilitates optimal decision-making, tailoring treatment strategies to each patient's unique circumstances.

Rehabilitation and Supportive Care

Meningioma treatment extends beyond medical interventions. Rehabilitation therapies, including physical therapy, occupational therapy, and speech therapy, are essential for individuals to regain functional abilities and improve their quality of life. Supportive care, including psychological support and symptom management, helps patients cope with the emotional and physical challenges associated with meningioma treatment.

Promising Future Directions

The future of meningioma treatment holds promising prospects. Advancements in genomic profiling and molecular characterization of tumors will enable targeted therapies to become more precise and effective. Immunotherapies and novel drug combinations are also being explored to enhance treatment outcomes. Additionally, ongoing research aims to identify biomarkers that predict tumor behavior and response to treatment, further guiding personalized therapeutic approaches.

Conclusion

Meningioma treatment has witnessed significant advancements, providing patients with renewed hope and improved outcomes. Innovative surgical techniques, targeted radiation therapy, emerging molecular therapies, and multidisciplinary care are transforming the landscape of meningioma treatment. By staying at the forefront of medical innovation, researchers and healthcare professionals strive to improve patient outcomes, reduce side effects, and enhance quality of life. With continued research, collaboration, and patient-centered care, the path to overcoming meningiomas becomes increasingly illuminated.

[brightmoontrigon]

deer of 9 colors 🌙🌈

hello friends! my recovery from meningioma surgery has been coming along, albeit with both its ups (partial improvement in my eyesight, feeling strong enough to get around mostly at my normal level) and downs (diabetes insipidus, sinus congestion, weird episodes of joint pain, and having some ongoing visual field impairment). still, I'm very thankful for the good things. I'm trying to make a little art again and wanted to invoke this auspicious creature for some luck and benevolence <3

[emma-m-black]

Doctor White - Chapter One

Tom Koracick x OC (FanFiction)

This is a super rough draft of a Tom Koracick x OC story I've had in my head. I got a ton of chapters done, but then kind of his a block at a cliff hanger and I figure, perhaps if I post it, maybe I can figure out what to do next.

Rating is probably close to PG, don't think there is any spicey bits, pretty tame.

Chapter One:

Elizabeth walked through the halls of Grey Sloan Memorial Hospital in utter astonishment. She was finally here, the Hospital she had dreamed of working at. Tomorrow she would be in her final year of residency.

She could hear the room before she saw it. The sounds of multiple voices attempting to talk over one another as they vied for control over their conversations. She paused before she turned the corner to the staff cafeteria. Reaching down, she rubbed the palms of her hands against the dark velvet fabric of her dress. Elizabeth told herself it was to smooth out the wrinkles, but really it was to dry the sweat from her palms.

"I don't want to go in there either." Spoke a voice from behind her, it jarred her from her thoughts and her already racing heart sped up even more.

"Can you see my pit stains from back there, I had hoped this dress wouldn't show." Elizabeth let out a laugh as she turned around to see who had spoken to her.

A man quite a bit older than her stood before her. Slightly greying hair and a large smirk on his clean-shaven face. "Doctor Thomas Koracick." He began as he stuck out a hand in greeting and approached, "You must be one of the brilliant new Interns?"

"Actually I'm a transferring resident, a smart, brilliant, scared out of my mind, and regretting every moment that led me to being here all of a sudden senior resident." Tom gave a slight chuckle and glanced down to his still waiting hand. "Shit, sorry…, Doctor Elizabeth White, pleasure to meet you." She spoke as she straightened8 her back and reaching out to grasp his hand.

"White? Any relation to Doctor Wilfred White?" Asked Tom.

"Yeah. He is my father."

"I'm sorry." Said Tom, releasing Elizabeth's hand.

"Sorry?" Elizabeth questioned not use to that response when it came to her family legacy.

"That you have to deal with that ego on a daily basis. What was it like growing up with him as a father? I can just imagine what dinners must have been like." Said Tom as his eyes raked over her with a smile. "So were you planning on following in his footsteps."

"Cardio no, I'm interested in Ophthalmology."

The smile on Tom's face dropped. His mouth opened, and then he closed it, seemingly at a loss for words.

Elizabeth let out a laugh. "I'm joking. Neurosurgery is my specialty, the brain is what led me to becoming a surgeon. I had a grade two Atypical Meningioma." Said Elizabeth as she raised the hand and tapped her skull. "Had to learn everything brain related when I was diagnosed, and now all I want is brains. All I think about now is brain tumours."

"Sexy." Tom said with a smile. "The tumour, that is. I'm assuming you've had surgery, and you aren't about to enter your internship with a ticking time bomb?"

"Yeah, I was nineteen at the time."

"Reoccurrence?"

"Radiation treatments after the total resection for good measure. So far, no reoccurrence."

"And you've been keeping up with your scans?"

"Yes."

"Who was your surgeon?"

"Doctor Samuel Gravely."

"Samuel's a quack. I'll find you tomorrow, and we will do a CT, make sure he didn't mess up that legacy of a brain of yours." Tom stepped forward and placed a fist to his hip and extended his elbow toward Elizabeth. "Now come on, I want to start some rumours before your first day."

Elizabeth stared at him with wide eyes, unable to respond to his proposal. "Come on, trust me, it will give you some street cred, all the gossip tomorrow will be about you. Which gives you the advantage of standing out to all the important people." Elizabeth laughed and slipped her arm around his before he walked the two of them into the busy room, the busy room of which a good few people stopped to watch as both her and Doctor Koracick.

She could hear a few whispered words asking who she was and why she was with Doctor Koracick. It was then though that she spotted Doctor Bailey, who had also just seemed to have spotted her.

"Ah, Doctor White, I'm glad you could make it." Miranda said as she approached her. Elizabeth noticed the side eye that was given to Tom as she came to a stop in front of them. "Doctor Koracick, do you two know each other?"

"We've been bonding over her sexy brain tumour." Tom responded.

"Interesting." Miranda narrowed her vision at the older man. "Doctor Webber!"

Elizabeth watched as an older man walk towards her, she knew from her pry into the hospital's staff that this was the Doctor Richard Webber.

When Doctor Webber was in front of them, he eyed Elizabeth suspiciously.

"Doctor Webber, I would like to introduce you to Doctor Elizabeth White. She was the one I told you about."

"White…" Richard spoke as he extended a hand. "Like the Cardio…"

"Yes, Dr. White is my father, Doctor Webber."

"Doctor Bailey told me that she hired a prodigy, but I never thought…" he trailed off.

"I hope I can live up to his reputation."

"I hope not." Said Tom with a smirk on his lips. "He's a prude. No offence." He finished looking to Elizabeth.

"I'm sure we will see great things from Doctor White. She was top of her class at Harvard, and I had to offer her a great deal to get her to leave Duke."

"I would have come regardless." Elizabeth laughed. "This was always my top choice."

"Doctor Koracick, I didn't realize you were still here." Came the happy voice of Amelia Shepherd.

"Yeah, I had allowed for a few extra days to stick around, you know, in case you became a cabbage." As Tom talked, a group started to form around Elizabeth.

"And who is your friend?" Asked Amelia.

Miranda took notice of the people around them and quickly threw a sweeping hand out. "This is our new Senior Resident Doctor, Elizabeth White…" Miranda stretched out the last name, giving everyone time to process what she was saying.

"Wait like…"

"Yes." Miranda said quickly.

"Who…"

"She is his daughter."

"But I thought…"

Miranda waved her hands in the air. "Let's leave the poor girl alone, she can answer all your questions tomorrow. Now go, mingle, all of you. Including you, Doctor Koracick"

Elizabeth extracted her arm from Tom's and placed a hand to his chest instead. "Don't worry, you get to check me out tomorrow." Elizabeth threw a wink at him before walking away towards the drink table.

A dark squint was sent from Miranda to Tom. "Her brain. I'm checking out her brain tomorrow."

"I'm watching you Koracick, I'm watching you."

Chapter Two

[jcsmicasereports]

Primary dural-based parafalcine diffuse large b-cell lymphoma mimicking meningioma by Amr El Mohamad in Journal of Clinical Case Reports Medical Images and Health Sciences

Abstract

Background: Primary dural-based diffuse large B-cell lymphoma is very rare. Only few cases were reported in the literature. Case presentation: Herein, we present a case of an immunocompetent patient with primary dural-based diffuse large B-cell lymphomas mimicking meningioma associated with ghost tumor phenomenon without any evidence of a systemic lymphoma. Conclusion: Primary central nervous system lymphomas are rare. Clinicians should always consider this lesion as a differential diagnosis if radiological findings are not indicative of typical one meningiomas.

Key words: Dural-based tumor, diffuse large B-cell lymphoma, ghost tumor, MATRIX regimen, central nervous system.

Introduction

Primary central nervous system lymphomas (CNSLs) (PCNSLs) are rare and account for 2%–5% of all brain tumor cases, whereas secondary CNSLs are more common [1,2]. One study has shown that the most common intraparenchymal histological type is diffuse large B-cell lymphoma, as among 26 patients with PCNSL, 25 had diffuse large B-cell lymphoma [3]. Although primary dural-based lymphomas are rare, the most common area of involvement is the cerebral hemispheres. Most dural-based lymphomas are secondary and present as extra-nodal systemic diffuse large B-cell lymphomas. Primary dural-based lymphomas are usually histologically marginal-zone lymphomas, representing a group of lymphomas that have been historically classified together because they appear to arise from post-germinal center and marginal-zone B cells and share a similar immunophenotype, and few cases were reported to be diffuse large B lymphomas [4]. Here, we present a case of an immunocompetent patient with primary dural-based diffuse large B-cell lymphomas mimicking meningioma associated with ghost tumor phenomenon without any evidence of systemic disease.

Case Presentation

A 58-year-old male individual previously healthy and immunocompetent presented with headache, recurrent vomiting, and memory problems lasting for 3 days. No loss of consciousness, seizure, subjective weakness, or fever was observed. On physical examination, the patient’s Glasgow coma scale score was 15; his pupils were 3 mm in diameter, equal, and reactive; and the patient had nominal aphasia without motor and sensory deficit. He had normal cerebellar functions, and cranial nerve exams revealed no deficit. Head computed tomography (CT) (Fig. 1) showed a 2.2 × 3.8 cm (transverse × anteroposterior) iso-dense lesion with internal hypodensity in the left parasagittal frontal region extending to the right frontal region. Extensive perilesional edema was observed with effacement of the sulci and mass effect on bilateral frontal horns, associated with 3-mm midline shift. Head magnetic resonance imaging (MRI) showed an isointense parasagittal lesion on T1 and heterogeneous intense on T2, with redemonstration of perifocal edema (Fig. 2). Head T1-weighted imaging with contrast enhancement (Fig. 3) showed a large, left frontal, parafalcine, irregular-shaped mass located below the superior sagittal sinus level. It measured 4 × 3 × 3.3 cm in anteroposterior, mediolateral, and craniocaudal, respectively. It showed diffusion restriction (Fig. 4). There was central hyperintensity on T2-weighted imaging, without post-contrast enhancement area representing cyst formation. It exerts a mass effect characterized by effacement of the adjacent sulci, compression of the left lateral ventricle, and a 3-mm shift of the midline structures to the right side, and the impression of our neuroradiologist was atypical meningioma. Regarding extensive edema, dexamethasone was started at a dose of 4 mg, thrice a day, and the patient was planned for craniotomy and resection of the tumor. Initially, the patient was reluctant to undergo surgery; however, subsequently, the patient agreed to undergo surgery after approximately 10 days. During surgery, parasagittal craniotomy was performed; however, to our surprise, no definite mass lesion was found at the proposed site, in contrast to the findings described on imaging. The falx was thinned out and partly deficient. A biopsy sample was obtained from this abnormally appearing falx. Moreover, we obtained biopsy samples under neuronavigation guidance from abnormally appearing tissue, which was completely intra-axial, deep down in the lesion visualized on navigation. On postoperative day 1, MRI head with contrast enhancement (Fig. 5) showed that the previously seen lesion had a significant regression in size. Its right frontal extension and adjacent enhanced meningeal tail showed size reduction. Moreover, some regression in the perilesional vasogenic edema was observed. A significant regression in the previously described enhancement was noted at the left-side lentiform nucleus and external capsule. The MR spectroscopy study showed an increased choline/N-acetyl aspartate ratio and elevated lactate level within the lesion.

The histopathology results of the first brain biopsy samples (Figs.6–7) obtained from the falx cerebri showed meningothelial hyperplasia with calcification and focal perivascular lymphocytic infiltrate composed of small and large, atypical lymphocytes. Immunohistochemical staining was performed; however, the area of interest disappeared. The pathology team recommended another fresh biopsy to have the final diagnosis and flowmetry studies. So, the patient underwent redo craniotomy using the same incision, and multiple biopsy samples were taken. The second fresh brain biopsy (Figs. 8–9) showed multiple brain fragments with predominant perivascular atypical lymphoid infiltrates. Most cells were medium to large with moderate cytoplasm, atypical irregular nuclei having vesicular chromatin, variably prominent nucleoli, and several mitoses, including atypical one. Necrotic areas were also seen. Immunohistochemistry of the second biopsy (Fig.10 A-D) showed that atypical perivascular cells were positive for CD45, CD20, CD79a, BCl2, BCl6, MUM1, OCT2, and C-MYC, and negative for CD10, CD21, TDT, ALK1, EBV-LMP1, CD3, and CD5; however, few reactive/residual lymphocytes were positive for these enzymes. Moreover, 80% of lymphoid cellular nuclei were positive for Ki67. These findings were consistent with diffuse large B-cell lymphoma, not otherwise specified.

Whole-body positron emission tomography (PET) showed intense fluorodeoxyglucose (FDG) uptake higher than that in the healthy brain cortex, without evidence of coexisting systemic disease. In addition to PET scan, contrast-enhanced chest, abdomen, pelvis CT did not show any other lesions in the body; furthermore, workup for viral markers and autoimmune conditions were all unremarkable, thus confirming the diagnosis of “primary dural-based diffuse large B-cell lymphoma,” distinguishing it from secondary CNSL. The patient was transferred to the Oncology Department and started on three cycles of the methotrexate, cytarabine, thiotepa, and rituximab (MATRIX) protocol, which is the current standard treatment regimen for PCNSLs [5]. Three months after the diagnosis and after receiving two cycles of the MATRIX protocol, brain MRI with contrast enhancement (Fig. 11A, B) showed regression of the lesion, and PET scan showed complete metabolic resolution in terms of decreased FDG activity of the previously seen PCNSL without signs of lymphoma activity elsewhere. Subsequently, the patient received the third cycle of the MATRIX protocol without specific complications. Two weeks later, autologous stem cell transplantation (50 × 106/kg) was performed as part of the consolidation phase of treatment. Six weeks later, conditioning chemotherapy with carmustine–thiotepa was administered, followed by stem cell infusion (CD34 = 12 million/kg). The post-transplant course was complicated with mucositis, folliculitis, diarrhea, febrile neutropenia, and prolonged thrombocytopenia. Two months after transplantation, PET scan was repeated and showed complete metabolic resolution of initially seen PCNSL involvement. Currently, the patient is being followed by the hematology team; the patient is in good health and remission. The last outpatient follow-up was 8 months after the first surgery. The patient was seen by the vascular surgery (for permcath removal) and oncology teams. At this time, the patient was stable with complete remission; then, the patient was lost to follow-up. Another head MRI was performed and showed almost total regression of the lesion.

Discussion

Lymphomas in CNS are classified as primary, arising de novo from brain parenchyma, leptomeninges, eye, and spinal cord and as secondary to systemic lymphoma, which can be dural-based lesions. Secondary CNSLs are more common than PCNSLs. Most PCNSLs are intraparenchymal diffuse large B-cell lymphomas with a predilection to occur in the frontal lobe and then deep nucleic and periventricular locations; the infratentorial cerebellum is the most common location. However, primary dural-based lymphomas are rare, and even when found, they are histologically marginal-zone lymphomas. Few cases of primary dural-based diffuse large B-cell lymphoma have been reported in the literature [4,6]. Furthermore, PCNSLs are more common in immunocompromised patients with a mean age of 34 years, and they occur in immunocompetent individuals at an older age with a mean of 52 years [7]. The patient in this case report was 58 years old and immunocompetent without significant previous medical conditions. The latest review of the literature on primary dural-based lymphoma has been conducted by Quinn et al., who have found only 24 reported cases of primary dural-based diffuse large B-cell lymphoma, which confirms the rarity of the disease and subsequently the limited knowledge regarding this disease entity [8]. CNSLs have rapid response to steroids with shrinkage in size and initial remission [9]. Moreover, the initial response to steroids is associated with a better response to chemoradiotherapy and good prognosis [9]. In the patient in this case report, there was an unintentional delay of surgery for approximately 10 days, and the patient was on steroids (dexamethasone). In this case report, the failure to identify a discrete lesion of the size expected as perceived on initial imaging, despite proper surgical planning using neuronavigation, was probably due to the rapid regression of the tumor in response to steroids. This phenomenon agrees with the scientific literature reporting about the disappearance of lymphomas in response to steroids (ghost tumors) [10,11]. The pathogenesis of primary dural-based lymphoma remains unknown as there is no lymphoid tissue in the dura. It is hypothesized that it is related to chronic infection, autoimmune disease, or chronic inflammatory condition, which recruits polyclonal lymphocytes resulting in monoclonal lymphomas [6]. In contrast, the patient in this case report did not have any chronic conditions. All workups were negative, including the entire viral panel and autoimmune markers. Basic research is needed to determine the etiology of PCNSL, especially dural-based lymphomas. In the patient in this case report, the initial radiological findings were mimicking those of a meningioma: dural-based and uniformly enhanced. There was significant surrounding edema, significant diffusion restriction, and blooming in susceptibility-weighted image, which goes more with higher-grade meningioma or another high-grade lesion. One review has shown that primary dural-based lymphomas can display the “dural tail “sign, further confusing the preoperative diagnosis with meningioma [12], which did happen in the patient in this case report. Therefore, we suggest that in case of a dural-based lesion that has non-typical features of grade 1 meningioma, clinicians should consider lymphoma in the differential diagnosis and avoid steroids unless necessary due to edema and mass effect keeping in mind the ghost tumor phenomena of lymphoma.

The role of surgery in PCNSLs is limited mainly to histological diagnosis through biopsy or tumor debulking in case of increased intracranial pressure or impending brain herniation. Some studies have shown no benefit of complete surgical resection of PCNSLs; however, a recent systematic review of 244 articles has shown evidence in support of cytoreductive surgery [13]. Previously, whole-brain radiotherapy (WBRT) was the recommended treatment; however, this treatment modality resulted in a high rate of relapse and a decrease in performance status and cognitive impairment, and with the improvement in survival with high-dose methotrexate, WBRT is no longer recommended. Currently, newly diagnosed PCNSLs are initially treated with induction chemotherapy until complete radiological response, followed by consolidation therapy, to prolong the overall survival [14]. The International Extra Nodal Lymphoma Study Group-32 trial has shown that a methotrexate-based MATRIX regimen results in a good outcome and control rate in PCNSL [5], and it is the standard induction chemotherapy. Ferreri AJM, in his article “The role of autologous stem cell transplantation in PCNSL” has compared various consolidation phase treatment modalities, including beam radiation, carmustine–thiotepa regimens, and autologous stem cell transplantation, and the results showed that autologous stem cell transplantation resulted in good outcomes [15]. The patient in this case report showed a good response to treatment with almost total resolution of PCNSL with three cycles of MATRIX chemotherapy, followed by conditioning chemotherapy with stem cell infusion.

Conclusion

PCNSL is a rare entity. Clinicians should always consider it in differential diagnosis of meningioma if the radiological findings are not typical for meningioma. When there is a high index of suspicion of lymphoma, repeating neuroimaging, particularly MRI, before surgery, especially if the surgery is delayed while the patient is on steroids, may help develop a better management plan while dealing with this rare lesion. In case of lesion disappearance, falx biopsy can be an option. The aim of surgery in PCNSL is mainly biopsy or debulking to decrease intracranial pressure in case of significant mass effect.

List of abbreviation:

Primary central nervous system lymphomas (PCNSLs)

Central nervous system lymphomas (CNSLs)

Head computed tomography (CT)

magnetic resonance imaging (MRI)

positron emission tomography (PET)

fluorodeoxyglucose (FDG)

whole-brain radiotherapy (WBRT)

[pepsiicoia]

Hi, On May 13th, I will be undergone brain cancer surgery to remove a large meningioma and radiation chemotherapy. The recovery time for this type of procedure is a minimum of two months. I am employed as a bartender and will have no income during the recovery period. Donations will go towards living expenses when I am unable to work. Your contributions will be greatly appreciated! Please donate whatever you can. Goal: $1800

WHAT. (There’s no way in hell this isn’t a scam, also I’m a minor with no money errr)

[spartalabouche]

Hi, On May 13th, I will be undergone brain cancer surgery to remove a large meningioma and radiation chemotherapy. The recovery time for this type of procedure is a minimum of two months. I am employed as a bartender and will have no income during the recovery period. Donations will go towards living expenses when I am unable to work. Your contributions will be greatly appreciated! Please donate whatever you can. MY GOAL IS: $1800 DONATIONS & REBLOGS ARE GREATLY APPRECIATED👏

boooo bad scam. dont donate to this person their blog was made literally today also im sorry what kind of username is mysticfartwombat for your brain surgery fund blog

[lov3rgrrrl]

Hi, On May 13th, I will be undergone brain cancer surgery to remove a large meningioma and radiation chemotherapy. The recovery time for this type of procedure is a minimum of two months. I am employed as a bartender and will have no income during the recovery period. Donations will go towards living expenses when I am unable to work. Your contributions will be greatly appreciated! Please donate whatever you can. Goal: $1800

scam :(

[tw33k-tucker]

surgery to remove a large meningioma and radiation chemotherapy. The recovery time for this type of procedure is a minimum of two months. I am employed as a bartender and will have no income during the recovery period. Donations will go towards living expenses when I am unable to work. Your contributions will be greatly appreciated! Please donate whatever you can. MY GOAL IS: $1800 DONATIONS & REBLOGS ARE GREATLY APPRECIATED THANKS 👏

I'm sorry but I am LITERALLY a child, again I'm sorry, but you might wanna send these to an adult instead of a 12(almost 13) year old, because I do not have a job to help, because I am a child

I hope you get your goal though

[lastlycoris]

What surgeries do you have scheduled?

Three surgeries.

Two brain aneurysm coilings. We're going to go through the carotid artery, snake a catheter into the circle of Willis / brain vasculature, and deploy a coil in a sizable anterior cerebral artery aneurysm - an aneurysm is essentially a focal sac that develops in a blood vessel - think of it as a balloon under high pressure - a weakness that can -pop-.

The aneurysms in these cases are big and are at risk of popping with devastating consequences. Unlike the middle meningeal artery I mentioned before, the vessels involved (circle of Willis) directly supply the brain.

Ever experienced a water pipe leak? You have a hole in the water supply somewhere, and you're no longer able to get water from the faucet due to the inadequate water pressure. That's what happens when an aneurysm pops - the brain loses a good portion of its blood supply, and you have what we called a hemorrhagic stroke with all the loss of movement and function that goes with it.

It's actually a bit more complicated than that. Blood is also an irritant in the brain and can cause the brain vessels to constrict in a phenomenon known as vasospasm, which again deprives the brain of oxygen and nutrients.

So we're going to coil these aneurysms off before they pop and do that.

----

Third case is a pituitary macroadenoma resection. Essentially, the guy came into clinic with a very specific visual field loss known as bitemporal hemianopsia - he lost the outermost fields of his vision with the inner part of his field intact.

That specific visual loss pattern tells us that something is affecting the optic nerves in a particular way, specifically the optic chiasm where the two optic nerves share fibers before going to the eyes. We were not surprised to find a pituitary macroadenoma pressing from under the optic chiasm on the head CT - pituitary adenomas are the second most common type of intracranial tumor (meningioma is first).

We are going to remove the tumor by going through his nose and entering into the sphenoid sinus, where the sella turcica is - the place where the pituitary lives. And yes we will be pulling out bits of tumor from his nose.

The advantage of this is that I do not have open up the skull to get to the tumor - and by not opening up the skull, I don't have to interact with the rest of the brain. That is a good thing.

Small thing to worry about is that patient has annoying variant anatomy involving the sphenoid sinus, known as a post-sellar sphenoid sinus. Normally you have a centimeter or so of bone between the sinus and where the brain stem lies. People with post-sellar sphenoid sinuses don't have that.

And yes, poking the brain stem is bad.

--

That's my outpatient schedule for the most part. And then they stuck me on emergent call duties for Trauma for the afternoon / later part of the night.

The things I do to avoid going back to jail.

[stillnotyourmusebitch]

I agree doctors need to step up. My sister has had migraines for over 4 years and the doctor always said things like it's tension headaches, lose weight blah blah blah. When she was explaining about being tired all the time to the doctor they put it off as exercise more. But she ended up in hospital with blood transfusion and iron drip coz she was so anemic and don't get me started on the fact my sister has to have brain surgery in June this year because she had to have a 4cm meningioma removed. All because doctors kept putting it down to her being fat.

She is doing better now but if she was given an MRI all those years ago and the blood tests she wouldn't have had to go through all that shit.

This is why fat shaming can have tragic consequences.

[carinasharma]

Revolutionary Advances in Head and Neck Cancer Surgery: What You Should Know

Head and neck cancers encompass a diverse group of malignancies that affect areas such as the mouth, throat, larynx, nose, sinuses, and salivary glands. Recent advancements in surgical techniques have significantly improved patient outcomes, offering hope to many. This article delves into these groundbreaking developments, highlighting the expertise available at Action Cancer Hospital in Delhi.​

Understanding Head and Neck Cancers

Head and neck cancers originate in the squamous cells lining the moist surfaces inside the head and neck. Risk factors include tobacco use, excessive alcohol consumption, and human papillomavirus (HPV) infection. Early detection is crucial, as it significantly enhances the effectiveness of treatment.

Traditional Surgical Approaches

Historically, treating head and neck cancers involved extensive surgeries that often led to significant physical and functional impairments. Procedures like radical neck dissections, while effective in removing tumors, frequently resulted in challenges related to speech, swallowing, and appearance.

Minimally Invasive Techniques

The advent of minimally invasive surgical techniques has revolutionized the treatment landscape:​

Transoral Robotic Surgery (TORS): Utilizing robotic systems, surgeons can access tumors through the mouth, eliminating the need for external incisions. This approach offers enhanced precision and reduces recovery times.

Endoscopic Transorbital Approach: A groundbreaking procedure where surgeons access tumors through the eye socket. In a notable UK-first operation, a meningioma tumor was successfully removed from a patient's cavernous sinus using this method, resulting in minimal scarring and a swift recovery.

Personalized Treatment Plans

Modern oncology emphasizes personalized treatment plans, tailoring interventions based on the tumor's genetic profile and the patient's overall health. This approach ensures optimal outcomes with minimized side effects.​

Advances in Reconstructive Surgery

Post-tumor removal, reconstructive surgery plays a pivotal role in restoring function and appearance:​

Microvascular Free Tissue Transfer: This technique involves transplanting tissue from one part of the body to the surgical site, restoring form and function effectively.​

3D Printing and Custom Implants: The use of 3D printing technology allows for the creation of patient-specific implants, enhancing the precision and aesthetic outcomes of reconstructive procedures.​

The Role of Multidisciplinary Teams

A collaborative approach involving surgeons, oncologists, radiologists, pathologists, and rehabilitation specialists ensures comprehensive care. This multidisciplinary strategy addresses all facets of the patient's well-being, from diagnosis through recovery.​

Patient Stories: Triumph Over Adversity

Real-life accounts underscore the impact of advanced surgical interventions:​

Beth Riehle: After losing 80% of her tongue to cancer, Beth turned to social media platforms like TikTok to document her journey of relearning speech, inspiring many with her resilience. ​

Aaron Whittington: At just 16, Aaron underwent surgery to remove half of his tongue due to aggressive cancer. His determination to resume normal activities post-recovery highlights the importance of early intervention and comprehensive care. ​

Janine Avanti: An actress and bodybuilder, Janine's routine plastic surgery consultation led to the discovery and removal of a malignant tumor in her neck, emphasizing the value of vigilance and expert medical attention. ​

Importance of Early Detection

Early diagnosis remains a cornerstone in the successful treatment of head and neck cancers. Regular medical check-ups and being attentive to symptoms like persistent sores, difficulty swallowing, or unexplained weight loss can lead to timely interventions.​

Choosing the Right Surgical Team

Selecting an experienced surgical team is paramount. Factors to consider include:​

Expertise in Advanced Techniques: Surgeons proficient in the latest minimally invasive procedures can offer better outcomes.​

Multidisciplinary Collaboration: A team that collaborates across specialties ensures comprehensive care.​

Patient-Centered Approach: Facilities that prioritize personalized care plans tailored to individual needs enhance the treatment experience.​

Action Cancer Hospital: Excellence in Head and Neck Cancer Surgery

Located in Delhi, Action Cancer Hospital stands out as a premier institution for head and neck cancer treatment. With a team of seasoned professionals adept in cutting-edge surgical techniques and a commitment to holistic patient care, the hospital has garnered recognition for its excellence in oncology services.​

Conclusion

The landscape of head and neck cancer surgery has witnessed remarkable advancements, offering patients improved prognoses and quality of life. Embracing these innovations, coupled with early detection and personalized care, paves the way for enhanced treatment experiences. For those seeking expert care in Delhi, Action Cancer Hospital exemplifies the integration of advanced surgical techniques with compassionate patient management.

[angiosarcoma]

It started with just a constant low level headache at the end of summer. Every day, all the time.

But easy enough to ignore, not really even worth taking a Tylenol (as if would matter it never responded to pain meds)

It was just there. First medical appointment “likely a tension headache. Do this and that”but it never helped. Final got an order for a CT scan, which the ordering MD didn’t think would result in anything. Scheduled six weeks out. Finally performed the week before Christmas, with the results released to me 12/27. A 3cm x 3cm x 2 cm mass. A bit over a square inch in total.

“Likely a meningioma. 95% are benign”

Referral to neurosurgeon. Follow up MRI

“Likely giant cell tumor. 95% are benign”

Surgery scheduled.

Tons of support family, friends, coworkers. “You’ll be in and out, young and strong, right as rain”

Portion of skull removed and replaced with titanium mesh. Recovery easy as can be. Only two nights in hospital. Surgery on Monday, feeling nearly right by Friday.

Friday 4pm. Call from neurosurgeon. Biopsy back. None of those.

Angiosarcoma. Rare sarcoma. Malignant.

Immediate referral to oncology.

Recovery weekend is fucked. I am for all practical purposes is royally fucked as well.

[health-gates]

Spinal Cord Tumor Surgery in India

Introduction

Spinal cord tumors are abnormal growths that develop within or near the spinal cord, causing pain, neurological deficits, and mobility issues. Spinal Cord Tumor Surgery in India offers advanced medical care, skilled neurosurgeons, and cost-effective treatment options for patients seeking relief and recovery.

What is Spinal Cord Tumor Surgery in India?

Spinal cord tumor surgery is a procedure performed to remove benign or malignant tumors affecting the spinal cord, nerve roots, or surrounding structures. It is essential for preventing further neurological damage and improving the patient’s quality of life.

Types of Spinal Cord Tumors

Intramedullary Tumors – Tumors that grow inside the spinal cord (e.g., astrocytomas, ependymomas).2. Extramedullary Tumors – Tumors that develop outside the spinal cord but within the spinal canal (e.g., meningiomas, schwannomas).3. Metastatic Tumors – Cancerous tumors that spread to the spine from other parts of the body.

Who Needs Spinal Cord Tumor Surgery in India?

Patients who experience the following symptoms may require surgery:

Constant back or neck pain

Weakness or numbness in organs

Coordination or balance loss

bladder or bowel relaxation

Paralysis in severe cases

Procedure for Spinal Cord Tumor Surgery in India

Preoperative diagnosis: MRI, CT scan, and biopsy to determine the location and type of tumor.

Anesthesia: General anesthesia is administered to a painless process.

Surgical removal: Neurosurgeons remove the tumor using minimal invasive or open surgical techniques.

Spinal stabilization: If necessary, spinal fusion or implant is used to restore stability.

Postoperative Care: Intensive monitoring, physiotherapy, and rehabilitation for recovery.

📅 Book an Appointment Today! 📞 Contact us at +91 9468115100

[moulana-malappuram]

Best Hospital for Brain Tumor Surgery: A Guide to Causes, Symptoms, and Treatment Plans

A brain tumor is an abnormal growth of tissue within the brain. These tumors can be categorized as either cancerous (malignant) or non-cancerous (benign). Malignant tumors can spread to other parts of the body, while benign tumors remain confined to their original location.

Brain tumors can develop in different areas of the head, including the skull, meninges, and brain tissue. The severity and type of symptoms depend on the tumor’s size and location. Moulana Hospital recognized as the best hospital for brain tumor surgery near Palakkad, provides advanced treatment and expert care for patients with brain tumors.

What causes brain tumors?

The precise causes of brain tumors remain uncertain. However, several risk factors may contribute to a higher likelihood of developing a brain tumor, including:

Age

Brain tumors can occur at any age, but they are most common in children and older adults.

Family History

People with a family history of brain tumors are at increased risk of developing the disease.

Genetic Syndromes

Some genetic syndromes, such as neurofibromatosis and tuberous sclerosis, increase the risk of developing brain tumors.

Exposure to Radiation

Exposure to high levels of radiation, such as from X-rays or nuclear fallout, can increase the risk of developing brain tumors.

Certain Chemicals

Exposure to certain chemicals, such as vinyl chloride and arsenic, has been linked to an increased risk of brain tumors.

How many types of brain tumors are there?

Medical history has documented over 150 distinct types of brain tumors. Below are some of the most common types of primary brain tumors:

Gliomas

Gliomas are the most common type of brain tumor. They arise from glial cells, which are cells that support the neurons in the brain.

Meningiomas

Meningiomas are tumors that arise from the meninges, which are the membranes that cover the brain and spinal cord.

Medulloblastomas

Medulloblastomas are tumors that arise from the cerebellum, which is a part of the brain that controls coordination and balance.

Pituitary Adenomas

Pituitary adenomas are tumors that arise from the pituitary gland, which is a gland located at the base of the brain.

Symptoms Of Brain Tumor

The brain tumor symptoms will vary depending on the location and size of the tumor. Some common symptoms of brain tumors include:

Headaches

Seizures

Vision changes

Hearing changes

Balance problems

Treatment of Brain Tumor

Surgery

Surgery is the most common treatment for brain tumors. The goal of surgery is to remove as much of the tumor as possible.

Radiation Therapy

Radiation therapy uses high-energy rays to kill cancer cells. Radiation therapy can be used to shrink a tumor before surgery or to kill any remaining cancer cells after surgery.

Chemotherapy

Chemotherapy uses drugs to kill cancer cells. Chemotherapy can be used to treat brain tumors that cannot be surgically removed or that have spread to other parts of the body.

Brain tumors are a complicated condition, and treatment varies based on individual cases. The most effective approach is to consult a neurologist, assess the prognosis, and choose a top multispecialty hospital nearby. Moulana Hospital is home to some of the finest neurologists and neurosurgeons in Kerala, dedicated to providing expert care for your loved ones.